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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

Brain-specific angiogenesis inhibitor 2

BAI2, brain-specific angiogenesis inhibitor 2
BAI1, a p53-target gene, encodes brain-specific angiogenesis inhibitor, a seven-span transmembrane protein and is thought to be a member of the secretin receptor family. Brain-specific angiogenesis proteins BAI2 and BAI3 are similar to BAI1 in structure, have similar tissue specificities and may also play a role in angiogenesis. [provided by RefSeq, Jul 2008] (from NCBI)
Top mentioned proteins: brain-specific angiogenesis inhibitor 1, BAI3, POLYMERASE, p53, ACID
Papers on BAI2
Identification of biomarkers for metastatic osteosarcoma based on DNA microarray data.
Wang, In Neoplasma, 2014
DEGs with high degrees (brain-specific angiogenesis inhibitor 2 (BAI2), formin-like 1 (FMNL1), dual-specificity phosphatase 7 (DUSP7), transient receptor potential melastatin 2 (TRPM2), CBP80/20-dependent translation initiation factor (KIAA0427) and C120rf35) in the differential coexpression network were found.
International Union of Basic and Clinical Pharmacology. XCIV. Adhesion G protein-coupled receptors.
Schiöth et al., Amsterdam, Netherlands. In Pharmacol Rev, 2014
The new names, with old and alternative names within parentheses, are: ADGRA1 (GPR123), ADGRA2 (GPR124), ADGRA3 (GPR125), ADGRB1 (BAI1), ADGRB2 (BAI2), ADGRB3 (BAI3), ADGRC1 (CELSR1), ADGRC2 (CELSR2), ADGRC3 (CELSR3), ADGRD1 (GPR133), ADGRD2 (GPR144), ADGRE1 (EMR1, F4/80), ADGRE2 (EMR2), ADGRE3 (EMR3), ADGRE4 (EMR4), ADGRE5 (CD97), ADGRF1 (GPR110), ADGRF2 (GPR111), ADGRF3 (GPR113), ADGRF4 (GPR115), ADGRF5 (GPR116, Ig-Hepta), ADGRG1 (GPR56), ADGRG2 (GPR64, HE6), ADGRG3 (GPR97), ADGRG4 (GPR112), ADGRG5 (GPR114), ADGRG6 (GPR126), ADGRG7 (GPR128), ADGRL1 (latrophilin-1, CIRL-1, CL1), ADGRL2 (latrophilin-2, CIRL-2, CL2), ADGRL3 (latrophilin-3, CIRL-3, CL3), ADGRL4 (ELTD1, ETL), and ADGRV1 (VLGR1, GPR98).
Epigenetic genome-wide analysis identifies BEX1 as a candidate tumour suppressor gene in paediatric intracranial ependymoma.
Warr et al., London, United Kingdom. In Cancer Lett, 2014
The most frequently methylated genes were BEX1 (27/40 cases), BAI2 (20/40), CCND2 (18/40), and CDKN2A (14/40).
Identification of brain-specific angiogenesis inhibitor 2 as an interaction partner of glutaminase interacting protein.
Mohanty et al., İzmir, Turkey. In Biochem Biophys Res Commun, 2011
This is the first study reporting BAI2 as an interaction partner of GIP.
Antidepressant-like behavior in brain-specific angiogenesis inhibitor 2-deficient mice.
Yokota et al., Tokyo, Japan. In J Physiol Sci, 2011
Brain-specific angiogenesis inhibitor 2 (BAI2) is a transmembrane protein that is predominantly expressed in the brain.
FRET-based biosensors for the detection and quantification of AI-2 class of quorum sensing compounds.
Sayre et al., United States. In Methods Mol Biol, 2010
LuxP-BAI2 binding induces changes in fluorescence resonance energy transfer (FRET) between CFP and YFP, whose magnitude of change is ligand concentration dependent.
Brain-specific angiogenesis inhibitor 2 (BAI2) may be activated by proteolytic processing.
Yokota et al., Tokyo, Japan. In J Recept Signal Transduct Res, 2010
BAI2 is a functional GPCR regulated by proteolytic processing and activates the NFAT pathway
Anatomical and histological profiling of orphan G-protein-coupled receptor expression in gastrointestinal tract of C57BL/6J mice.
Tokita et al., Tsukuba, Japan. In Cell Tissue Res, 2009
GPR85, GPR37, GPR37L1, brain-specific angiogenesis inhibitor (BAI) 1, BAI2, BAI3, and GPRC5B mRNAs were preferentially expressed in the muscle-myenteric nerve layer, similar to GPCRs that are expressed in both the central and enteric nerve systems and that play multiple regulatory roles throughout the gut-brain axis.
cDNA microarray analysis of pigment epithelium-derived factor-regulated gene expression profile in prostate carcinoma cells.
Lu et al., Shanghai, China. In Int J Urol, 2009
Real-time reverse transcription polymerase chain reaction, in accordance with the microarray analysis, indicated that PEDF treatment caused an upregulation in the mRNA expression level of stanniocalcin 2, brain-specific angiogenesis inhibitor 2 and growth arrest, DNA-damage-inducible, alpha, and downregulation in the messenger ribonucleic acid level of fibroblast growth factor 3, teratocarcinoma-derived growth factor, neuropilin1, and endothelial Per/ARNT/Sim domain protein1, respectively.
Supersilylated tetraphosphene derivatives M2[t-Bu3SiPPPPSi-t-Bu3] (M = Li, Na, Rb, Cs) and Ba[t-Bu3SiPPPPSi-t-Bu3]: reactivity and cis-trans isomerization.
Lerner et al., Frankfurt am Main, Germany. In Inorg Chem, 2009
The reaction of Na2[t-Bu3SiPPPPSi-t-Bu3] with BaI2 gave, conveniently, the corresponding barium derivative Ba[t-Bu3SiPPPPSi-t-Bu3].
Gene expression profiling: canonical molecular changes and clinicopathological features in sporadic colorectal cancers.
Kim et al., Seoul, South Korea. In World J Gastroenterol, 2008
Amongst 47 differentially expressed genes, seven (PISD, NIBP, BAI2, STOML1, MRPL21, MRPL16, and MKKS) were newly found to correlate with tumorigenesis and tumor growth.
Syntheses, structure, and a Mössbauer and magnetic study of Ba(4)Fe(2)I(5)S(4).
Ibers et al., Evanston, United States. In Inorg Chem, 2008
The compound Ba4Fe2I5S4 has been prepared at 1223-1123 K by the "U-assisted" reaction of FeS, BaS, S, and U with BaI2 as a flux.
Recent advances in tailoring the aggregation of heavier alkaline earth metal halides, alkoxides and aryloxides from non-aqueous solvents.
Fromm, Basel, Switzerland. In Dalton Trans, 2006
The first concept deals with the "cutting out" of structural fragments from a solid state structure of a binary compound, which will be explained with reference to BaI2.
Proteomic analysis of the tetraspanin web using LC-ESI-MS/MS and MALDI-FTICR-MS.
Le Naour et al., Villejuif, France. In Proteomics, 2006
Different types of membrane proteins were identified, including adhesion molecules (integrins, Lu/B-CAM, GA733 proteins), receptors and signaling molecules (BAI2, PKC, G proteins), proteases (ADAM10, TADG15), and membrane fusion proteins (syntaxins) as well as poorly characterized proteins (CDCP1, HEM-1, CTL1, and CTL2).
Brain-specific angiogenesis inhibitor 2 regulates VEGF through GABP that acts as a transcriptional repressor.
Kim et al., Kwangju, South Korea. In Febs Lett, 2006
Results point out that brain-specific angiogenesis inhibitor 2 controls VEGF transcription through GA-binding protein under normal conditions and cerebral ischemia.
Expression of brain-specific angiogenesis inhibitor 2 (BAI2) in normal and ischemic brain: involvement of BAI2 in the ischemia-induced brain angiogenesis.
Kim et al., Kwangju, South Korea. In J Cereb Blood Flow Metab, 2002
involvement of BAI2 in the ischemia-induced brain angiogenesis
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