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433 documents found
1: Title: EGLN1/c-Myc Induced Lymphoid-Specific Helicase Inhibits Ferroptosis through Lipid Metabolic Gene Expression Changes.
Authors: Jiang, Yiqun, et.al. .
Journal: Theranostics, Vol. 7 (13): 3293-3305, 2017 .
Snippet: Therefore, our study elucidates the molecular basis of the c-Myc/EGLN1-mediated induction of LSH expression that inhibits ferroptosis, which can be exploited for the development of therapeutic strategies targeting ferroptosis for the treatment of cancer.
Affiliation: Key Laboratory of Carcinogenesis and Cancer Invasion, Ministry of Education, Xiangya Hospital, Central South University, 87 Xiangya Road, Changsha, Hunan, 410008 China. Cancer Research Institute, Central South University, 110 Xiangya Road, Changsha, Hunan, 410078 China. Department of Thoracic Surgery, Second Xiangya Hospital, Central South University, Changsha, China. Institute of Medical Sciences, Xiangya Hospital, Central South University, 87 Xiangya Road, Changsha, Hunan, 410008 China. Department of Pathology, Xiangya Hospital, Central South University, 87 Xiangya Road, Changsha, Hunan, 410008 China. Shanghai Institute of Material Medica, Chinese Academy of Sciences (CAS), 555 Zu Chongzhi Road, Zhangjiang Hi-Tech Park, Shanghai, 201203, China. Department of Pharmacology, School of Pharmaceutical Sciences, Central South University, Changsha, Hunan 410078 China. Department of Pathology, Yale School of Medicine, New Haven, CT, 06520 U.S.A. .
2: Title: The VHL Tumor Suppressor Gene: Insights into Oxygen Sensing and Cancer.
Authors: Kaelin, William G (Jr) .
Journal: Transactions of the American Clinical and Climatological Association (Trans Am Clin Climatol Assoc), Vol. 128, 2017 .
Snippet: VHL, EglN1, and HIF2α polymorphisms have been linked to familial polycythemia and adaptation to high altitude.
Affiliation: BOSTON, MASSACHUSETTS. .
3: Title: High altitude and pre-eclampsia: Adaptation or protection.
Authors: Ahmed, Sarah I Y, et.al. .
Journal: Medical hypotheses (Med Hypotheses), Vol. 104, 2017 .
Snippet: Our results demonstrated that the genetic variants of EPAS1, ADAM9, and EGLN1 increased approximately three-fold in the cases of preeclampsia compared to normal pregnancy.
Affiliation: Department of Molecular Biology, Institute of Endemic Diseases, University of Khartoum, Khartoum, Sudan. Electronic address: emailstosarah@gmail.com. Department of Molecular Biology, Institute of Endemic Diseases, University of Khartoum, Khartoum, Sudan. Field & Research Stations, Institute of Endemic Diseases, University of Khartoum, Khartoum, Sudan. .
4: Title: Evolutionary history of Tibetans inferred from whole-genome sequencing.
Authors: Hu, Hao, et.al. .
Journal: PLoS genetics (Plos Genet), Vol. 13 (4): e1006675, 2017 .
Snippet: The CMS test ranked EPAS1 and EGLN1 as the top two positive selection candidates, and in addition identified PTGIS, VDR, and KCTD12 as new candidate genes.
Affiliation: Department of Epidemiology, University of Texas MD Anderson Cancer Center, Houston, Texas, United States of America. Nuffield Department of Medicine, University of Oxford, Oxford, United Kingdom. Institute for Systems Biology, Seattle, Washington, United States of America. Department of Anthropology, University of Utah, Salt Lake City, Utah, United States of America. Department of Medicine, University of Utah School of Medicine and George E. Wahlin Veterans Administration Medical Center, Salt Lake City, Utah, United States of America. Department of Human Genetics, University of Utah, Salt Lake City, Utah, United States of America. Department of Molecular and Cellular Therapeutics, The Royal College of Surgeons in Ireland, Dublin, Ireland. Skaggs School of Pharmacy and Pharmaceutical Science, UC San Diego, La Jolla, California, United States of America. Department of Physiology, Anatomy and Genetics, University of Oxford, Oxford, United Kingdom. Department of Medicine, Division of Physiology, University of California San Diego, La Jolla, California, United States of America. .
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5: Title: Hypoxia-inducible factor 1-alpha does not regulate osteoclastogenesis but enhances bone resorption activity via prolyl-4-hydroxylase 2.
Authors: Hulley, Philippa A, et.al. .
Journal: The Journal of pathology (J Pathol), Vol. 242 (3): 322-333, 2017 .
Snippet: These data indicate that HIF predominantly functions as a regulator of osteoclast-mediated bone resorption, with little effect on osteoclast differentiation.
Affiliation: Nuffield Department of Orthopaedics Rheumatology & Musculoskeletal Sciences, University of Oxford, Oxford, UK. Nuffield Department of Medicine, University of Oxford, Oxford, UK. BHF Experimental MR Unit, University of Oxford, Oxford, UK. Nuffield Department of Orthopaedics Rheumatology & Musculoskeletal Sciences, Nuffield Orthopaedic Centre, University of Oxford, Oxford, UK. .
6: Title: Increased hypoxia-inducible factor-1α in striated muscle of tumor-bearing mice.
Authors: Devine, Raymond D, et.al. .
Journal: American journal of physiology. Heart and circulatory physiology (Am J Physiol Heart Circ Physiol), Vol. 312 (6): H1154-H1162, 2017 .
Snippet: These results indicate that HIF-1α may contribute, in part, to the metabolic changes that occur during cancer cachexia.NEW & NOTEWORTHY We used proteomics and metadata analysis software to identify contributors to metabolic changes in striated muscle during cancer cachexia.
Affiliation: Dorothy M. Davis Heart and Lung Research Institute, The Ohio State University Wexner Medical Center, Columbus, Ohio. Molecular, Cellular and Developmental Biology Graduate Program, The Ohio State University, Columbus, Ohio. Division of Biosciences, College of Dentistry, The Ohio State University, Columbus, Ohio. Dorothy M. Davis Heart and Lung Research Institute, The Ohio State University Wexner Medical Center, Columbus, Ohio; Loren.Wold@osumc.edu. Department of Physiology and Cell Biology, The Ohio State University, Columbus, Ohio; and. College of Nursing, The Ohio State University, Columbus, Ohio. .
7: Title: Gain-of-function EGLN1 prolyl hydroxylase (PHD2 D4E:C127S) in combination with EPAS1 (HIF-2α) polymorphism lowers hemoglobin concentration in Tibetan highlanders.
Authors: Tashi, Tsewang, et.al. .
Journal: Journal of molecular medicine (Berlin, Germany) (J Mol Med), Vol. 95 (6): 665-670, 2017 .
Snippet: While EGLN1 and EPAS1 genotypes lower Hb, this study indicates additional Hb modifiers.
Affiliation: Division of Hematology, University of Utah, Salt Lake City, UT, USA. Department of Pathology and ARUP Laboratories, University of Utah, Salt Lake City, UT, USA. Research Center for High-Altitude Medicine, Qinghai University, Xining, China. Division of Hematology and Oncology, Department of Medicine, University of Illinois at Chicago, Chicago, IL, USA. Departments of Medicine and Obstetrics/Gynecology, University of Colorado Denver, Aurora, CO, USA. MD Anderson Cancer Center, University of Texas, Houston, TX, USA. Department of Molecular Biology, Jessenius Faculty of Medicine in Martin, Comenius University in Bratislava, Bratislava, Slovakia. Sher-i-Kashmir Institute of Medical Sciences, Srinagar, India. Regional Centre for Biotechnology, Faridabad, Haryana, India. Division of Hematology, University of Utah, Salt Lake City, UT, USA. josef.prchal@hsc.utah.edu. Department of Pathology and ARUP Laboratories, University of Utah, Salt Lake City, UT, USA. josef.prchal@hsc.utah.edu. Department of Pathophysiology and 1st Department of Medicine, 1st Faculty Medicine, Charles University in Prague, Prague, Czech Republic. josef.prchal@hsc.utah.edu. Hematology, SOM 5C310, University of Utah School of Medicine, Salt Lake City, UT, 84132-2408, USA. josef.prchal@hsc.utah.edu. Research Center for High-Altitude Medicine, Qinghai University, Xining, China. geriligao@hotmail.com. .
8: Title: Hypoxia Pathway Mutations in Pheochromocytomas and Paragangliomas.
Authors: Amorim-Pires, Diana, et.al. .
Journal: Cytogenetic and genome research (Cytogenet Genome Res), Vol. 150 (3-4): 227-241, 2016 .
Snippet: This article is focused on reviewing the tumorigenic mechanisms resultant from VHL, SDHx, EGLN1, and EPAS1 mutations, as well as the associated tumors, namely PCC/PGL, and extra manifestations such as polycythemia.
Affiliation: Medical Faculty, University of Porto, Porto, Portugal. .
9: Title: Sherpas share genetic variations with Tibetans for high-altitude adaptation.
Authors: Bhandari, Sushil, et.al. .
Journal: Molecular genetics & genomic medicine (Mol Genet Genomic Med), Vol. 5 (1): 76-84, 2017 .
Snippet: Sherpa individuals carrying the derived alleles of EPAS1 (rs113305133, rs116611511 and rs12467821), EGLN1 (rs186996510 and rs12097901) and TED have lower hemoglobin levels when compared with those wild-type allele carriers.
Affiliation: State Key Laboratory of Genetic Resources and EvolutionKunming Institute of ZoologyChinese Academy of SciencesKunming650223China; Kunming College of Life ScienceUniversity of Chinese Academy of SciencesBeijing100049China; Nepal Academy of Science and TechnologyGPO Box: 3323, KhumaltarLalitpurNepal. State Key Laboratory of Genetic Resources and Evolution Kunming Institute of Zoology Chinese Academy of Sciences Kunming 650223 China. High Altitude Medical Research Center School of Medicine Tibetan University Lhasa 850000 China. State Key Laboratory of Genetic Resources and EvolutionKunming Institute of ZoologyChinese Academy of SciencesKunming650223China; Institute of Primate Translational MedicineKunming University of Science and TechnologyKunming650500China. National Key Laboratory of High Altitude Medicine High Altitude Medical Research Institute Xining 810012 China. .
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10: Title: Tumor suppressor SPOP ubiquitinates and degrades EglN2 to compromise growth of prostate cancer cells.
Authors: Zhang, Linli, et.al. .
Journal: Cancer letters (Cancer Lett), Vol. 390, 2017 .
Snippet: Different from EglN1 which function largely depends on the role of hypoxia-induce factor alpha (HIFα) in tumors, the functional significance and the upstream regulatory mechanisms of EglN2, especially in prostate cancer setting, remain largely unclear.
Affiliation: Department of Oncology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei 430030, PR China; Department of Pathology, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, MA 02215, USA. Department of Oncology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei 430030, PR China. Department of Pathology, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, MA 02215, USA. Department of Oncology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei 430030, PR China. Electronic address: gqhu@tjh.tjmu.edu.cn. Department of Pathology, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, MA 02215, USA. Electronic address: Jguo3@bidmc.harvard.edu. .
11: Title: Discovery of a murine model of clinical PAH: Mission impossible?
Authors: Dai, Zhiyu, et.al. .
Journal: Trends in cardiovascular medicine (Trends Cardiovasc Med), Vol. 27 (4): 229-236, 2017 .
Snippet: Pulmonary arterial hypertension (PAH) is a lung vascular disease characterized with a progressive increase of pulmonary vascular resistance and obliterative pulmonary vascular remodeling resulting in right heart failure and premature death.
Affiliation: Department of Pharmacology, University of Illinois College of Medicine, Chicago, IL 60612, USA; Center for Lung and Vascular Biology, University of Illinois College of Medicine, Chicago, IL 60612, USA. Department of Pharmacology, University of Illinois College of Medicine, Chicago, IL 60612, USA; Center for Lung and Vascular Biology, University of Illinois College of Medicine, Chicago, IL 60612, USA. Electronic address: yyzhao@uic.edu. .
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12: Title: Evolutionary selected Tibetan variants of HIF pathway and risk of lung cancer.
Authors: Lanikova, Lucie, et.al. .
Journal: Oncotarget, Vol. 8 (7): 11739-11747, 2017 .
Snippet: EGLN1/PHD2 and EPAS1/HIF-2α, both crucial components of hypoxia sensing, are the two best-established loci contributing to high altitude adaptation.
Affiliation: Division of Hematology, University of Utah School of Medicine, Salt Lake City, Utah, USA. Institute of Molecular Genetics, Academy of Sciences, Prague, Czech Republic. Institute for Clinical and Experimental Pathology, ARUP Laboratories, Salt Lake City, Utah, USA. Department of Pathology, University of Utah School of Medicine, Salt Lake City, Utah, USA. University of Texas, MD Anderson Cancer Center, Houston, Texas, USA. Department of Molecular Biology, Comenius University in Bratislava, Jessenius Faculty of Medicine in Martin, Slovak Republic. B.P. Koirala Memorial Cancer Hospital, Bharatpur, Chitwan, Nepal. Maryland Institute for Applied Environmental Health, and University of Maryland College Park School of Public Health, Maryland, USA. Division of Public Health, Department of Family & Preventive Medicine and Huntsman Cancer Institute, University of Utah School of Medicine, Salt Lake City, Utah, USA. Department of Dermatology, University of Utah School of Medicine, Salt Lake City, Utah, USA. Department of Pathophysiology and 1st Department of Medicine, 1st Faculty of Medicine, Charles University in Prague, Czech Republic. .
13: Title: Inhibition of Hypoxia-Induced Retinal Angiogenesis by Specnuezhenide, an Effective Constituent of Ligustrum lucidum Ait., through Suppression of the HIF-1α/VEGF Signaling Pathway.
Authors: Wu, Jianming, et.al. .
Journal: Molecules (Basel, Switzerland) (Molecules), Vol. 21 (12), 2016 .
Snippet: In the present study, we established a vascular endothelial growth factor A (VEGFA) secretion model of human acute retinal pigment epithelial-19 (ARPE-19) cells by exposure of 150 μM CoCl₂ to the cells and determined the VEGFA concentrations, the mRNA expressions of VEGFA, hypoxia inducible factor-1α (HIF-1α) & prolyl hydroxylases 2 (PHD-2), and the protein expressions of HIF-1α and PHD-2 after treatment of 3-(5'-hydroxymethyl-2'-furyl)-1-benzylindazole (YC-1, 1.0 μg/mL) or SPN (0.2, 1.0 and 5.0 μg/mL).
Affiliation: Laboratory of Chinese Materia Medica, Department of Pharmacology, School of Pharmacy, Southwest Medical University, Luzhou 646000, Sichuan, China. jianmingwu@swmu.edu.cn. Post-Doctoral Research Station, KangHong Pharmaceutical Group, Chengdu 610036, Sichuan, China. jianmingwu@swmu.edu.cn. Post-Doctoral Mobile Station, West China School of Pharmacy, Sichuan University, Chengdu 610041, Sichuan, China. jianmingwu@swmu.edu.cn. Post-Doctoral Research Station, KangHong Pharmaceutical Group, Chengdu 610036, Sichuan, China. kexiao@cnkh.com. Post-Doctoral Research Station, KangHong Pharmaceutical Group, Chengdu 610036, Sichuan, China. fu_wei99@163.com. Post-Doctoral Research Station, KangHong Pharmaceutical Group, Chengdu 610036, Sichuan, China. 1483687108@qq.com. Post-Doctoral Research Station, KangHong Pharmaceutical Group, Chengdu 610036, Sichuan, China. arken20@163.com. Post-Doctoral Research Station, KangHong Pharmaceutical Group, Chengdu 610036, Sichuan, China. dy_ts110@163.com. Post-Doctoral Research Station, KangHong Pharmaceutical Group, Chengdu 610036, Sichuan, China. manapppp@163.com. Post-Doctoral Research Station, KangHong Pharmaceutical Group, Chengdu 610036, Sichuan, China. zhaomanqian@cnkh.com. Post-Doctoral Research Station, KangHong Pharmaceutical Group, Chengdu 610036, Sichuan, China. hxf@cnkh.com. Post-Doctoral Mobile Station, West China School of Pharmacy, Sichuan University, Chengdu 610041, Sichuan, China. zrzzl@vip.sina.com. .
14: Title: Haematopoietic prolyl hydroxylase-1 deficiency promotes M2 macrophage polarization and is both necessary and sufficient to protect against experimental colitis.
Authors: Van Welden, Sophie, et.al. .
Journal: The Journal of pathology (J Pathol), Vol. 241 (4): 547-558, 2017 .
Snippet: Here, we demonstrated that Phd1 deletion in endothelial and haematopoietic cells (Phd1(f/)(f) Tie2:cre) protected mice from dextran sulphate sodium (DSS)-induced colitis, with reduced epithelial erosions, immune cell infiltration, and colonic microvascular dysfunction, whereas the response of Phd2(f/+) Tie2:cre and Phd3(f/)(f) Tie2:cre mice to DSS was similar to that of their littermate controls.
Affiliation: Department of Gastroenterology, Ghent University, Ghent, Belgium. Heisenberg Research Group, Department of Clinical Pathobiochemistry, Institute for Clinical Chemistry and Laboratory Medicine, Technische Universität Dresden, Dresden, Germany. Department of Internal Medicine, Ghent University, Ghent, Belgium. Inflammation Research Centre VIB, Zwijnaarde, Belgium. Department of Pulmonary Medicine, Ghent University, Ghent, Belgium. Laboratory of Radiopharmacy, Ghent University, Ghent, Belgium. Infinity Imaging Laboratory (iMinds Medical IT-IBiTech-MEDISIP), Ghent University, Ghent, Belgium. Department of Clinical Chemistry, Microbiology, and Immunology, Ghent University, Ghent, Belgium. IBD Centre, Department of Gastroenterology, Humanitas Clinical and Research Centre, Rozzano, Italy. Division of Medical Biology, Department of Psychiatry, Technische Universität Dresden, Dresden, Germany. Laboratory of Angiogenesis and Vascular metabolism, Vesalius Research Centre, KU Leuven, VIB, Leuven, Belgium. Department of Rheumatology, Ghent University, Ghent, Belgium. .
15: Title: Notch Downregulation and Extramedullary Erythrocytosis in Hypoxia-Inducible Factor Prolyl 4-Hydroxylase 2-Deficient Mice.
Authors: Myllymäki, Mikko N M, et.al. .
Journal: Molecular and cellular biology (Mol Cell Biol), Vol. 37 (2), 2017 .
Snippet: Mutations in HIF prolyl 4-hydroxylase 2 (HIF-P4H-2) (PHD2/EGLN1), the major downregulator of HIFα subunits, are found in familiar erythrocytosis, and large-spectrum conditional inactivation of HIF-P4H-2 in mice leads to severe erythrocytosis.
Affiliation: Biocenter Oulu, Faculty of Biochemistry and Molecular Medicine, Oulu Center for Cell-Matrix Research, University of Oulu, Oulu, Finland. Department of Pathology, Oulu University Hospital, and Department of Pathology, Cancer and Translational Medicine Research Unit, University of Oulu, Oulu, Finland. Biocenter Oulu, Faculty of Biochemistry and Molecular Medicine, Oulu Center for Cell-Matrix Research, University of Oulu, Oulu, Finland raisa.serpi@oulu.fi. .
16: Title: Gene panel sequencing improves the diagnostic work-up of patients with idiopathic erythrocytosis and identifies new mutations.
Authors: Camps, Carme, et.al. .
Journal: Haematologica, Vol. 101 (11): 1306-1318, 2016 .
Snippet: Twenty-two were novel variants in erythrocytosis-associated genes (EGLN1, EPAS1, VHL, BPGM, JAK2, SH2B3) and in novel genes included in the panel (e.g.
Affiliation: National Institute for Health Research (NIHR) Comprehensive Biomedical Research Centre, Oxford, UK. Wellcome Trust Centre for Human Genetics, University of Oxford, UK. Nuffield Department of Medicine, University of Oxford, UK nayia.petousi@ndm.ox.ac.uk. Hematology Department, Centro Hospitalar e Universitário de Coimbra, Portugal. Department of Pediatrics and Adolescent Medicine, University Medical Center, Ulm, Germany. Centre for Cancer Research and Cell Biology, Queen's University, Belfast, UK. Nuffield Department of Medicine, University of Oxford, UK. Department of Physiology, Anatomy and Genetics, University of Oxford, UK. .
17: Title: PHD2 Is a Regulator for Glycolytic Reprogramming in Macrophages.
Authors: Guentsch, Annemarie, et.al. .
Journal: Molecular and cellular biology (Mol Cell Biol), Vol. 37 (1), 2017 .
Snippet: However, if and how PHD enzymes affect macrophage metabolism are enigmatic.
Affiliation: Institute for Cardiovascular Physiology, Georg August University Göttingen, Göttingen, Germany. Department of Clinical Pathobiochemistry, Institute of Clinical Chemistry and Laboratory Medicine, Technical University Dresden, Dresden, Germany. Microarray and Deep-Sequencing Core Facility, University Medical Center Göttingen, Göttingen, Germany. Institute for Cellular Biochemistry, Georg August University Göttingen, Göttingen, Germany. Department of Cardiology, King's College London British Heart Foundation Centre, London, United Kingdom. Institute for Cardiovascular Physiology, Georg August University Göttingen, Göttingen, Germany doerthe.katschinski@med.uni-goettingen.de. .
18: Title: Prolyl hydroxylase mediated inhibition of fatty acid synthase to combat tumor growth in mammary gland carcinoma.
Authors: Singh, Manjari, et.al. .
Journal: Breast cancer (Tokyo, Japan) (Breast Cancer), Vol. 23 (6): 820-829, 2016 .
Snippet: Considering the above, it is hypothesized that chemical activation of Phd-2 can curtail down HIF-1α and subsequently fatty acid synthase expression.
Affiliation: Department of Pharmaceutical Sciences, School of Biosciences and Biotechnology, Babasaheb Bhimrao Ambedkar University (A Central University), Vidya Vihar, Raebareli Road, Lucknow, 226025, India. Department of Pharmaceutical Sciences, Faculty of Health Medical Sciences Indigenous and Alternative Medicine, SHIATS-Deemed to be University, Naini, Allahabad, Uttar Pradesh, India. Department of Pharmaceutical Sciences, School of Biosciences and Biotechnology, Babasaheb Bhimrao Ambedkar University (A Central University), Vidya Vihar, Raebareli Road, Lucknow, 226025, India. gauravpharm@gmail.com. .
19: Title: Identification of Small-Molecule PHD2 Zinc Finger Inhibitors that Activate Hypoxia Inducible Factor.
Authors: Arsenault, Patrick R, et.al. .
Journal: Chembiochem : a European journal of chemical biology (Chembiochem), Vol. 17 (24): 2316-2323, 2016 .
Snippet: In mammals, there are three PHD paralogues, and PHD2 has emerged as a particularly critical one for regulating HIF target genes such as erythropoietin (EPO), which controls red cell mass and hematocrit.
Affiliation: Department of Pathology and Laboratory Medicine, Perelman School of Medicine, University of Pennsylvania, 605 Stellar Chance Labs, 422 Curie Blvd, Philadelphia, PA, 19104, USA. The Wistar Institute, 3601 Spruce St., Philadelphia, PA, 19104, USA. .
20: Title: Neuronal prolyl-4-hydroxylase 2 deficiency improves cognitive abilities in a murine model of cerebral hypoperfusion.
Authors: Gruneberg, Daniel, et.al. .
Journal: Experimental neurology (Exp Neurol), Vol. 286, 2016 .
Snippet: Previous studies in rodent models of dementia indicated a favorable effect of the hypoxia-inducible factor (HIF) targets VEGF (vascular endothelial growth factor) and erythropoietin (Epo).
Affiliation: Institute of Physiology and Pathophysiology, University of Heidelberg, Germany. Department of Anesthesiology, University of Heidelberg, Germany. Institute of Physiology and Pathophysiology, University of Heidelberg, Germany. Electronic address: reiner.kunze@physiologie.uni-heidelberg.de. .
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