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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Aug 2016.

Transmembrane and coiled-coil domain family 1

TMCC-1, Kiaa0779
Top mentioned proteins: HAD, p53, ACID, KLF15, CAN
Papers on TMCC-1
Genetic determinants of uterine fibroid size in the multiethnic NIEHS uterine fibroid study.
Wiener et al., Birmingham, United States. In Int J Mol Epidemiol Genet, 2014
Several other SNPs, namely those located in MYT1L, TMCC1 and BRCA1, reached promising associations.
Gene-centric meta-analyses for central adiposity traits in up to 57 412 individuals of European descent confirm known loci and reveal several novel associations.
Taylor et al., In Hum Mol Genet, 2014
Previously unreported rs2811337-G near TMCC1 was associated with increased WHR (β ± SE, 0.048 ± 0.008, P = 7.7 × 10(-9)) as was rs7302703-G in HOXC10 (β = 0.044 ± 0.008, P = 2.9 × 10(-7)) and rs936108-C in PEMT (β = 0.035 ± 0.007, P = 1.9 × 10(-6)).
Transmembrane and coiled-coil domain family 1 is a novel protein of the endoplasmic reticulum.
Qi et al., Hong Kong, Hong Kong. In Plos One, 2013
In this study, we identified a new protein of the ER, TMCC1 (transmembrane and coiled-coil domain family 1).
Antiproliferative effects of the major tea polyphenol, (-)-epigallocatechin gallate and retinoic acid in cervical adenocarcinoma.
Iwasaka et al., Saga, Japan. In Gynecol Oncol, 2008
METHODS: Cell growth rate was examined after treatment for 4, 7 and 10 days with 0-100 microM EGCG and/or 1 microM RA in two cervical adenocarcinoma cell lines, HeLa and TMCC-1.
Refinement of the locus for hereditary congenital facial palsy on chromosome 3q21 in two unrelated families and screening of positional candidate genes.
Padberg et al., Nijmegen, Netherlands. In Eur J Hum Genet, 2006
In addition, mutation analysis on seven candidate genes: KLF15, FLJ40083, PODXL2, TMCC1, PLEXIN-A1, PLEXIN-D1, and GATA-2, was performed.
Inhibitory effect of the tea polyphenol, (-)-epigallocatechin gallate, on growth of cervical adenocarcinoma cell lines.
Iwasaka et al., Saga, Japan. In Cancer Lett, 2006
TRAP assay is used for telomerase activity, flow cytometry analysis and pKi-67 immunofluoroscein staining in cervical adenocarcinoma cell lines (OMC-4, TMCC-1).
Identifying new candidate genes for hereditary facial paresis on chromosome 3q21-q22 by RNA in situ hybridization in mouse.
van Bokhoven et al., Nijmegen, Netherlands. In Genomics, 2005
The remaining 4 genes (Klf15, Flj40083, Kiaa0779, and Podxl2) were found to be expressed at spatial and temporal positions during mouse development that correlate with HCFP regions in humans, defining these genes as primary candidates in HCFP.
The tea polyphenol, (-)-epigallocatechin gallate effects on growth, apoptosis, and telomerase activity in cervical cell lines.
Iwasaka et al., Saga, Japan. In Gynecol Oncol, 2004
RESULTS: EGCG inhibited growth more than 90% in HEN-18 and HEC-18, whereas growth inhibition was less in ME180, TMCC-1, HeLa, SiHa, HEC-18T, and HEN-18S.
Cytologic changes in two cervical carcinoma cell lines after transfection of the wild-type p53 gene.
Sugimori et al., Saga, Japan. In Acta Obstet Gynecol Scand, 1996
METHOD: A dexamethazone-inducible wt-p53 cDNA was introduced into two cervical carcinoma cell lines (TMCC-1 and ME180) and morphological changes were examined under a phase contrast microscope and following Papanicolaou staining.
Growth suppression of a cervical cancer cell line (TMCC-1) by the human wild-type p53 gene.
Sugimori et al., Saga, Japan. In Gynecol Oncol, 1996
To investigate the effects of human wild-type p53 expression on the proliferation of cervical carcinoma cells, a plasmid, pMO7-hp53, which contains a full-length cDNA of the human wild-type p53 (wt-p53) gene, was transfected into a cell line (TMCC-1) derived from an endocervical type, human papilloma virus-positive adenocarcinoma of the uterine cervix.
Changes in CA125 release and surface expression caused by drugs in uterine cervix adenocarcinoma cells.
Konishi et al., Kyoto, Japan. In Ann Nucl Med, 1993
TMCC-1, uterine cervical adenocarcinoma cells, were exposed to dexamethasone (DEX), sodium n-butyrate (NaB), dibutyryl cyclic AMP (dbcAMP), retinoic acid (RA), calcitriol (VD3), and interferon-gamma (IFN-gamma).
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