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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 08 Dec 2016.

Kinetochore associated 1

ROD
This gene encodes a protein that is one of many involved in mechanisms to ensure proper chromosome segregation during cell division. Experimental evidence indicated that the encoded protein functioned in a similar manner to that of the Drosophila rough deal protein. [provided by RefSeq, Jul 2008] (from NCBI)
Top mentioned proteins: CD4, HAD, CAN, CD45, ACID
Papers on ROD
Therapeutic effect of CNP on renal osteodystrophy by antagonizing the FGF-23/MAPK pathway.
New
Qin et al., Hefei, China. In J Recept Signal Transduct Res, Nov 2015
UNASSIGNED: Renal osteodystrophy (ROD) is highly prevalent in chronic kidney disease (CKD).
Genome-Wide Analysis of Ocular Adnexal Lymphoproliferative Disorders Using High-Resolution Single Nucleotide Polymorphism Array.
New
Goto et al., Tokyo, Japan. In Invest Ophthalmol Vis Sci, Jun 2015
PURPOSE: We identified the genomic signature of ocular adnexal lymphoproliferative disorders (LPDs), especially ocular adnexal mucosa-associated lymphoid tissue (MALT) lymphoma, IgG4-related ophthalmic disease (IgG4-ROD), reactive lymphoid hyperplasia (RLH), and diffuse large B-cell lymphoma (DLBCL).
Synthesis and biological evaluation of DAPY-DPEs hybrids as non-nucleoside inhibitors of HIV-1 reverse transcriptase.
New
Daelemans et al., Shanghai, China. In Bioorg Med Chem, Mar 2015
A series of new DAPY-DPEs hybrids, combined the important pharmacophores of DAPYs and DPEs, has been synthesized and biologically evaluated for their anti-HIV activities against wild-type HIV-1 strain IIIB, double RT mutant (K103N+Y181C) strain RES056 and HIV-2 strain ROD in MT-4 cell cultures.
GABA(A) receptor subtype-selectivity of novel bicuculline derivatives.
Sieghart et al., Vienna, Austria. In Curr Med Chem, 2014
ROD 185 is a previously investigated structural analogue of the GABA site antagonist bicuculline, and a positive allosteric modulator acting via the benzodiazepine binding site.
Conserved determinants of lentiviral genome dimerization.
Summers et al., Baltimore, United States. In Retrovirology, 2014
We prepared RNAs corresponding to native and mutant forms of the 5' leaders of HIV-1 (NL4-3 strain), HIV-2 (ROD strain), and two divergent strains of simian immunodeficiency virus (SIV; cpz-TAN1 and -US strains), and probed for potential roles of the DIS and U5:AUG base pairing on intrinsic and NC-dependent dimerization by mutagenesis, gel electrophoresis, and NMR spectroscopy.
ROD-CONE DYSTROPHY ASSOCIATED WITH WILLIAMS SYNDROME.
Sadda et al., Los Angeles, United States. In Retin Cases Brief Rep, 2014
PURPOSE: To describe a case of rod-cone dystrophy associated with Williams syndrome.
HIV-2 infects resting CD4+ T cells but not monocyte-derived dendritic cells.
Schwartz et al., In Retrovirology, 2014
We focused on the requirement of Vpx for productive HIV-2 infection, using the reference HIV-2 ROD strain, the proviral clone GL-AN, as well as two primary HIV-2 isolates.
Aurora B kinase-dependent recruitment of hZW10 and hROD to tensionless kinetochores.
GeneRIF
Chan et al., Edmonton, Canada. In Curr Biol, 2008
Aurora B kinase activity is required for the accumulation of tension-sensitive mitotic-checkpoint components, such as ZW10 and ROD, in order to maintain mitotic-checkpoint arrest.
Renal osteodystrophy, phosphate homeostasis, and vascular calcification.
Review
Lund et al., Saint Louis, United States. In Semin Dial, 2007
New advances in the pathogenesis of renal osteodystrophy (ROD) change the perspective from which many of its features and treatment are viewed.
[Guideline for the management of chronic kidney disease-related mineral and bone disorders (CKD-MBD)].
Review
Kakuta, Funaishikawa, Japan. In Clin Calcium, 2007
Abnormalities of mineral and bone metabolism in patients with chronic kidney disease (CKD) have traditionally been assessed and managed in terms of renal osteodystrophy (ROD).
Development of renal bone disease.
Review
Ferreira, Lisbon, Portugal. In Eur J Clin Invest, 2006
Renal osteodystrophy (ROD) develops as the early stages of chronic renal failure (CRF) and covers a spectrum of bone changes observed in the uraemic patient, which extend from high remodelling bone disease (frequently known as osteitis fibrosa) to low turnover, or adynamic disease.
Minimizing bone abnormalities in children with renal failure.
Review
ZiĆ³lkowska, Warsaw, Poland. In Paediatr Drugs, 2005
Renal osteodystrophy (ROD), a metabolic bone disease accompanying chronic renal failure (CRF), is a major clinical problem in pediatric nephrology.
Role of parathyroid hormone and therapy with active vitamin D sterols in renal osteodystrophy.
Review
Salusky et al., Los Angeles, United States. In Semin Dial, 2005
Renal osteodystrophy (ROD) represents a spectrum of bone lesions ranging from a high-turnover to a low-turnover state.
The role of Drosophila CID in kinetochore formation, cell-cycle progression and heterochromatin interactions.
Impact
Karpen et al., Los Angeles, United States. In Nat Cell Biol, 2001
Deconvolution fluorescence microscopy showed that CID chromatin is physically separate from proteins involved in sister cohesion (MEI-S332), centric condensation (PROD), kinetochore function (ROD, ZW10 and BUB1) and heterochromatin structure (HP1).
Rough deal and Zw10 are required for the metaphase checkpoint in Drosophila.
Impact
Karess et al., Gif-sur-Yvette, France. In Nat Cell Biol, 2000
Here we show that the Drosophila kinetochore components Rough deal (Rod) and Zeste-White 10 (Zw10) are required for the proper functioning of the metaphase checkpoint in flies.
Human Zw10 and ROD are mitotic checkpoint proteins that bind to kinetochores.
Impact
Yen et al., Philadelphia, United States. In Nat Cell Biol, 2000
Here we show that human Zeste White 10 (Zw10) and Rough deal (Rod) are new components of the mitotic checkpoint, as cells lacking these proteins at kinetochores fail to arrest in mitosis when exposed to microtubule inhibitors.
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