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Arylsulfatase family, member J

Sulfatases (EC, such as ARSJ, hydrolyze sulfate esters from sulfated steroids, carbohydrates, proteoglycans, and glycolipids. They are involved in hormone biosynthesis, modulation of cell signaling, and degradation of macromolecules (Sardiello et al., 2005 [PubMed 16174644]).[supplied by OMIM, Mar 2008] (from NCBI)
Top mentioned proteins: CD55, Insulin, CAN, ASK, ACID
Papers on ASJ
Environmental CO2 inhibits Caenorhabditis elegans egg-laying by modulating olfactory neurons and evokes widespread changes in neural activity.
de Bono et al., Cambridge, United Kingdom. In Proc Natl Acad Sci U S A, Aug 2015
Many of the C. elegans sensory neurons we examined, including the AWC olfactory neurons, the ASJ and ASK gustatory neurons, and the ASH and ADL nociceptors, respond to a rise in CO2 with a rise in Ca(2+).
Cis- and trans-regulatory mechanisms of gene expression in the ASJ sensory neuron of Caenorhabditis elegans.
Miranda-Vizuete et al., Sevilla, Spain. In Genetics, May 2015
Here, we identify cis and trans regulatory elements that drive gene expression in the bilateral sensory neuron ASJ, located in the head of the nematode Caenorhabditis elegans.
Ins-4 and daf-28 function redundantly to regulate C. elegans L1 arrest.
Baugh et al., Durham, United States. In Dev Biol, 2014
Expression of candidate agonists is largely overlapping in L1 stage larvae, suggesting a role of the intestine, chemosensory neurons ASI and ASJ, and the interneuron PVT in control of L1 development.
Chemosensation of bacterial secondary metabolites modulates neuroendocrine signaling and behavior of C. elegans.
Kim et al., Cambridge, United States. In Cell, 2014
Secondary metabolites phenazine-1-carboxamide and pyochelin activate a G-protein-signaling pathway in the ASJ chemosensory neuron pair that induces expression of the neuromodulator DAF-7/TGF-β.
Light and pheromone-sensing neurons regulates cold habituation through insulin signalling in Caenorhabditis elegans.
Kuhara et al., Kōbe, Japan. In Nat Commun, 2013
Here we show that light and pheromone-sensing neurons (ASJ) regulate cold habituation through insulin signalling.
Chemical noise produced by equilibrium adsorption/desorption of surface pyridine at Au-Ag-Au bimetallic atom-scale junctions studied by fluctuation spectroscopy.
Bohn et al., United States. In J Am Chem Soc, 2013
Concentration-dependent adsorption free energies are interpreted as arising from a distribution of binding energies, N(E(b)), on the Ag ASJ.
CNP-1 (ARRD-17), a novel substrate of calcineurin, is critical for modulation of egg-laying and locomotion in response to food and lysine sensation in Caenorhabditis elegans.
Lee et al., Kwangju, South Korea. In J Mol Biol, 2012
cnp-1 is expressed in ASK, ADL, ASH and ASJ sensory neurons as TAX-6.
The thioredoxin TRX-1 regulates adult lifespan extension induced by dietary restriction in Caenorhabditis elegans.
Swoboda et al., Huddinge, Sweden. In Biochem Biophys Res Commun, 2011
A trx-1::GFP translational fusion expressed from its own promoter in ASJ neurons (Ptrx-1::trx-1::GFP) rescued the trx-1 deletion-mediated suppression of the lifespan extension caused by mutation of eat-2.
Optogenetic long-term manipulation of behavior and animal development.
Gottschalk et al., Frankfurt am Main, Germany. In Plos One, 2010
Finally, we could optically induce profound changes in animal development: Long-term photoactivation of ASJ neurons, which regulate larval growth, bypassed the constitutive entry into the "dauer" larval state in daf-11 mutants.
The thioredoxin TRX-1 modulates the function of the insulin-like neuropeptide DAF-28 during dauer formation in Caenorhabditis elegans.
Swoboda et al., Huddinge, Sweden. In Plos One, 2010
We report that the C. elegans thioredoxin TRX-1 acts in ASJ head sensory neurons as a novel modulator of the insulin-like neuropeptide DAF-28 during dauer formation.
HIF-1 antagonizes p53-mediated apoptosis through a secreted neuronal tyrosinase.
Hengartner et al., Zürich, Switzerland. In Nature, 2010
The antiapoptotic property of HIF-1 is mediated by means of transcriptional upregulation of the tyrosinase family member TYR-2 in the ASJ sensory neurons.
C. elegans phototransduction requires a G protein-dependent cGMP pathway and a taste receptor homolog.
Xu et al., Ann Arbor, United States. In Nat Neurosci, 2010
By recording the photoreceptor neuron ASJ in wild-type and various mutant worms, we found that phototransduction in ASJ is a G protein-mediated process and requires membrane-associated guanylate cyclases, but not typical phosphodiesterases.
Investigation of the regulation of transcriptional changes in Ancylostoma caninum larvae following serum activation, with a focus on the insulin-like signalling pathway.
Gasser et al., Australia. In Vet Parasitol, 2009
The exit from dauer in the free-living nematode Caenorhabditis elegans is under the control of a single amphidial neuron (ASJ) of the insulin-like signalling pathway.
Sulfated signal from ASJ sensory neurons modulates stomatin-dependent coordination in Caenorhabditis elegans.
Morgan et al., Cleveland, United States. In J Biol Chem, 2006
Whereas unc-1 is expressed in the majority of neurons of C. elegans, expression of SSU-1 protein in only the two ASJ amphid interneurons is sufficient to restore the wild type phenotype.
Molecular cloning and initial characterization of three novel human sulfatases.
Obaya, Oviedo, Spain. In Gene, 2006
The identification, molecular cloning and initial characterization of three new members of this human gene family is reported.
Control of larval development by chemosensory neurons in Caenorhabditis elegans.
Horvitz et al., Cambridge, United States. In Science, 1991
When the neuron classes ADF, ASG, ASI, and ASJ are killed, animals develop as dauer larvae regardless of environmental conditions.
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