gopubmed logo
 
find other proteinsAll proteins
GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 08 Dec 2016.

Ubiquitin specific peptidase 6

TRE2, USP6, TRE17, HRP-1
Papers using TRE2 antibodies
Impaired mammary gland and lymphoid development caused by inducible expression of Axin in transgenic mice
Supplier
Costantini Frank et al., In The Journal of Cell Biology, 1998
... 6.4-kb NheI-EcoRV fragment of MT–Axin–IRES2–GFP–hβG3′ was then inserted into the same restriction sites downstream of TRE2, which consisted of seven copies of the tet operator and a minimal CMV promoter (CLONTECH Laboratories, Inc.).
Papers on TRE2
The oncogenic TBC domain protein USP6/TRE17 regulates cell migration and cytokinesis.
GeneRIF
Haucke et al., Berlin, Germany. In Biol Cell, 2012
manipulating USP6 expression levels alters the ability of cells to migrate and to divide. Cell proliferation and progression through cytokinesis depend on USP6 expression
TRE17/USP6 oncogene translocated in aneurysmal bone cyst induces matrix metalloproteinase production via activation of NF-kappaB.
GeneRIF
Chou et al., Philadelphia, United States. In Oncogene, 2010
TRE17 is sufficient to initiate tumorigenesis, identify MMPs as novel TRE17 effectors that likely contribute to aneurysmal bone cyst pathogenesis.
Frequency of USP6 rearrangements in myositis ossificans, brown tumor, and cherubism: molecular cytogenetic evidence that a subset of "myositis ossificans-like lesions" are the early phases in the formation of soft-tissue aneurysmal bone cyst.
GeneRIF
Oliveira et al., Rochester, United States. In Skeletal Radiol, 2008
No USP6 rearrangements were found in cherubism or brown tumors. USP6 rearrangements were identified in 2 patients with myositis ossificans.
Mutational analysis of the TRE2 oncogene encoding an inactive RabGAP.
GeneRIF
Vandenbol et al., Gembloux, Belgium. In Biotechnol Lett, 2007
The lack of secondary structure of the region flanking the TBC domain in TRE2 may explain why this region plays a role in the lack of GAP activity, even when a potentially functional TBC domain is present.
Calcium/calmodulin regulates ubiquitination of the ubiquitin-specific protease TRE17/USP6.
GeneRIF
Chou et al., Philadelphia, United States. In J Biol Chem, 2005
Ca2+/CaM has a role in regulating ubiquitination through direct interaction with TRE17
share on facebooktweetadd +1mail to friends