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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Aug 2016.

Avian reticuloendotheliosis viral

RelB, Transcription Factor RelB, Irel
Top mentioned proteins: p65, NF-kappaB, p52, Rel, CAN
Papers using RelB antibodies
Functions of nuclear factor kappaB in bone.
Agarwal Sudha, In PLoS ONE, 2009
... Polyclonal anti- RelB antibody was purchased from Santa Cruz Biotechnology Inc (Santa Cruz, CA) ...
Noncanonical NF-kappaB activation requires coordinated assembly of a regulatory complex of the adaptors cIAP1, cIAP2, TRAF2 and TRAF3 and the kinase NIK
Guttridge Denis C. et al., In The Journal of Cell Biology, 2007
... and COX IV were purchased from BD (556433), RelB (sc-226), TRAF3 (M20), p100 (K27), and Pol II (sc-899X) were purchased from Santa Cruz Biotechnology, Inc., myosin forms MyHC ...
Proteasome inhibitor PS-341 induces apoptosis in cisplatin-resistant squamous cell carcinoma cells by induction of noxa
Miyamoto Shigeki et al., In Molecular Cancer, 2005
... p65 (C20) and RelB (C19) antibodies were purchased from Santa Cruz Biotechnology (Santa Cruz, CA), p50 ...
Deletion of IFT20 in the mouse kidney causes misorientation of the mitotic spindle and cystic kidney disease
Pazour Gregory J. et al., In The Journal of Cell Biology, 2001
... (clone 8E7, 1:300; Upstate Biotechnology), glyceraldehyde 3-phosphate dehydrogenase (GAPDH; clone 14C10, 1:1,000; Cell Signaling Technology), RelB (N-17, 1:250; Santa Cruz Biotechnology, Inc.) ...
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Papers on RelB
Non-canonical pathway network modelling and ubiquitination site prediction through homology modelling of NF-κB.
Febin Prabhu Dass et al., Vellore, India. In Gene, Feb 2016
Unlike the Classical pathway, due to absence of parallel studies for p100/RelB, a quality match could not be performed, but future studies are in pipeline to replicate the methodology for NF-κB1 activation and compare the results with existing observations.
Human T-cell leukemia virus type 1 (HTLV-1) Tax1 oncoprotein but not HTLV-2 Tax2 induces the expression of OX40 ligand by interacting with p52/p100 and RelB.
Fujii et al., Niigata, Japan. In Virus Genes, Feb 2016
To induce the OX40L expression, Tax1 but not Tax2 was observed to interact with NF-κB2/p100/p52 and RelB and the distinct interaction activity was mediated by the Tax1 amino acid region of 225-232.
Eva1 Maintains the Stem-like Character of Glioblastoma-Initiating Cells by Activating the Noncanonical NF-κB Signaling Pathway.
Kondo et al., Sapporo, Japan. In Cancer Res, Feb 2016
We further demonstrate that Eva1 induced GIC proliferation through the activation of the RelB-dependent noncanonical NF-κB pathway by recruiting TRAF2 to the cytoplasmic tail.
[Role of glycogen synthase kinase 3β in maturation and function of murine myeloid dendritic cells in vitro].
Qiu et al., Guangzhou, China. In Nan Fang Yi Ke Da Xue Xue Bao, Jan 2016
The expression of GSK-3β and RelB was examined by Western blotting in DC2.4 cells transfected with a lentiviral vector over-expressing murine GSK-3β gene.
Mono-(2-Ethylhexyl) Phthalate Promotes Pro-Labor Gene Expression in the Human Placenta.
Rosen et al., New Brunswick, United States. In Plos One, Dec 2015
Placental corticotropin-releasing hormone (CRH) and cyclooxygenase-2 (COX-2) are key mediators of parturition and are regulated by the non-canonical NF-kB (RelB/p52) signaling pathway.
NEMO Prevents Steatohepatitis and Hepatocellular Carcinoma by Inhibiting RIPK1 Kinase Activity-Mediated Hepatocyte Apoptosis.
Pasparakis et al., Köln, Germany. In Cancer Cell, Dec 2015
Here, we show that complete NF-κB inhibition by combined LPC-specific ablation of RelA, c-Rel, and RelB did not phenocopy NEMO deficiency, but constitutively active IKK2-mediated NF-κB activation prevented hepatocellular damage and HCC in NEMO(LPC-KO) mice.
The necroptosis adaptor RIPK3 promotes injury-induced cytokine expression and tissue repair.
Chan et al., Worcester, United States. In Immunity, 2014
These effects were caused by impaired NF-κB subunit RelB and p50 activation and by impaired caspase 1-mediated processing of interleukin-1β (IL-1β).
NF-κB pathways in hematological malignancies.
Zauli et al., Trieste, Italy. In Cell Mol Life Sci, 2014
In this review, we describe NF-κB and its two pathways: the canonical pathway (RelA/p50) and the non-canonical pathway (RelB/p50 or RelB/p52).
α-catenin acts as a tumour suppressor in E-cadherin-negative basal-like breast cancer by inhibiting NF-κB signalling.
Ma et al., Houston, United States. In Nat Cell Biol, 2014
This stabilization in turn prevents nuclear localization of RelA and p50, leading to decreased expression of TNF-α, IL-8 and RelB.
NF-κB signaling regulates myelination in the CNS.
Prinz et al., Freiburg, Germany. In Front Mol Neurosci, 2013
A key transcription factor involved in these processes is nuclear factor-κB (NF-κB), a hetero or homodimer of the Rel family of proteins, including p65, c-Rel, RelB, p50, and p52.
RelB: an outlier in leukocyte biology.
Yoza et al., Winston-Salem, United States. In J Leukoc Biol, 2013
RelB is one of the more unusual members of the NF-κB family.
Control of RelB during dendritic cell activation integrates canonical and noncanonical NF-κB pathways.
Hoffmann et al., San Diego, United States. In Nat Immunol, 2012
The NF-κB protein RelB controls dendritic cell (DC) maturation and may be targeted therapeutically to manipulate T cell responses in disease.
Lymphotoxin-β receptor signaling through NF-κB2-RelB pathway reprograms adipocyte precursors as lymph node stromal cells.
Caamaño et al., Birmingham, United Kingdom. In Immunity, 2012
This effect involved activation of the NF-κB2-RelB signaling pathway and inhibition of the expression of the key adipogenic factors Pparγ and Cebpα.
The role of NF-κB activation during protection against Leishmania infection.
Visekruna et al., Marburg an der Lahn, Germany. In Int J Med Microbiol, 2012
Since the discovery of the five members of the NF-κB transcription factor family, RelA, c-Rel, RelB, p50 and p52, considerable progress has been made toward better understanding how the different NF-κB homo- and heterodimers regulate such distinct subsets of target genes.
Hypercapnia induces cleavage and nuclear localization of RelB protein, giving insight into CO2 sensing and signaling.
Cummins et al., Dublin, Ireland. In J Biol Chem, 2012
RelB is a CO(2)-sensitive NF-kappaB family member that may contribute to the beneficial effects of hypercapnia in inflammatory diseases of the lung
The IKKα-dependent NF-κB p52/RelB noncanonical pathway is essential to sustain a CXCL12 autocrine loop in cells migrating in response to HMGB1.
Marcu et al., Stony Brook, United States. In J Immunol, 2012
The RelB noncanonical pathway is critical to sustain CXCL12/SDF1 production in order for cells to migrate toward chromatin architectural protein HMGB1.
Nuclear factor κB subunits RelB and cRel negatively regulate Toll-like receptor 3-mediated β-interferon production via induction of transcriptional repressor protein YY1.
Moynagh et al., Ireland. In J Biol Chem, 2012
Nuclear factor kappaB subunits RelB and cRel negatively regulate Toll-like receptor 3-mediated beta-interferon production via induction of transcriptional repressor protein YY1.
Inverse relationship between PSA and IL-8 in prostate cancer: an insight into a NF-κB-mediated mechanism.
St Clair et al., Lexington, United States. In Plos One, 2011
RelB plays a critical role in the response of PCa to radiotherapy and the inverse expression of IL-8 and PSA
Nuclear factor kappa B activation occurs in the amnion prior to labour onset and modulates the expression of numerous labour associated genes.
Bennett et al., London, United Kingdom. In Plos One, 2011
women entering labor exhibit low or moderate NFkappaB activition, the process does not appear to involve classical pathways of NFkappaB activation but rather is correlated with an increase in nuclear p65-Rel-B dimers.
NF-κB as a target for modulating inflammatory responses.
Feldmann et al., Trieste, Italy. In Curr Pharm Des, 2011
The review describes NF-κB and its two pathways: the canonical, mediated by the p65 and p50 subunits, and the non-canonical, mediated by the subunits RelB, p52 and p50.
More papers using RelB antibodies
The combined absence of the transcription factors Rel and RelA leads to multiple hemopoietic cell defects
Joyce David A et al., In Arthritis Research & Therapy, 1998
... Rabbit anti-IκBα (C-21) polyclonal antibody, anti-NF-κB p65, anti-NF-κB p50, anti-c-Rel and anti-RelB were obtained from Santa Cruz Biotechnology, Santa Cruz, CA, USA ...
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