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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 08 Dec 2016.

Protein S

proteins A, PROS1, PROS, a protein S
This gene encodes a vitamin K-dependent plasma protein that functions as a cofactor for the anticoagulant protease, activated protein C (APC) to inhibit blood coagulation. It is found in plasma in both a free, functionally active form and also in an inactive form complexed with C4b-binding protein. Mutations in this gene result in autosomal dominant hereditary thrombophilia. An inactive pseudogene of this locus is located at an adjacent region on chromosome 3. [provided by RefSeq, Feb 2009] (from NCBI)
Papers on proteins A
Small and large PROS1 deletions but no other types of rearrangements detected in patients with protein S deficiency.
GeneRIF
Halldén et al., Kristianstad, Sweden. In Thromb Haemost, 2012
mutation deletions but no other types of rearrangements detected in patients with protein S deficiency
Non-severe allergic asthma is associated with elevated plasma protein C and protein S.
GeneRIF
Okumura et al., In Thromb Haemost, 2012
Letter: non-severe allergic asthma is associated with elevated plasma protein C and protein S.
PROS1 Heerlen polymorphism is associated with increased free plasma tissue factor pathway inhibitor levels.
GeneRIF
Mulder et al., In Thromb Haemost, 2012
genetic polymorphism is associated with increased free plasma tissue factor pathway inhibitor levels
Platelet-mediated proteolytic down regulation of the anticoagulant activity of protein S in individuals with haematological malignancies.
GeneRIF
Brinkman et al., Amsterdam, Netherlands. In Thromb Haemost, 2012
proteolytic cleavage is increased in patients with essential thrombocythaemia and reduced in patients with chemotherapy-induced thrombocytopenia
Genotype and laboratory and clinical phenotypes of protein s deficiency.
GeneRIF
Spannagl et al., München, Germany. In Am J Clin Pathol, 2012
Persistently low protein S activity levels are highly indicative of a genetic alteration in PROS1.
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