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8 documents found
1: Title: Fas-Antisense Long Noncoding RNA and Acute Myeloid Leukemia: Is There any Relation?
Authors: Sayad, Arezou, et.al. .
Journal: Asian Pacific journal of cancer prevention : APJCP (Asian Pac J Cancer Prev), Vol. 19 (1): 45-48, 2018 .
Snippet: FAS has an important role in regulation of apoptotic pathways and there is an inverse correlation between FAS-AS1 expression level and production of the soluble form of Fas, so that it might have potential as a therapeutic target to improve chemotherapy effectiveness.
Affiliation: Department of Medical Genetics, Shahid Beheshti University of Medical sciences, Tehran, Iran. Email: Mohammad_823@yahoo.com .
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2: Title: Expression Analysis of Long Non-coding RNAs in the Blood of Multiple Sclerosis Patients.
Journal: Journal of molecular neuroscience : MN (J Mol Neurosci), 2017 .
Snippet: In the present study, we evaluated the expression of three lncRNAs with putative roles in the regulation of immune response, namely TNF-α and heterogeneous nuclear ribonucleoprotein L (THRIL), Fas cell surface death receptor- antisense 1 (FAS-AS1), and plasmacytoma variant translocation 1 (PVT1) in circulating blood cells of 50 Iranian relapsing-remitting multiple sclerosis (RRMS) patients compared with healthy subjects by means of quantitative real-time polymerase chain reaction (PCR).
Affiliation: Neurophysiology Research Center, Hamadan University of Medical Sciences, Hamadan, Iran. Department of Medical Genetics, Shahid Beheshti University of Medical Sciences, Tehran, Iran. Department of Medical Genetics, Mashhad University of Medical Sciences, Mashhad, Iran. Urogenital Stem Cell Research, Shahid Beheshti University of Medical Sciences, Tehran, Iran. Department of Neurology, Hamedan University of Medical Sciences, Hamadan, Iran. Department of Medical Genetics, Shahid Beheshti University of Medical Sciences, Tehran, Iran. mohammad_823@yahoo.com. Urogenital Stem Cell Research, Shahid Beheshti University of Medical Sciences, Tehran, Iran. mohammad_823@yahoo.com. .
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3: Title: Data in support of transcriptional regulation and function of Fas-antisense long noncoding RNA during human erythropoiesis.
Authors: Villamizar, Olga, et.al. .
Journal: Data in brief (Unknown Journal), Vol. 7, 2016 .
Snippet: From these data, we identified lncRNA Fas-antisense 1 (Fas-AS1 or Saf) described in the research article.
Affiliation: Department of Medical Microbiology, Immunology and Cell Biology, Southern Illinois University School of Medicine, Springfield, IL, USA. Department of Medical Microbiology, Immunology and Cell Biology, Southern Illinois University School of Medicine, Springfield, IL, USA; Department of Pharmacology and Toxicology and Cancer Institute, University of Mississippi Medical Center, Jackson, MS, USA. Department of Medical Microbiology, Immunology and Cell Biology, Southern Illinois University School of Medicine, Springfield, IL, USA; Simmons Cancer Institute, Springfield, IL, USA. Department of Hematology, St. Jude Children׳s Research Hospital, Memphis, TN, USA. .
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4: Title: Fas-antisense long noncoding RNA is differentially expressed during maturation of human erythrocytes and confers resistance to Fas-mediated cell death.
Authors: Villamizar, Olga, et.al. .
Journal: Blood cells, molecules & diseases (Blood Cells Mol Dis), Vol. 58, 2016 .
Snippet: Here, a culture model of human erythropoiesis revealed that lncRNA Fas-antisense 1 (Fas-AS1 or Saf) was induced during differentiation through the activity of essential erythroid transcription factors GATA-1 and KLF1.
Affiliation: Department of Medical Microbiology, Immunology and Cell Biology, Southern Illinois University School of Medicine, Springfield, IL, USA. Department of Medical Microbiology, Immunology and Cell Biology, Southern Illinois University School of Medicine, Springfield, IL, USA; Department of Pharmacology and Toxicology, Cancer Institute, University of Mississippi Medical Center, Jackson, MS, USA. Department of Medical Microbiology, Immunology and Cell Biology, Southern Illinois University School of Medicine, Springfield, IL, USA; Simmons Cancer Institute at SIU, Springfield, IL, USA. Department of Hematology, St. Jude Children's Research Hospital, Memphis, TN, USA. Department of Medical Microbiology, Immunology and Cell Biology, Southern Illinois University School of Medicine, Springfield, IL, USA; Simmons Cancer Institute at SIU, Springfield, IL, USA. Electronic address: awilber@siumed.edu. .
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5: Title: [Expression of FAS-AS1 in breast cancer and its effect on the host gene sFas].
Authors: Zhou, S W, et.al. .
Journal: Zhonghua yi xue za zhi, Vol. 96 (12): 949-53, 2016 .
Snippet: Expression of sFas and cell proliferation was significantly reduced in FAS-AS1-overexpressing MCF-7 cells (P<0.05).
Affiliation: Department of Head & Neck, and Breast Surgery, Xinxiang Central Hospital, Xinxiang, Henan 453000, China. .
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6: Title: Long noncoding RNA Saf and splicing factor 45 increase soluble Fas and resistance to apoptosis.
Authors: Villamizar, Olga, et.al. .
Journal: Oncotarget, Vol. 7 (12): 13810-26, 2016 .
Snippet: Neoplastic cells are frequently resistant to Fas-mediated apoptosis, evade Fas signals through down regulation of Fas and produce soluble Fas proteins that bind FasL thereby blocking apoptosis.
Affiliation: Department of Medical Microbiology, Immunology and Cell Biology, Southern Illinois University School of Medicine, Springfield, Illinois, USA. Department of Microbiology, Pontificia Universidad Javeriana, Bogotá, Colombia. Department of Hematology, St. Jude Children's Research Hospital, Memphis, Tennessee, USA. .
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7: Title: Time, dose and ataxia telangiectasia mutated (ATM) status dependency of coding and noncoding RNA expression after ionizing radiation exposure.
Authors: Kabacik, S, et.al. .
Journal: Radiation research (Radiat Res), Vol. 183 (3): 325-37, 2015 .
Snippet: Conversely, FAS-AS1 was up-regulated up to fivefold by 5 Gy irradiation.
Affiliation: Public Health England, Centre for Radiation, Chemical and Environmental Hazards, OX11 0RQ, United Kingdom. .
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8: Title: FAS-antisense 1 lncRNA and production of soluble versus membrane Fas in B-cell lymphoma.
Authors: Sehgal, L, et.al. .
Journal: Leukemia, Vol. 28 (12): 2376-87, 2014 .
Snippet: EZH2-mediated repression of FAS-AS1 promoter can be released by DZNeP (3-Deazaneplanocin A) or overcome by ectopic expression of FAS-AS1, both of which increase levels of FAS-AS1 and correspondingly decrease expression of sFas.
Affiliation: Department of Lymphoma and Myeloma, The University of Texas MD Anderson Cancer Center, Houston, TX, USA. .
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