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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Aug 2016.

Scratch homolog 1, zinc finger protein

This gene is a member of the Snail family of C2H2-type zinc finger transcription factors. It codes for a neural-specific transcriptional repressor that binds to E-box motifs. The protein may promote neural differention and may be involved in cancers with neuroendocrine features. [provided by RefSeq, Jul 2008] (from NCBI)
Top mentioned proteins: CAN, catalase, HAD, V1a, OUT
Papers on Scratch
Bone morphogenetic protein 4 regulates microRNA expression in breast cancer cell lines in diverse fashion.
Kallioniemi et al., Tampere, Finland. In Genes Chromosomes Cancer, Mar 2016
Scratch assay with miR-16-5p and miR-106b-5p inhibitors on BMP4-treated MDA-MB-231 cells resulted in enhanced cell migration, suggesting that these miRNAs are engaged in BMP4-induced motility.
Aquaporin-3 Inhibition Reduces the Growth of NSCLC Cells Induced by Hypoxia.
Kuang et al., Harbin, China. In Cell Physiol Biochem, Feb 2016
Scratch-wound healing and transwell assays were used to detect cell migration and invasion.
Relationship between thin-film bond strength as measured by a scratch test, and indentation hardness for bonding agents.
Hotta et al., Gifu, Japan. In Dent Mater, Feb 2016
OBJECTIVES: To evaluate thin-film bond strength between a bonding agent and human dentin, using a scratch test, and the characteristics and accuracy of measurement.
VIRALpro: a tool to identify viral capsid and tail sequences.
Baldi et al., Rennes, France. In Bioinformatics, Feb 2016
AVAILABILITY AND IMPLEMENTATION: The data, software and corresponding web server are available from as part of the SCRATCH suite.
Identification of SNAIL1 Peptide-Based Irreversible Lysine-Specific Demethylase 1-Selective Inactivators.
Suzuki et al., Kyoto, Japan. In J Med Chem, Feb 2016
LSD1 interacts physically with SNAIL1, a member of the SNAIL/SCRATCH family of transcription factors.
Response to anti-VEGF-A treatment of endothelial cells in vitro.
Nicol├▓ et al., Genova, Italy. In Exp Eye Res, Feb 2016
Influence in would healing was evaluated through scratch assay.
Robust Quantitative Scratch Assay.
Jurisica et al., Toronto, Canada. In Bioinformatics, Jan 2016
UNASSIGNED: The wound healing assay (or scratch assay) is a technique frequently used to quantify the dependence of cell motility - a central process in tissue repair and evolution of disease - subject to various treatments conditions.
Cat-Scratch Disease: Case Report and Review of the Literature.
Biancone et al., Torino, Italy. In Transplant Proc, Sep 2015
Cat-scratch disease (CSD) is caused by Bartonella henselae and characterized by self-limited fever and granulomatous lymphadenopathy.
Optimization of the Wound Scratch Assay to Detect Changes in Murine Mesenchymal Stromal Cell Migration After Damage by Soluble Cigarette Smoke Extract.
Paxson et al., In J Vis Exp, 2014
Current methods for quantifying cell migration, including scratch assays, trans-well migration assays (Boyden chambers), micropillar arrays, and cell exclusion zone assays, possess a range of limitations in reproducibility, customizability, quantification, and cost-effectiveness.
Insect bite reactions.
Mann et al., Benares, India. In Indian J Dermatol Venereol Leprol, 2013
Papular urticaria is discussed including its epidemiology, the 5 stages of skin reaction, the SCRATCH principle as an aid in diagnosis, and the recent evidence supporting participation of types I, III, and IV hypersensitivity reactions in its causation is summarized.
The SNAIL family member SCRATCH1 is not expressed in human tumors.
Ansieau et al., Lyon, France. In Oncol Rep, 2010
SCRT1 transcripts are undetectable in nearly all of the examined tumors suggesting that SCRATCH1 plays a minor role, if any, in tumorigenesis.
Cat scratch disease presenting as acute mastoiditis.
Moxham et al., Canada. In Laryngoscope, 2009
OBJECTIVE: To present the first published case of Cat Scratch Disease presenting as acute mastoiditis and review the relevant literature to discuss the Otolaryngologic manifestations of this disease and its treatment.
A novel role for the Ste20 kinase SLK in adhesion signaling and cell migration.
Sabourin et al., Ottawa, Canada. In Cell Adh Migr, 2009
Scratch wounding of confluent monolayers results in SLK activation.
Functional gene variation in the human norepinephrine transporter: association with attention deficit hyperactivity disorder.
Kim et al., Belmont, United States. In Ann N Y Acad Sci, 2007
Further analyses identified Slug and Scratch as candidates involved in repression of SLC6A2 transcription generated by the -3081(T) allele.
The expression of Scratch genes in the developing and adult brain.
Nieto et al., San Juan de Alicante, Spain. In Dev Dyn, 2006
From perinatal to adult stages, Scratch1 remains strongly expressed in almost all brain regions, although it is not found in some ventral structures such as motor nuclei and hypothalamic regions.
The neurobiology of itch.
Schmelz et al., Kyoto, Japan. In Nat Rev Neurosci, 2006
Scratch-induced pain can abolish itch, and analgesic opioids can generate itch, which indicates an antagonistic interaction.
Prodromal itching in childhood asthma.
Hennessen et al., In Lancet, 1984
Scratch marks were seen in 5 patients.
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