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SWI/SNF related, matrix associated, actin dependent regulator of chromatin, subfamily a, member 1

This gene encodes a member of the SWI/SNF family of proteins. Members of this family have helicase and ATPase activities and are thought to regulate transcription of certain genes by altering the chromatin structure around those genes. Alternatively spliced transcript variants encoding different isoforms have been found for this gene. [provided by RefSeq, Jul 2008] (from NCBI)
Papers using SWI antibodies
UBF levels determine the number of active ribosomal RNA genes in mammals
Hannan Ross D. et al., In The Journal of Cell Biology, 1994
... Anti–trimethyl H3K9, anti–dimethyl H3K9, anti–trimethyl H3K4, anti–dimethyl H3K4, anti-H4, and anti-SNF2H antibodies were obtained from Abcam.
Papers on SWI
Chromatin remodelers Isw1 and Chd1 maintain chromatin structure during transcription by preventing histone exchange.
Workman et al., Kansas City, United States. In Nat Struct Mol Biol, 2012
Deletion of ISW1 and CHD1 causes widespread intragenic transcription.
Histone H3 lysine 36 methylation targets the Isw1b remodeling complex to chromatin.
Howe et al., Vancouver, Canada. In Mol Cell Biol, 2012
Histone H3 lysine 36 methylation promoted the chromatin interaction of the Isw1b chromatin-remodeling complex in Saccharomyces cerevisiae.
Nucleosome remodeler SNF2L suppresses cell proliferation and migration and attenuates Wnt signaling.
Becker et al., München, Germany. In Mol Cell Biol, 2012
The effect of SNF2L depletion on gene expression portray the cell in a state of activated Wnt signaling with increased proliferation and locomotion. High levels of SNF2L expression in normal melanocytes contrast with undetectable expression in melanoma.
Human CCAAT/enhancer-binding protein β interacts with chromatin remodeling complexes of the imitation switch subfamily.
Gutiérrez et al., Concepción, Chile. In Biochemistry, 2012
Functional interaction is characterized between C/EBPbeta and SNF2H/ACF1, involving mainly suppression of C/EBPbeta transactivation activity in the presence of SNF2H and ACF1.
Convergent structural alterations define SWItch/Sucrose NonFermentable (SWI/SNF) chromatin remodeler as a central tumor suppressive complex in pancreatic cancer.
Pollack et al., Stanford, United States. In Proc Natl Acad Sci U S A, 2012
expression profiling analyses revealed that SWI/SNF likely antagonizes Polycomb repressive complex 2, implicating this as one possible mechanism of tumor suppression
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