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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

Nerve growth factor receptor

Bex, NADE, p75NTR-associated cell death executor, Bex3, NGFRAP1
Top mentioned proteins: CAN, ACID, Bex1, NGF, p75
Papers on Bex
The proteins interacting with C-terminal of μ receptor are identified by bacterial two-hybrid system from brain cDNA library in morphine-dependent rats.
Gong et al., Beijing, China. In Life Sci, Jan 2016
Column overlay assays, immunocytochemistry and coimmunoprecipitation were used to demonstrate the interaction of MOR-C and p75NTR-associated cell death executor (NADE).
Brain-Expressed X-linked (BEX) proteins in human cancers.
Rönnstrand et al., Lund, Sweden. In Biochim Biophys Acta, Dec 2015
The Brain-Expressed X-linked (BEX) family proteins are comprised of five human proteins including BEX1, BEX2, BEX3, BEX4 and BEX5.
Effects of oocyte donor age and embryonic stage of development on transcription of genes coding for enzymes of the prostaglandins and progesterone synthesis pathways in bovine in vitro produced embryos.
Lopes-da-Costa et al., Lisbon, Portugal. In Zygote, Dec 2015
Day 7 quality grade 1-2 embryos were selected for RNA extraction and gene transcription analysis by qRT-PCR, in a 2 × 2 factorial design [age (PP or C) × embryonic stage (compact morulae and early blastocysts, CM + EBL, or blastocysts and expanded blastocysts, BL + BEX); 15 embryos/group].
Bex3 Dimerization Regulates NGF-Dependent Neuronal Survival and Differentiation by Enhancing trkA Gene Transcription.
Arévalo et al., Salamanca, Spain. In J Neurosci, Jun 2015
Here, we show that rat Bex3 protein specifically regulates TrkA expression by acting at the trkA gene promoter level.
Combined sub-optimal doses of rosuvastatin and bexarotene impair angiotensin II-induced arterial mononuclear cell adhesion through inhibition of Nox5 signaling pathways and increased RXR/PPARα and RXR/PPARγ interactions.
Sanz et al., Valencia, Spain. In Antioxid Redox Signal, May 2015
Rosuvastatin (Rosu) and bexarotene (Bex) exert anti-inflammatory activity, but serious dose-related adverse effects have emerged.
Biophysical Studies on BEX3, the p75NTR-Associated Cell Death Executor, Reveal a High-Order Oligomer with Partially Folded Regions.
Almeida et al., Rio de Janeiro, Brazil. In Plos One, 2014
The pro-apoptotic activity of BEX3 in association with a number of additional proteins has been widely supported; however, to the best of our knowledge, very limited data are available on the conformation of any of the members of the BEX family.
Brain expressed and X-linked (Bex) proteins are intrinsically disordered proteins (IDPs) and form new signaling hubs.
Vilar et al., Madrid, Spain. In Plos One, 2014
The family of Brain Expressed and X-linked (Bex) proteins consists of 5 members in humans (Bex1-5).
Amyloid-β pathology and APOE genotype modulate retinoid X receptor agonist activity in vivo.
LaDu et al., Chicago, United States. In J Biol Chem, 2014
Previous data demonstrate that bexarotene (Bex), retinoid X receptor (RXR) agonist, reduces soluble and insoluble amyloid-β (Aβ) in Alzheimer disease (AD)-transgenic mice either by increasing the levels of mouse apolipoprotein E (apoE) or increasing ABCA1/ABCG1-induced apoE lipoprotein association/lipidation.
Gene targeting study reveals unexpected expression of brain-expressed X-linked 2 in endocrine and tissue stem/progenitor cells in mice.
Kamiya et al., Tokyo, Japan. In J Biol Chem, 2014
Genome-wide gene expression analyses in liver cells performed in this study have revealed a strong expression of X-linked genes that include members of the brain-expressed X-linked (Bex) gene family in stem/progenitor cells.
An emerging phenotype of Xq22 microdeletions in females with severe intellectual disability, hypotonia and behavioral abnormalities.
Stefanova et al., Tokyo, Japan. In J Hum Genet, 2014
The possible candidate region responsible for such distinctive features has been narrowed down to the neighboring region for PLP1, including the interleukin 1 receptor accessory protein-like 2 (IL1RAPL2) gene and the clustered brain expressed X-linked (BEX) genes.
Novel age-dependent targets in vestibular schwannomas.
Mehrian-Shai et al., Los Angeles, United States. In Hum Genomics, 2013
We confirmed these results by real-time PCR for SNF1LK2, NGFRAP1L1 (BEX 5), GMNN, and EPHA2.
Adenylating enzymes in Mycobacterium tuberculosis as drug targets.
Aldrich et al., Minneapolis, United States. In Curr Top Med Chem, 2011
Additionally, the enzymes NadE, GuaA, PanC, and MshC involved in the respective synthesis of NAD, guanine, pantothenate, and mycothiol will be discussed as well as BirA that is responsible for biotinylation of the acyl CoA-carboxylases.
[Expression and significance of p75NTR in dorsal root ganglia in different injury models].
Zhang et al., Changsha, China. In Zhong Nan Da Xue Xue Bao Yi Xue Ban, 2008
Expression of p75NTR increased in glia, but was downregulated in sensory neurons in sciatic nerve injury and spinal cord injury.
The tuberous sclerosis gene products hamartin and tuberin are multifunctional proteins with a wide spectrum of interacting partners.
Hengstschläger et al., Vienna, Austria. In Mutat Res, 2008
Besides tuberin, the proteins DOCK7, ezrin/radixin/moesin, FIP200, IKKbeta, Melted, Merlin, NADE(p75NTR), NF-L, Plk1 and TBC7 have been found to interact with hamartin.
The TSC1 gene product hamartin interacts with NADE.
Ohno et al., Yonago, Japan. In Mol Cell Neurosci, 2007
hamartin binds to NADE to regulate neuronal cell function and loss of this association is likely to contribute to the brain pathology in tuberous sclerosis complex
Characterization of human dopamine responsive protein DRG-1 that binds to p75NTR-associated cell death executor NADE.
Lu et al., Shanghai, China. In Brain Res, 2006
DRG-1 may contribute to the dopamine-induced cell growth, which is negatively regulated by NADE
Characterization of NADE, NRIF and SC-1 gene expression during mouse neurogenesis.
Verdi et al., West Scarborough, United States. In Brain Res Dev Brain Res, 2003
NADE mRNA level remained invariant across development.
Identification of members of the Bex gene family as olfactory marker protein (OMP) binding partners.
Margolis et al., Baltimore, United States. In J Neurochem, 2003
In situ hybridization demonstrates cellular co-localization of olfactory marker protein mRNA with mRNAs for Bex1, Bex2, and Bex3 in olfactory receptor neuronss in olfactory tissue of the mouse
Nerve growth factor-dependent regulation of NADE-induced apoptosis.
Sato et al., New York City, United States. In Vitam Horm, 2002
NADE (p75NTR-associated cell death executor) is a p75NTR-associated protein that mediates apoptosis in response to NGF by interacting with the cell death domain of p75NTR.
Exoenzyme S binds its cofactor 14-3-3 through a non-phosphorylated motif.
Hallberg, Umeå, Sweden. In Biochem Soc Trans, 2002
Recently, a phosphorylation-independent interaction has been reported to occur between 14-3-3 and a small number of proteins, for example the 43 kDa inositol polyphosphate 5-phosphatase, glycoprotein Ib, p75NTR-associated cell-death executor (NADE) and the bacterial ADP-ribosyltransferase toxin exoenzyme S (ExoS).
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