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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

Breast carcinoma amplified sequence 4

Top mentioned proteins: BCAS3, SET, CAN, HAD, erbB-2
Papers on BCAS4
Characterization of cellular and extracellular DNA in saliva.
Kibayashi et al., Tokyo, Japan. In Leg Med (tokyo), Nov 2015
The methylation patterns of the BCAS4 gene were different among donors, but no significant difference was observed between cellular and extracellular DNA.
Characterization of the genomic features and expressed fusion genes in micropapillary carcinomas of the breast.
Reis-Filho et al., London, United Kingdom. In J Pathol, 2014
Forced expression of in-frame fusion genes (SLC2A1-FAF1 and BCAS4-AURKA) resulted in increased viability of breast cancer cells.
Detecting and visualizing gene fusions.
Seifert et al., M√ľnchen, Germany. In Methods, 2013
We discuss several gene fusion events, like BCAS3-BCAS4 that was only detected in the breast cancer cell line MCF7.
The determination of tissue-specific DNA methylation patterns in forensic biofluids using bisulfite modification and pyrosequencing.
McCord et al., Miami, United States. In Electrophoresis, 2012
Among the different loci tested, we have identified a panel of markers, C20orf117, ZC3H12D, BCAS4, and FGF7, that can be used in the determination of these four tissue types.
Hypermethylated FAM5C and MYLK in serum as diagnosis and pre-warning markers for gastric cancer.
Liu et al., Shanghai, China. In Dis Markers, 2011
Eighty two hypermethylated genes were acquired by array analysis and 5 genes (BCAS4, CHRM2, FAM5C, PRAC and MYLK) were selected as the candidate genes.
Fusion gene microarray reveals cancer type-specificity among fusion genes.
Skotheim et al., Oslo, Norway. In Genes Chromosomes Cancer, 2011
Two additional fusion genes, BCAS4-BCAS3 in the MCF-7 breast cancer cell line and CCDC6-RET in the TPC-1 thyroid cancer cell line were validated as true positive fusion transcripts.
DNA profiling of primary serous ovarian and fallopian tube carcinomas with array comparative genomic hybridization and multiplex ligation-dependent probe amplification.
Dorsman et al., Amsterdam, Netherlands. In J Pathol, 2007
Other previously unappreciated gained/amplified genes included PSMB4 on 1q, MTSS1 on 8q, TEAD4 and TSPAN9 on 12p, and BCAS4 on 20q.
Multigene deletions on chromosome 20q13.13-q13.2 including SALL4 result in an expanded phenotype of Okihiro syndrome plus developmental delay.
Kohlhase et al., Freiburg, Germany. In Hum Mutat, 2007
In this patient, four genes (MOCS3, DPM1, ADNP, BCAS4) are deleted, which were not affected in the other three cases, suggesting that the deletion of one or more of these genes contributes to the mental retardation.
Fusion transcripts and transcribed retrotransposed loci discovered through comprehensive transcriptome analysis using Paired-End diTags (PETs).
Wei et al., Singapore, Singapore. In Genome Res, 2007
One was validated as the product of a fusion gene between BCAS4 and BCAS3 resulting from an amplification followed by a translocation event between the two loci, chr20q13 and chr17q23.
Cloning of BCAS3 (17q23) and BCAS4 (20q13) genes that undergo amplification, overexpression, and fusion in breast cancer.
Kallioniemi et al., Tampere, Finland. In Genes Chromosomes Cancer, 2002
BCAS3 (17q23) and BCAS4 (20q13) undergo amplification, overexpression, and fusion in breast cancer.
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