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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Aug 2016.


repetin, Rptn
Top mentioned proteins: S-100, Thl, LOR, Filaggrin, Involucrin
Papers on repetin
Diminished serum repetin levels in patients with schizophrenia and bipolar disorder.
Zhao et al., Xi'an, China. In Sci Rep, 2014
Repetin (RPTN) protein is a member of S100 family and is known to be expressed in the normal epidermis.
Hair follicle disruption facilitates pathogenesis to UVB-induced cutaneous inflammation and basal cell carcinoma development in Ptch(+/-) mice.
Athar et al., Birmingham, United States. In Am J Pathol, 2014
Expression of epidermal IL1f6, S100a8, vitamin D receptor, repetin, and major histocompatibility complex II, biomarkers depicting susceptibility to cutaneous inflammation, was also higher.
Development of an ichthyosiform phenotype in Alox12b-deficient mouse skin transplants.
Krieg et al., Heidelberg, Germany. In J Invest Dermatol, 2009
Western blot analysis revealed restoration of profilaggrin processing that used to be disturbed in neonatal mutant skin and overexpression of filaggrin, involucrin, and repetin.
Disruption of epidermal specific gene expression and delayed skin development in AP-2 gamma mutant mice.
Winger et al., Logan, United States. In Dev Biol, 2008
Summary Sentence: Conditional ablation of AP-2 gamma results in a delay in skin development and abnormal expression of p63, K14, K1, filaggrin, repetin and secreted Ly6/Plaur domain containing 1, key genes required for epidermal development and differentiation.
Effects of fibroblasts and microenvironment on epidermal regeneration and tissue function in long-term skin equivalents.
Stark et al., Heidelberg, Germany. In Eur J Cell Biol, 2007
These SEs demonstrate sustained epidermal viability - over 12 weeks - with regular differentiation as substantiated by in vivo-like patterns of all differentiation products, exemplified here by the cornified envelope components loricrin and repetin.
Up-regulation of loricrin expression by cell adhesion molecule nectin-1 through Rap1-ERK signaling in keratinocytes.
Takai et al., Suita, Japan. In J Biol Chem, 2007
The amounts of repetin and SPRRP, other components of cornified cell envelopes, were markedly elevated probably due to a compensatory mechanism to overcome the impaired expression of loricrin.
Isolation and characterization of human repetin, a member of the fused gene family of the epidermal differentiation complex.
Hohl et al., Lausanne, Switzerland. In J Invest Dermatol, 2005
The human repetin gene is a member of the "fused" gene family and localized in the epidermal differentiation complex on chromosome 1q21.
Cornulin, a new member of the "fused gene" family, is expressed during epidermal differentiation.
Hohl et al., Lausanne, Switzerland. In J Invest Dermatol, 2005
This protein, however, has the previously unreported structural features of the "fused gene" family combining sequences and structural similarities of both the S100 proteins and precursor proteins of the cornified cell envelope as in profilaggrin, trichohyalin, and repetin.
Trichohyalin mechanically strengthens the hair follicle: multiple cross-bridging roles in the inner root shealth.
Marekov et al., Bethesda, United States. In J Biol Chem, 2003
Second, trichohyalin serves as a cross-bridging reinforcement protein of the cornified cell envelope of the inner root sheath cells by becoming cross-linked to several known or novel barrier proteins, including involucrin, small proline-rich proteins, repetin, and epiplakin.
Permeability barrier dysfunction in transgenic mice overexpressing claudin 6.
Troy et al., Ottawa, Canada. In Development, 2002
Barrier dysfunction was manifested biochemically by the aberrant expression of late epidermal differentiation markers, including K1, filaggrin, loricrin, transglutaminase 3, involucrin, repetin, members of the SPRR family and the transcriptional regulator Klf4.
Lessons from loricrin-deficient mice: compensatory mechanisms maintaining skin barrier function in the absence of a major cornified envelope protein.
Roop et al., Houston, United States. In J Cell Biol, 2000
At least one of the compensatory mechanisms preventing a more severe skin phenotype in newborn Lor(-/-) mice is an increase in the expression of other CE components, such as SPRRP2D and SPRRP2H, members of the family of "small proline rich proteins", and repetin, a member of the "fused gene" subgroup of the S100 gene family.
Repetin (Rptn), a new member of the "fused gene" subgroup within the S100 gene family encoding a murine epidermal differentiation protein.
Marks et al., Heidelberg, Germany. In Genomics, 1997
We report the cloning and characterization of a murine epidermal differentiation gene, repetin (Rptn), exhibiting striking similarity to the genes of the intermediate filament-associated proteins profilaggrin and trichohyalin.
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