Autolytic transition of mu-calpain upon activation as resolved by antibodies distinguishing between the pre- and post-autolysis forms.
In PLoS ONE, 1991
... Temecula, CA), mouse anti-Tuj1 (1∶2000, Covance, Princeton, NJ), rabbit anti-GFAP (1∶500, DAKO, Carpinteria, CA), rabbit anti-Synapsin I (1∶500, Millipore), mouse anti-Cux1 (1∶100, Abnova, Taipei, Taiwan), rabbit anti-Satb2 ...
Ganglioside GQ1b induces dopamine release through the activation of Pyk2.
Beijing, China. In Mol Cell Neurosci, Jan 2016
Further studies revealed that activated Pyk2 stimulated ERK1/2 and p-38, while only the ERK1/2 activation was indispensable for GQ1b induced DA release, which interacted with Synapsin I directly and led to its phosphorylation, then depolymerization of F-actin, thus contributed to DA release.
Epilepsy associated with autism and attention deficit hyperactivity disorder: is there a genetic link?
Roma, Italy. In Brain Dev, 2014
The majority of the candidate genes are involved in synaptic formation/remodeling/maintenance (NRX1, CNTN4, DCLK2, CNTNAP2, TRIM32, ASTN2, CTNTN5, SYN1), neurotransmission (SYNGAP1, GABRG1, CHRNA7), or DNA methylation/chromatin remodeling (MBD5).
Glutamatergic neurotransmission in the synapsin I and II double knock-out mouse.
Oslo, Norway. In Semin Cell Dev Biol, 2011
We here briefly describe the important characteristics of the synapsin proteins, and review recent studies on transgenic mice devoid of the gene products encoded by the synapsin I and II genes, where both neurochemical, cell biological and electrophysiological methods have been employed.