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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Aug 2016.

Interferon-induced protein with tetratricopeptide repeats 3

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Top mentioned proteins: ISG54, ACID, STAT1, CAN, MxA
Papers using RIG-G antibodies
Thioredoxin reductase
Gorman Jeffrey J. et al., In Molecular & Cellular Proteomics : MCP, 1999
... included a mouse monoclonal antibody to SOD2, a mouse polyclonal antiserum to interferon-induced protein with tetratricopeptide repeats 3 (IFIT3) obtained from Abcam (Cambridge, UK), rabbit polyclonal ...
Papers on RIG-G
RIG-G inhibits the proliferation of NB4 cells and propels ATRA-induced differentiation of APL cells.
Tong et al., Shanghai, China. In Leuk Res, Jan 2016
RIG-G (retinoic acid-induced gene G) was originally identified in ATRA (all-trans retinoic acid)-treated NB4 acute promyelocytic leukemia (APL) cells.
Genome-wide transcriptional profiling reveals that HIV-1 Vpr differentially regulates interferon-stimulated genes in human monocyte-derived dendritic cells.
Aida et al., Wako, Japan. In Virus Res, Nov 2015
The differential expression profiles of select genes involved in innate immune responses (interferon-stimulated genes [ISGs]), including MX1, MX2, ISG15, ISG20, IFIT1, IFIT2, IFIT3, IFI27, IFI44L, and TNFSF10, were validated by real-time quantitative PCR; the results were consistent with the microarray data.
Global transcriptomic profiling of bovine endometrial immune response in vitro. II. Effect of bovine viral diarrhea virus on the endometrial response to lipopolysaccharide.
Wathes et al., United Kingdom. In Biol Reprod, Oct 2015
Top down-regulated genes were RSAD2, ISG15, BST2, MX2, OAS1, USP18, IFIT3, IFI27, SAMD9, IFIT1, and DDX58, whereas TRIM56, C3, and OLFML1 were most up-regulated.
Differential Antigen-presenting B Cell Phenotypes from Synovial Microenvironment of Patients with Rheumatoid and Psoriatic Arthritis.
Díaz-González et al., Moldova. In J Rheumatol, Oct 2015
Expression of interferon-induced protein with tetratricopeptide repeats 4 (IFIT4) in immune-selected CD20+ cells from patients with RA was assessed by quantitative realtime PCR.
Interferon-stimulated gene (ISG) 60, as well as ISG56 and ISG54, positively regulates TLR3/IFN-β/STAT1 axis in U373MG human astrocytoma cells.
Tanaka et al., Hirosaki, Japan. In Neurosci Res, Oct 2015
In the present study, we demonstrated that poly IC induces the expression of ISG60, also named as IFIT3, in U373MG cells.
Microparticles released from Mycobacterium tuberculosis-infected human macrophages contain increased levels of the type I interferon inducible proteins including ISG15.
Saunders et al., Sydney, Australia. In Proteomics, Sep 2015
Prominent among these were the type I interferon inducible proteins, ISG15, IFIT1, IFIT2, and IFIT3.
The metabolomics of asthma control: a promising link between genetics and disease.
Lasky-Su et al., Boston, United States. In Immun Inflamm Dis, Sep 2015
Integrative ORA identified 17 significantly enriched pathways related to cellular immune response, interferon signaling, and cytokine-related signaling, for which arachidonic acid, PGE2 and S1P, in addition to six genes (CHN1, PRKCE, GNA12, OASL, OAS1, and IFIT3) appeared to drive the pathway results.
Murine Bone Marrow-Derived Dendritic Cells Transduced by Light-Helper-Dependent Herpes Simplex Virus-1 Amplicon Vector Acquire a Mature Dendritic Cell Phenotype.
Mavromara et al., Athens, Greece. In Hum Gene Ther Methods, Jun 2015
Further comparisons of the gene expression of infected DCs showed that while interferon-stimulated genes such as Ifit1, Ifit3, Mx2, Isg15, and Cxcl10 were induced by both BD and LHD amplicon vectors, early proinflammatory cytokine gene expression (Tnfa, Il1a, Il1b, Il6, Il10, Il12b, Cxcl1, and Cxcl16) and DC maturation were mediated only by the LHD amplicons.
[Inhibition of replication and transcription of WSN influenza A virus by IFIT family genes].
Liu et al., In Sheng Wu Gong Cheng Xue Bao, 2015
Further study reveals that overexpression of IFIT2 or IFIT3 could inhibit IAV replication and transcription, and cause the dose-dependent inhibition of polymerase activity of vRNP.
Gene Expression Profiling Identifies Interferon Signalling Molecules and IGFBP3 in Human Degenerative Annulus Fibrosus.
Grad et al., Galway, Ireland. In Sci Rep, 2014
This study indicates interferon-alpha signalling pathway activation with IFIT3 and IGFBP3 up-regulation, which may affect cellular function in human degenerative disc.
The impact of PPARα activation on whole genome gene expression in human precision cut liver slices.
Kersten et al., Wageningen, Netherlands. In Bmc Genomics, 2014
IFITM1, IFIT1, IFIT2, IFIT3) and numerous other immune-related genes (e.g.
A death-promoting role for ISG54/IFIT2.
Reich, Stony Brook, United States. In J Interferon Cytokine Res, 2013
ISG54/IFIT2 does not function as a monomer, and it forms complexes with itself and with the related ISG56/IFIT1 and ISG60/IFIT3 proteins to elicit complex cellular responses.
Interferon induced IFIT family genes in host antiviral defense.
Jiang et al., Pullman, United States. In Int J Biol Sci, 2012
Most mammals have IFIT1, IFIT2, IFIT3 and IFIT5; however, bird, marsupial, frog and fish have only IFIT5.
Translating the brain transcriptome in neuroAIDS: from non-human primates to humans.
Fox et al., Omaha, United States. In J Neuroimmune Pharmacol, 2012
Differential expression of mRNAs involved in inflammation and immune response are a common finding in both monkey and human samples - even in HIV infected people on combination antiretroviral therapy, a shared set of genes is upregulated in the brains of both infected monkeys and humans: B2M, IFI44, IFIT3, MX1, STAT1.
IFN-induced TPR protein IFIT3 potentiates antiviral signaling by bridging MAVS and TBK1.
Wang et al., Shanghai, China. In J Immunol, 2011
our study characterizes IFIT3 as an important modulator in innate immunity
Intact JAK-STAT signaling pathway is a prerequisite for STAT1 to reinforce the expression of RIG-G gene.
Tong et al., Shanghai, China. In Exp Cell Res, 2011
concluded that the expression of RIG-G was independent on the classical JAK-STAT pathway, but could be greatly increased by it.
The ISG56/IFIT1 gene family.
Sen et al., Cleveland, United States. In J Interferon Cytokine Res, 2011
The ISG56/IFIT1 family of genes is clustered on human chromosome 10 and is comprised of 4 members, ISG56/IFIT1, ISG54/IFIT2, ISG60/IFIT3, and ISG58/IFIT5, whose homologs are evolutionarily conserved from mammals to amphibians.
Identification of alpha interferon-induced genes associated with antiviral activity in Daudi cells and characterization of IFIT3 as a novel antiviral gene.
Zoon et al., Bethesda, United States. In J Virol, 2010
Identification of alpha interferon-induced genes associated with antiviral activity in Daudi cells and characterization of IFIT3 as a novel antiviral gene.
[Role of the interferon-stimulated response elements I/II in expression regulation of the retinoic acid induced gene G].
Tong et al., Shanghai, China. In Zhonghua Yi Xue Yi Chuan Xue Za Zhi, 2010
transcription factors for the reporter gene. CONCLUSION: Both ISRE I and ISRE II on the RIG-G promoter are the binding sites for the complex of transcription factors. They are required for RIG-G expression, and ISRE I has a preferential role over ISRE II.
[A novel molecular mechanism of interferon alpha-regulated expression of retinoic acid-induced gene G].
Tong et al., Shanghai, China. In Zhonghua Zhong Liu Za Zhi, 2010
The complex STAT2/IRF-9 is the key factor mediating the expression of RIG-G gene regulated by IFN-alpha.
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