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Zinc finger protein 24

ZNF191, zinc finger protein 191, Zfp191, ZNF24, ZF-12
putative zinc finger protein [RGD, Feb 2006] (from NCBI)
Top mentioned proteins: CAN, vascular endothelial growth factor, ZFP, fibrillin-1, GST
Papers on ZNF191
Novel endogenous angiogenesis inhibitors and their therapeutic potential.
Ge et al., Singapore, Singapore. In Acta Pharmacol Sin, Oct 2015
In this review, we highlight ten novel endogenous protein angiogenesis inhibitors discovered within the last five years, including ISM1, FKBPL, CHIP, ARHGAP18, MMRN2, SOCS3, TAp73, ZNF24, GPR56 and JWA.
MicroRNA-940 promotes tumor cell invasion and metastasis by downregulating ZNF24 in gastric cancer.
Li et al., Shanghai, China. In Oncotarget, Oct 2015
Mechanistically, we found that miR-940 promoted GC cell migration, invasion, and metastasis in vivo by directly and functionally repressing the expression of Zinc Finger Transcription Factor 24 (ZNF24).
The endogenous zinc finger transcription factor, ZNF24, modulates the angiogenic potential of human microvascular endothelial cells.
Moses et al., Boston, United States. In Faseb J, Apr 2015
We have previously identified a zinc finger transcription factor, ZNF24 (zinc finger protein 24), as a novel inhibitor of tumor angiogenesis and have demonstrated that ZNF24 exerts this effect by repressing the transcription of VEGF in breast cancer cells.
Recognition Code of ZNF191(243-368) and Its Interaction with DNA.
Huang et al., Zhengzhou, China. In Bioinorg Chem Appl, 2014
ZNF191(243-368) is the C-terminal region of ZNF191 which contains a putative DNA-binding domain of four Cys2His2 zinc finger motifs.
Exploring causal networks underlying fat deposition and muscularity in pigs through the integration of phenotypic, genotypic and transcriptomic data.
Rosa et al., Madison, United States. In Bmc Syst Biol, 2014
One genomic region on SSC6 showed significant associations with three relevant phenotypes, off-midline10th-rib backfat thickness, loin muscle weight, and average intramuscular fat percentage, and also with the expression of seven genes, including ZNF24, SSX2IP, and AKR7A2.
Expression of ZNF396 in basal cell carcinoma.
Takeuchi et al., Gifu, Japan. In Arch Dermatol Res, 2014
Zfp191 represses differentiation and keeps various cells in the stem/progenitor stage.
Zinc finger protein 191 deficiency attenuates vascular smooth muscle cell proliferation, migration, and intimal hyperplasia after endovascular arterial injury.
Zhang et al., Shanghai, China. In J Vasc Surg, 2014
Zinc finger protein 191 (ZFP191) is a novel member of the SCAN domain family of Krüppel-like zinc finger transcription factors.
Nbn gene inactivation in the CNS of mouse inhibits the myelinating ability of the mature cortical oligodendrocytes.
Tong et al., Beijing, China. In Glia, 2014
In addition, a series of transcriptional factors including histonedeacetylase (HDAC), zinc finger protein 191 (ZFP-191) and myelin sheath regulatory factor (MRF) play distinct roles in regulating the myelination of the Nbn-deficient oligodendrocytes.
A proteomic characterization of factors enriched at nascent DNA molecules.
Fernandez-Capetillo et al., Madrid, Spain. In Cell Rep, 2013
For instance, our data support a link between DNA replication and the Williams-Beuren syndrome and identify ZNF24 as a replication factor.
Transcriptional repression of VEGF by ZNF24: mechanistic studies and vascular consequences in vivo.
Moses et al., Boston, United States. In Blood, 2013
We have previously reported the identification of a SCAN domain-containing C2H2 zinc finger protein, ZNF24, that represses the transcription of VEGF.
Identification of a functional nuclear localization signal mediating nuclear import of the zinc finger transcription factor ZNF24.
Zhang et al., Shanghai, China. In Plos One, 2012
ZNF24 is a member of the SCAN domain family of Krüppel-like zinc finger (ZF) transcription factors, which plays a critical role in cell proliferation and differentiation.
Unbiased discovery of interactions at a control locus driving expression of the cancer-specific therapeutic and diagnostic target, mesothelin.
Kern et al., Baltimore, United States. In J Proteome Res, 2012
Among other identified candidates, we found functional roles for ZNF24, NAB1 and RFX1 in MSLN expression by cancer cells.
A yeast two-hybrid screen identifies histone H2A.Z as a transcription factor ZNF24 interactor.
Zhang et al., Shanghai, China. In J Cell Biochem, 2012
ZNF24 is a pleiotropic factor that has a role in transcription regulation, hematopoiesis, brain development, and cancers, but the molecular mechanisms underlying its functions are not clearly understood.
Zinc finger transcription factor 191, directly binding to β-catenin promoter, promotes cell proliferation of hepatocellular carcinoma.
Yu et al., Shanghai, China. In Hepatology, 2012
ZNF191 can directly bind to the CTNNB1 promoter and activate the expression of beta-catenin and its downstream target genes such as cyclin D1 in hepatoma cell lines.
Resistance to lipopolysaccharide-induced endotoxic shock in heterozygous Zfp191 gene-knockout mice.
Fei et al., Shanghai, China. In Genet Mol Res, 2010
The loss of Zfp191 may suppress systemic inflammation by reducing these cytokine levels during lipopolysaccharide-induced endotoxic shock.
The transcriptional repression of platelet-derived growth factor receptor-beta by the zinc finger transcription factor ZNF24.
Zhang et al., Shanghai, China. In Biochem Biophys Res Commun, 2010
These data suggest that ZNF24 is involved in negative regulation of PDGFR-beta and may represent a novel repressor of PDGFR-beta transcription.
ZFP191 is required by oligodendrocytes for CNS myelination.
Popko et al., Chicago, United States. In Genes Dev, 2010
this study demonstrates that ZFP191 is required for the myelinating function of differentiated oligodendrocytes.
Zinc finger protein 191 (ZNF191/Zfp191) is necessary to maintain neural cells as cycling progenitors.
Nardelli et al., Paris, France. In Stem Cells, 2009
Data confirm ZNF191 as an essential factor acting for the promotion of the cell cycle and thus maintenance in the progenitor stage.
Post-genomic era and gene discovery for psychiatric diseases: there is a new art of the trade? The example of the HUMTH01 microsatellite in the Tyrosine Hydroxylase gene.
Mallet et al., Paris, France. In Mol Neurobiol, 2002
Thereafter, they cloned two proteins (ZNF191 and HBP1), specifically binding to HUMTH01, and demonstrated that allelic variations of HUMTH01 have a quantitative silencing effect on TH gene expression in vitro, and correlate with quantitative and qualitative changes in the binding by ZNF191.
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