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Zinc finger protein 202

ZNF202, zinc finger protein 202
Top mentioned proteins: HDL, ACID, SP1, CAN, SDP1
Papers on ZNF202
Sample-to-SNP kit: a reliable, easy and fast tool for the detection of HFE p.H63D and p.C282Y variations associated to hereditary hemochromatosis.
Refstrup et al., Copenhagen, Denmark. In Gene, 2012
These SNPs were: Coagulation factor II-gene F2 c.20210G>A, Coagulation factor V-gene F5 p.R506Q (c.1517G>A; rs121917732), Mitochondria SNP: mt7028 G>A, Mitochondria SNP: mt12308 A>G, Proprotein convertase subtilisin/kexin type 9-gene PCSK9 p.R46L (c.137G>T), Plutathione S-transferase pi 1-gene GSTP1 p.I105V (c313A>G; rs1695), LXR g.-171 A>G, ZNF202 g.-118 G>T.
Promoter alteration causes transcriptional repression of the POMGNT1 gene in limb-girdle muscular dystrophy type 2O.
Cruces et al., Madrid, Spain. In Eur J Hum Genet, 2012
Analysis of the downstream effects of this mutation revealed a decrease in the expression of POMGNT1 mRNA and protein because of negative regulation of the POMGNT1 promoter by the transcription factor ZNF202 (zinc-finger protein 202).
Population-Based Resequencing of LIPG and ZNF202 Genes in Subjects with Extreme HDL Levels.
Kamboh et al., Aurora, United States. In Front Genet, 2011
Endothelial lipase (LIPG) and zinc finger protein 202 (ZNF202) are two pivotal genes in high density lipoprotein (HDL metabolism).
Interleukin-18 and interleukin-12 together downregulate ATP-binding cassette transporter A1 expression through the interleukin-18R/nuclear factor-κB signaling pathway in THP-1 macrophage-derived foam cells.
Tang et al., Hengyang, China. In Circ J, 2011
Treatment with IL-18 plus IL-12 markedly augmented nuclear translocation of nuclear factor (NF)-κB but had no effect on expression and activity of liver X receptor α. IL-18 and IL-12 also significantly increased zinc finger protein 202 (ZNF202) levels and IFN-γ secretion.
Genomewide association study of movement-related adverse antipsychotic effects.
van den Oord et al., Richmond, United States. In Biol Psychiatry, 2010
RESULTS: Two SNPs for SAS, rs17022444 and rs2126709 with p = 1.2 x 10(-10) and p = 3.8 x 10(-7), respectively, and one for AIMS, rs7669317 with p = 7.7 x 10(-8), reached genomewide significance (Q value < .1). rs17022444 and rs7669317 were located in intergenic regions and rs2126709 was located in ZNF202 on 11q24.
Deletion hotspots in AMACR promoter CpG island are cis-regulatory elements controlling the gene expression in the colon.
Ho et al., Cincinnati, United States. In Plos Genet, 2009
The DNA sequences housing these deletions were found to be putative cis-regulatory elements for Sp1 at CG3 and CG10, and ZNF202 at CG12-16.
SOX17 directly activates Zfp202 transcription during in vitro endoderm differentiation.
Gearhart et al., Baltimore, United States. In Physiol Genomics, 2008
SOX17-Chromatin immunoprecipitation identified zinc finger protein 202 (Zfp202) as a direct target of SOX17 during endoderm differentiation of F9 embryonal carcinoma cells.
Functional promoter variant in zinc finger protein 202 predicts severe atherosclerosis and ischemic heart disease.
Tybjaerg-Hansen et al., Copenhagen, Denmark. In J Am Coll Cardiol, 2008
Homozygosity for a common functional promoter variant in ZNF202 predicts severe atherosclerosis and an increased risk of IHD.
Zinc Finger Protein 202: a new candidate gene for ischemic heart disease: The Copenhagen City Heart Study.
Tybjaerg-Hansen et al., Copenhagen, Denmark. In Atherosclerosis, 2006
This is the first study to suggest that ZNF202 could be a new candidate gene for ischemic heart disease and myocardial infarction in the general population.
High throughput SNP and expression analyses of candidate genes for non-syndromic oral clefts.
Beaty et al., Baltimore, United States. In J Med Genet, 2006
RESULTS: Thirteen candidate genes showed significant evidence of linkage in the presence of disequilibrium, and ten of these were found to be expressed in relevant embryonic tissues: SP100, MLPH, HDAC4, LEF1, C6orf105, CD44, ALX4, ZNF202, CRHR1, and MAPT.
Zinc Finger Protein 202, genetic variation, and HDL cholesterol in the general population.
Tybjaerg-Hansen et al., Copenhagen, Denmark. In J Lipid Res, 2006
findings show that genetic variation in ZNF202 is common in the general population. However, SNPs in the protein-coding region of ZNF202 do not make a major contribution to HDL cholesterol levels.
Functional zinc finger/sleeping beauty transposase chimeras exhibit attenuated overproduction inhibition.
George et al., Nashville, United States. In Febs Lett, 2005
We engineered and tested chimeric SB transposases with two different human zinc finger DNA binding domain elements, Sp1 and zinc finger 202 (ZNF202).
Transcriptional regulatory networks in lipid metabolism control ABCA1 expression.
Langmann et al., Regensburg, Germany. In Biochim Biophys Acta, 2005
Shutdown of ABCA1 expression in the absence of sterols or in certain tissues is mediated by corepressor complexes involving unliganded LXR, sterol-regulatory element binding protein 2 (SREBP2), Sp3, and the SCAN-domain protein ZNF202, which also impacts nuclear receptor signaling.
Screening for functional sequence variations and mutations in ABCA1.
Schmitz et al., Regensburg, Germany. In Atherosclerosis, 2004
In the promoter region, which is important for regulation of gene expression, we have identified several polymorphisms including one VNTR polymorphism, located at a putative ZNF202 binding site, which displayed different binding of ZNF202 in an electromobility shift assay.
Zinc finger protein ZNF202 structure and function in transcriptional control of HDL metabolism.
Langmann et al., Regensburg, Germany. In Curr Opin Lipidol, 2004
PURPOSE OF REVIEW: The zinc finger protein ZNF202 is a transcriptional repressor controlling promoter elements predominantly found in genes involved in lipid metabolism and energy homeostasis.
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