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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Aug 2016.

FGFR1 oncogene partner 2

wit3.0
Top mentioned proteins: Tec, Actin, CAN, OUT, POLYMERASE
Papers on wit3.0
High throughput screening of biologically functional small molecules for modulating the expression of FGFR1OP2/wit3.0 in fibroblasts.
Nishimura et al., Los Angeles, United States. In J Calif Dent Assoc, 2012
A small cytoskeleton molecule identified in oral wound fibroblasts, FGFR1OP2/wit3.0, has been shown to accelerate wound closure in vitro and in vivo.
Association between FGFR1OP2/wit3.0 polymorphisms and residual ridge resorption of mandible in Korean population.
Lee et al., Seoul, South Korea. In Plos One, 2011
BACKGROUND: A previous study on the genetic association between single nucleotide polymorphisms in FGFR1OP2/wit3.0 and the long term atrophy of edentulous mandible hypothesized that the excessive jawbone atrophy after dental extraction may be associated with abnormal oral mucosa contraction induced by the FGFR1OP2/wit 3.0 gene.
A genetic association study of single nucleotide polymorphisms in FGFR1OP2/wit3.0 and long-term atrophy of edentulous mandible.
Nishimura et al., Los Angeles, United States. In Plos One, 2010
FGFR1OP2/wit3.0, a cytoskeleton molecule initially identified in oral wound fibroblasts, has been shown to induce oral mucosa contraction after dental extraction.
Wound closure and wound management: A new therapeutic molecular target.
Nishimura et al., Los Angeles, United States. In Cell Adh Migr, 2010
(wit3.0)
Small cytoskeleton-associated molecule, fibroblast growth factor receptor 1 oncogene partner 2/wound inducible transcript-3.0 (FGFR1OP2/wit3.0), facilitates fibroblast-driven wound closure.
GeneRIF
Nishimura et al., Los Angeles, United States. In Am J Pathol, 2010
FGFR1OP2/wit3.0 may regulate cell motility and stimulate wound closure.
Oral fibroblast expression of wound-inducible transcript 3.0 (wit3.0) accelerates the collagen gel contraction in vitro.
Nishimura et al., Los Angeles, United States. In J Biol Chem, 2004
A differentially expressed cDNA, wound-inducible transcript 3.0 (wit3.0), was isolated from oral mucosal wound in rats (Sukotjo, C., Abanmy, A. A., Ogawa, T., and Nishimura, I. (2002) J. Dent.
Molecular cloning of wound inducible transcript (wit 3.0) differentially expressed in edentulous oral mucosa undergoing tooth extraction wound-healing.
GeneRIF
Nishimura et al., United States. In J Dent Res, 2002
expressed by the fibroblasts associated with tooth extraction wound-healing (wit 3.0)
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