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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 08 Dec 2016.

Shugoshin-like 1

Sgo1, SGO, Shugoshin
Papers using Sgo1 antibodies
Protein phosphatase 6 regulates mitotic spindle formation by controlling the T-loop phosphorylation state of Aurora A bound to its activator TPX2
Gruneberg Ulrike et al., In The Journal of Cell Biology, 2009
... rabbit anti-Myc (both from Sigma-Aldrich), mouse anti-Plk1 (Santa Cruz Biotechnology, Inc.), mouse anti-PPP2CA (BD), mouse anti-Sgo1 (Abcam), rabbit anti-Sgo2 (Bethyl Laboratories, ...
Papers on Sgo1
Lentivirus-mediated siRNA interference targeting SGO-1 inhibits human NSCLC cell growth.
Zhang et al., Xi'an, China. In Tumour Biol, 2012
Lentivirus-mediated siRNA interference targeting SGO-1 inhibits human NSCLC cell growth
Bub1 kinase and Sgo1 modulate pericentric chromatin in response to altered microtubule dynamics.
Bloom et al., Chapel Hill, United States. In Curr Biol, 2012
Bub1 and Sgo1 act as a rheostat to regulate the chromatin spring and maintain force balance. Through histone H2A S121 phosphorylation and recruitment of Sgo1, Bub1 kinase softens the chromatin spring in response to changes in microtubule dynamics.
Haploinsufficiency of SGO1 results in deregulated centrosome dynamics, enhanced chromosomal instability and colon tumorigenesis.
Rao et al., Oklahoma City, United States. In Cell Cycle, 2012
Haploinsufficiency of SGO1 results in genomic instability manifested as missegregation of chromosomes and formation of extra centrosomal foci in both murine embryonic fibroblasts and adult bone marrow cells.
A novel tumor-derived SGOL1 variant causes abnormal mitosis and unstable chromatid cohesion.
Sugimura et al., Hamamatsu, Japan. In Oncogene, 2011
These results suggest that SGOL1-P1 may function as a negative factor to native SGOL1, and that abundant expression of SGOL1-P1 may be responsible for chromosomal instability.
Bub1, Sgo1, and Mps1 mediate a distinct pathway for chromosome biorientation in budding yeast.
Pellman et al., Boston, United States. In Mol Biol Cell, 2011
found that Sgo1 overexpression rescues defects caused by metaphase inactivation of Mps1 and that Mps1 is required for Sgo1 localization to the kinetochore
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