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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

Cbp/p300-interacting transactivator with Glu/Asp-rich carboxy-terminal domain 1

This gene encodes a member of the CREB-binding protein/p300-interacting transactivator with Asp/Glu-rich C-terminal domain (CITED) family of proteins. The encoded protein, also known as melanocyte-specific gene 1, may function as a transcriptional coactivator and may play a role in pigmentation of melanocytes. Alternatively spliced transcript variants have been described. [provided by RefSeq, Jan 2009] (from NCBI)
Top mentioned proteins: CAN, p300, CBP, HAD, ACID
Papers on CITED1
Regulation of human subcutaneous adipocyte differentiation by EID1.
Lizcano et al., Maebashi, Japan. In J Mol Endocrinol, Feb 2016
Furthermore, increased expression of the beige phenotype markers CITED1 and CD137 was observed.
TGF-β-Induced Transcription Sustains Amoeboid Melanoma Migration and Dissemination.
Sanz-Moreno et al., London, United Kingdom. In Curr Biol, Dec 2015
We observe that downstream of TGF-β, SMAD2 and its adaptor CITED1 control amoeboid behavior by regulating the expression of key genes that activate contractile forces.
A synthetic niche for nephron progenitor cells.
Oxburgh et al., West Scarborough, United States. In Dev Cell, Aug 2015
Single-cell culture reveals phenotypic variability within the early CITED1-expressing NPC compartment, indicating that it is a mixture of cells with varying progenitor potential.
A stringent validation of mouse adipose tissue identity markers.
Nedergaard et al., Stockholm, Sweden. In Am J Physiol Endocrinol Metab, Jul 2015
Most of these markers unexpectedly turned out to be noninformative concerning tissue classification (Car4, Cited1, Ebf3, Eva1, Fbxo31, Fgf21, Lhx8, Hoxc8, and Hoxc9).
p53 Enables metabolic fitness and self-renewal of nephron progenitor cells.
Saifudeen et al., New Orleans, United States. In Development, May 2015
Conditional deletion of p53 in nephron progenitor cells (Six2Cre(+);p53(fl/fl)) induces progressive depletion of Cited1(+)/Six2(+) self-renewing progenitors and loss of cap mesenchyme (CM) integrity.
Microarray analysis of differentially expressed genes regulating lipid metabolism during melanoma progression.
Sudhakar et al., In Indian J Biochem Biophys, Apr 2015
The methods were validated by identifying known melanoma biomarkers (CITED1, FGFR2, PTPRF, LICAM, SPP1 and PHACTR1) in our results.
[Activation of phospholipase C-independent protein kinase C signaling pathway of parathyroid hormone enhances CITED1 expression in mouse osteoblasts].
Yang et al., Guangzhou, China. In Nan Fang Yi Ke Da Xue Xue Bao, Apr 2015
According to the genechip results, we selected 56 target genes related to PTH/nonPLC/PKC pathway, among which CITED1 showed higher expressions in [Gly(1), Arg(19)]hPTH(1-34)+ RP-cAMP group than in both the control group and [Gly(1), Arg(19)]hPTH(1-28)+RP-cAMP group (P<0.05), and its expression was the highest in PTH(1-34) group (P<0.05).
Loss of CITED1, an MITF regulator, drives a phenotype switch in vitro and can predict clinical outcome in primary melanoma tumours.
Jönsson et al., Lund, Sweden. In Peerj, 2014
CITED1 is a non-DNA binding transcriptional co-regulator whose expression can distinguish the 'proliferative' from 'invasive' signature in the phenotype-switching model of melanoma.
Isoproterenol Increases Uncoupling, Glycolysis, and Markers of Beiging in Mature 3T3-L1 Adipocytes.
Rayalam et al., Athens, United States. In Plos One, 2014
Lastly, markers of beige adipocytes (protein content of CD137 and gene transcript of CITED1) were also found to be upregulated at 48 hours of isoproterenol treatment.
Modifications of Human Subcutaneous ADMSC after PPARγ Activation and Cold Exposition.
Lizcano et al., Chía, Colombia. In Stem Cells Int, 2014
We found that adipocytes differentiated with total and partial agonists of PPARγ and exposed to 31°C are able to respond to cold significantly increasing the expression of thermogenic proteins such as UCP1, PGC1α, and CITED1, a marker of beige phenotype.
SIX2 and CITED1, markers of nephronic progenitor self-renewal, remain active in primitive elements of Wilms' tumor.
Lovvorn et al., Nashville, United States. In J Pediatr Surg, 2012
CITED1 expression remains active across all disease characteristics of Wilms tumor. It is found in the nucleus & cytosol. It is not found in normal kidney.
Defining and redefining the nephron progenitor population.
Little et al., Australia. In Pediatr Nephrol, 2011
This Six2(+)Cited1(+) population undergoes self-renewal throughout nephrogenesis while retaining the potential to epithelialize.
Organ-specific defects in insulin-like growth factor and insulin receptor signaling in late gestational asymmetric intrauterine growth restriction in Cited1 mutant mice.
de Caestecker et al., Nashville, United States. In Endocrinology, 2011
Loss of placental Cited1 leads to intrauterine organ growth restriction with decreased IGF-I and IGF-II expression and in IR and Igf1r signaling.
Aberrant promoter methylation in overexpression of CITED1 in papillary thyroid cancer.
Murata et al., Nagoya, Japan. In Thyroid, 2011
epigenetic regulation is involved in the overexpression of CITED1 in papillary thyroid cancer
Papillary thyroid carcinoma variants.
Khanafshar et al., Madison, United States. In Head Neck Pathol, 2011
Markers such as HBME-1 and CITED1 can assist in separating some difficult cases of follicular variants of papillary thyroid carcinomas from follicular adenomas.
TOX3 is a neuronal survival factor that induces transcription depending on the presence of CITED1 or phosphorylated CREB in the transcriptionally active complex.
Methner et al., Düsseldorf, Germany. In J Cell Sci, 2011
TOX3 induces transcription in neuron depending on the presence of CITED1 or phosphorylated CREB in the transcriptionally active complex.
Placental insufficiency associated with loss of Cited1 causes renal medullary dysplasia.
de Caestecker et al., Sydney, Australia. In J Am Soc Nephrol, 2009
Renal medullary dysplasia in Cited1 mutant mice is a direct consequence of decreased tissue oxygenation resulting from placental insufficiency.
Do benign thyroid nodules have malignant potential? An evidence-based review.
Fahey et al., New York City, United States. In World J Surg, 2008
In addition, both cytologic and molecular tumor markers, including Gal-3, CITED1, HBME-1, Ras, RET/PTC, and PAX8/PPAR gamma, have been identified in some histopathologically classified benign nodules.
The role of immunohistochemical markers in the diagnosis of follicular-patterned lesions of the thyroid.
Asa, Toronto, Canada. In Endocr Pathol, 2004
They include HBME-1, high molecular weight cytokeratins and ret, galectin-3 and TPO, galectin-3, fibronectin-1, CITED-1, HBME-1, and CK19.
Papillary and follicular thyroid carcinomas show distinctly different microarray expression profiles and can be distinguished by a minimum of five genes.
Eng et al., Columbus, United States. In J Clin Oncol, 2004
PTCs showed overexpression of CITED1, claudin-10 (CLDN10), and insulin-like growth factor binding protein 6 (IGFBP6) but showed no change in expression of caveolin-1 (CAV1) or -2 (CAV2); conversely, FTCs did not express CLDN10 and had decreased expression of IGFBP6 and/or CAV1 and CAV2.
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