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RAN binding protein 17

RANBP17
The transport of protein and large RNAs through the nuclear pore complexes (NPC) is an energy-dependent and regulated process. The import of proteins with a nuclear localization signal (NLS) is accomplished by recognition of one or more clusters of basic amino acids by the importin-alpha/beta complex; see MIM 600685 and MIM 602738. The small GTPase RAN (MIM 601179) plays a key role in NLS-dependent protein import. RAN-binding protein-17 is a member of the importin-beta superfamily of nuclear transport receptors.[supplied by OMIM, Jul 2002] (from NCBI)
Top mentioned proteins: HOX11L2, Rbx1, Cryptic, FGF18, p21
Papers on RANBP17
Analysis of Stage-Specific Gene Expression Profiles in the Uterine Endometrium during Pregnancy in Pigs.
Ka et al., Wŏnju, South Korea. In Plos One, 2014
Furthermore, several pregnancy-related hub genes such as ALPPL2, RANBP17, NF1B, SPP1, and CST6 were discovered through network analysis.
Heart failure entails significant changes in human nucleocytoplasmic transport gene expression.
Rivera et al., Valencia, Spain. In Int J Cardiol, 2013
XPO1, GABPB2, and RANBP17 were upregulated, while KALRN was downregulated in both DCM and ICM, and XPO5 only in DCM.
RANBP17 is localized to the XY body of spermatocytes and interacts with SPEM1 on the manchette of elongating spermatids.
Yan et al., Reno, United States. In Mol Cell Endocrinol, 2011
We identified Ran-binding protein 17 (RANBP17) as one of the interacting partners of sperm maturation 1 (SPEM1) using yeast 2-hybrid screening and immunoprecipitation assays.
Identification of RANBP16 and RANBP17 as novel interaction partners for the bHLH transcription factor E12.
GeneRIF
Smas et al., Toledo, United States. In J Cell Biochem, 2010
These biochemical and functional data reveal RANBP16 and RANBP17 as novel regulators of E2A protein action, and demonstrate specific interaction of E12 with RANBP17.
Mapping of 5q35 chromosomal rearrangements within a genomically unstable region.
Speleman et al., In J Med Genet, 2008
RESULTS AND CONCLUSION: Five of the breakpoints were located within an interval of approximately 265 kb encompassing the RANBP17 and TLX3 genes.
HOX11L2/TLX3 is transcriptionally activated through T-cell regulatory elements downstream of BCL11B as a result of the t(5;14)(q35;q32).
Penard-Lacronique et al., Paris, France. In Blood, 2007
It affects the BCL11B/CTIP2 locus on chromosome 14 and the RANBP17-TLX3/HOX11L2 region on chromosome 5.
Holoprosencephaly and preaxial polydactyly associated with a 1.24 Mb duplication encompassing FBXW11 at 5q35.1.
de Vries et al., Nijmegen, Netherlands. In J Hum Genet, 2005
The duplicated region encompasses seven genes: RANBP17, TLX3, NPM1, FGF18, FBXW11, STK10, and DC-UbP.
Various types of rearrangements target TLX3 locus in T-cell acute lymphoblastic leukemia.
Bernard et al., Paris, France. In Genes Chromosomes Cancer, 2004
In two additional instances, fusion of the BCL11B (also known as CTIP2) and RANBP17/TLX3 loci were shown to result from subtle genomic insertion/deletion within these loci.
t(5;14)/HOX11L2-positive T-cell acute lymphoblastic leukemia. A collaborative study of the Groupe Français de Cytogénétique Hématologique (GFCH).
Groupe Français de Cytogénétique Hématologique (GFCH) et al., Paris, France. In Leukemia, 2003
Involvement of the RANBP17/HOX11L2 locus was ascertained by fluorescence in situ hybridization in six variant or alternative (three-way translocation or cytogenetic partner other than 14q32) translocations out of the 223 patients.
Activation of HOX11L2 by juxtaposition with 3'-BCL11B in an acute lymphoblastic leukemia cell line (HPB-ALL) with t(5;14)(q35;q32.2).
Drexler et al., Braunschweig, Germany. In Genes Chromosomes Cancer, 2003
Expression of both BCL11B, which is hematologically restricted to T cells, and HOX11L2 was detected, whereas adjacent genes at 5q35 (RANBP17) and 14q32 (VRK1, HSU88895) were not dysregulated.
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