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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Dec 2016.

Bromodomain adjacent to zinc finger domain, 1A

ACF1, ATP-dependent chromatin remodeling complex, ATP-utilizing chromatin assembly and remodeling factor
The BAZ1A gene encodes the accessory subunit of the ATP-dependent chromatin assembly factor (ACF), a member of the ISWI ('imitation switch') family of chromatin remodeling complexes (summarized by Racki et al., 2009 [PubMed 20033039]).[supplied by OMIM, Apr 2010] (from NCBI)
Top mentioned proteins: SWI, Histone, ATPase, CAN, BRG1
Papers on ACF1
Akirin interacts with Bap60 and 14-3-3 proteins to regulate the expression of antimicrobial peptides in the kuruma shrimp (Marsupenaeus japonicus).
Wang et al., Jinan, China. In Dev Comp Immunol, Feb 2016
The limited regulating spectrum of Akirin might be associated with Bap60, a component of the Brahma (SWI/SNF) ATP-dependent chromatin-remodeling complex.
INO80 Chromatin Remodeler Facilitates Release of RNA Polymerase II from Chromatin for Ubiquitin-Mediated Proteasomal Degradation.
Papamichos-Chronakis et al., Paris, France. In Mol Cell, Jan 2016
Here, we report that the ATP-dependent chromatin remodeling complex INO80 is required for turnover of chromatin-bound RNAPII in yeast.
Backbone and side-chain NMR assignments for the bromodomain of mouse BAZ1A (ACF1).
Yao et al., Nanjing, China. In Biomol Nmr Assign, Dec 2015
UNASSIGNED: BAZ1A, a non-catalytic subunit of the chromatin remodeler complexes ACF and CHRAC, is thought to modulate the ATPase's activity of the complexes and participate in gene transcription, DNA damage checkpoint and double-strand break repair.
ACF chromatin-remodeling complex mediates stress-induced depressive-like behavior.
Nestler et al., New York City, United States. In Nat Med, Oct 2015
We establish that persistent upregulation of the ACF (ATP-utilizing chromatin assembly and remodeling factor) ATP-dependent chromatin-remodeling complex, occurring in the nucleus accumbens of stress-susceptible mice and depressed humans, is necessary for stress-induced depressive-like behaviors.
The yeast chromatin remodeler Rsc1-RSC complex is required for transcriptional activation of autophagy-related genes and inhibition of the TORC1 pathway in response to nitrogen starvation.
Tsuchiya et al., Hiroshima, Japan. In Biochem Biophys Res Commun, Oct 2015
The yeast RSC, an ATP-dependent chromatin-remodeling complex, is essential for mitotic and meiotic growth.
Development of a PCR Assay Based on a Single-Base Pair Substitution for the Detection of Aeromonas caviae by Targeting the gyrB Gene.
Chaivisuthangkura et al., Bangkok, Thailand. In J Aquat Anim Health, Sep 2015
The technique was able to detect 100% (29/29) of the A. caviae strains tested using either of two sets of primers (designated ACF1-ACR and ACF3-ACR), which produced 293-bp and 206-bp amplicons, respectively.
Inactivation of yeast Isw2 chromatin remodeling enzyme mimics longevity effect of calorie restriction via induction of genotoxic stress response.
Berger et al., Philadelphia, United States. In Cell Metab, 2014
We show that lifespan is also extended in C. elegans by reducing levels of athp-2, a putative ortholog of Itc1/ACF1, a critical subunit of the enzyme complex.
Molecular architecture of the ATP-dependent chromatin-remodeling complex SWR1.
Leschziner et al., Cambridge, United States. In Cell, 2013
The ATP-dependent chromatin-remodeling complex SWR1 exchanges a variant histone H2A.Z/H2B dimer for a canonical H2A/H2B dimer at nucleosomes flanking histone-depleted regions, such as promoters.
Heterozygous missense mutations in SMARCA2 cause Nicolaides-Baraitser syndrome.
Vermeesch et al., Leuven, Belgium. In Nat Genet, 2012
SMARCA2 encodes the core catalytic unit of the SWI/SNF ATP-dependent chromatin remodeling complex that is involved in the regulation of gene transcription.
Role for hACF1 in the G2/M damage checkpoint.
Becker et al., München, Germany. In Nucleic Acids Res, 2011
hACF1-containing factors are more generally involved in the DNA damage response.
Binding kinetics of human ISWI chromatin-remodelers to DNA repair sites elucidate their target location mechanism.
Rippe et al., Heidelberg, Germany. In Nucleus, 2011
ISWI proteins Snf2H, Snf2L as well as Acf1 accumulate at UV-induced DNA damage sites within tens of seconds and reach a plateau after a few minutes.
The ACF1 complex is required for DNA double-strand break repair in human cells.
Yasui et al., Sendai, Japan. In Mol Cell, 2011
The ACF1 and its complexes play important roles in DNA double-strand breaks (DSBs) repair.
Williams syndrome is an epigenome-regulator disease.
Kato et al., Tokyo, Japan. In Endocr J, 2010
This novel ATP-dependent chromatin remodeling complex facilitates VDR-mediated transrepression as well as transactivation with its ATP-dependent chromatin remodeling activity and promoter targeting property for the activator to access to the DNA.
SWI/SNF complexes, chromatin remodeling and skeletal myogenesis: it's time to exchange!
Puri et al., Los Angeles, United States. In Exp Cell Res, 2010
This review focuses on our current understanding of the role of the SWI/SNF ATP-dependent chromatin-remodeling complex in the activation of the myogenic program, by inducing conformational changes permissive for muscle-gene expression.
Developmental role for ACF1-containing nucleosome remodellers in chromatin organisation.
Becker et al., München, Germany. In Development, 2010
Altering the levels of ACF1 leads to global and variegated deviations from normal chromatin organisation with pleiotropic defects.
Mi-2/NuRD complex making inroads into DNA-damage response pathway.
Kumar et al., Washington, D.C., United States. In Cell Cycle, 2010
Beyond their well-established role in transcription, histone modifications and several classes of ATP-dependent chromatin-remodeling complex have been functionally linked to efficient DNA repair.
[ATP-dependent chromatin remodeling complex and its function in regulating chromatin structure].
Zeng et al., Changchun, China. In Yi Chuan, 2010
Recently, it is widely accepted that chromatin remodeling is carried out by at least two mechanisms: ATP-dependent chromatin remodeling complex and covalent modifications of histone tails by histone modification complexes.
Chromatin remodelers act globally, sequence positions nucleosomes locally.
Narlikar et al., San Francisco, United States. In J Mol Biol, 2009
The data support a model in which the human ACF anf yeast RSC remodeling enzymes move nucleosomes to new locations by a general sequence-independent mechanism.
The role of INI1/hSNF5 in gene regulation and cancer.
Penn et al., Toronto, Canada. In Biochem Cell Biol, 2009
In this review, we discuss a core subunit of the SWI/SNF ATP-dependent chromatin remodeling complex, known as INI1/hSNF5, in the context of transcriptional regulation and impact on cancer biology.
Newly identified prion linked to the chromatin-remodeling factor Swi1 in Saccharomyces cerevisiae.
Li et al., Chicago, United States. In Nat Genet, 2008
SWI/SNF, an evolutionarily conserved ATP-dependent chromatin-remodeling complex, has an important role in transcriptional regulation.
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