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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 11 Dec 2014.

Casein kinase 1, delta

Protein Kinase
Top mentioned proteins: CAN, MAPK, V1a, Akt, ACID
Papers on Protein Kinase
Effect of Hyperketonemia (Acetoacetate) on Nuclear Factor-κB and p38 Mitogen-Activated Protein Kinase Activation Mediated Intercellular Adhesion Molecule 1 Upregulation in Endothelial Cells.
Jain et al., Shreveport, United States. In Metab Syndr Relat Disord, 09 Jan 2015
We examined the hypothesis that hyperketonemia activates the nuclear factor-κB (NF-κB) and mitogen-activated protein kinase (MAPK) signaling pathways that regulate intercellular adhesion molecule 1 (ICAM-1) expression in endothelial cells.
Prenatal Testosterone Exposure Induces Hypertension in Adult Females via Androgen Receptor-Dependent Protein Kinase Cδ-Mediated Mechanism.
Sathishkumar et al., Houston, United States. In Hypertension, 08 Jan 2015
We tested whether androgens induce hypertension through transcriptional regulation and signaling of protein kinase C (PKC) in the mesenteric arteries.
Calcium/Calmodulin-Dependent Protein Kinase II Couples Wnt Signaling With Histone Deacetylase 4 and Mediates Dishevelled-Induced Cardiomyopathy.
Hardt et al., Heidelberg, Germany. In Hypertension, 08 Jan 2015
Recently, calcium/calmodulin-dependent protein kinase II (CaMKII) was reported to be a pivotal participant in myocardial remodeling.
Protein Kinase C Promotes Cardiac Fibrosis and Heart Failure by Modulating Galectin-3 Expression.
Sheng et al., Harbin, China. In Biochim Biophys Acta, 06 Jan 2015
UNLABELLED: Protein kinase C (PKC) and galectin-3 are two important mediators that play a key pathogenic role in cardiac hypertrophy and heart failure (HF).
Tetra-Substituted Pyridinylimidazoles As Dual Inhibitors of p38α Mitogen-Activated Protein Kinase and c-Jun N-Terminal Kinase 3 for Potential Treatment of Neurodegenerative Diseases.
Laufer et al., Tübingen, Germany. In J Med Chem, 05 Jan 2015
UNLABELLED: Tetra-substituted imidazoles were designed as dual inhibitors of c-Jun N-terminal kinase (JNK) 3 and p38α mitogen-activated protein (MAP) kinase.
The Two Faces of Receptor Interacting Protein Kinase-1.
Green et al., Memphis, United States. In Mol Cell, 20 Dec 2014
UNLABELLED: Receptor Interacting Protein Kinase-1 (RIPK1), a key player in inflammation and cell death, assumes opposite functions depending on the cellular context and its posttranslational modifications.
Mitogen Activated Protein Kinase Family Proteins and c-jun Signaling in Injury-induced Schwann Cell Plasticity.
Park et al., Pusan, South Korea. In Exp Neurobiol, Jun 2014
In this review, we focus on the role of mitogen activated protein kinase family proteins (MAP kinases) in SC dedifferentiation.
A Review of Signal Transduction of Endothelin-1 and Mitogen-activated Protein Kinase-related Pain for Nanophysiotherapy.
Kim et al., South Korea. In J Phys Ther Sci, May 2014
In this review, we summarize the mechanisms of endothelin-1 (ET-1)- and mitogen-activated protein kinase (MAPK)-related pain, and suggest their applications in pain physiotherapy.
Role of Protein Kinase C in Podocytes and Development of Glomerular Damage in Diabetic Nephropathy.
Schiffer et al., Hannover, Germany. In Front Endocrinol (lausanne), Dec 2013
This review focuses on the role of the protein kinase C (PKC) family in podocytes and points out the differential roles of classical, novel, and atypical PKCs in podocytes.
Mitogen-Activated Protein Kinase Signal Transduction in Solid Tumors.
Wang et al., Nanchang, China. In Asian Pac J Cancer Prev, Dec 2013
UNLABELLED: Mitogen-activated protein kinase (MAPK) is an important signaling pathway in living beings in response to extracellular stimuli.
Ets and GATA Transcription Factors Play a Critical Role in PMA-Mediated Repression of the ckβ Promoter via the Protein Kinase C Signaling Pathway.
Few et al., Malaysia. In Plos One, Dec 2013
BACKGROUND: Choline kinase is the most upstream enzyme in the CDP-choline pathway.
Drosophila and vertebrate casein kinase Idelta exhibits evolutionary conservation of circadian function.
Price et al., Kansas City, United States. In Genetics, 2009
The results demonstrate a high degree of evolutionary conservation of fly and vertebrate CKIdelta and of the functions affected by their period-shortening mutations.
Drosophila Smoothened phosphorylation sites essential for Hedgehog signal transduction.
Tomlinson et al., New York City, United States. In Nat Cell Biol, 2005
Together, Protein Kinase A, Casein Kinase 1 and Glycogen Synthase Kinase 3 silence the pathway in the absence of ligand by phosphorylating Ci at a defined cluster of sites, thereby promoting its proteolytic conversion to a transcriptional repressor (Ci-75).
Proteolysis of the Hedgehog signaling effector Cubitus interruptus requires phosphorylation by Glycogen Synthase Kinase 3 and Casein Kinase 1.
Kalderon et al., New York City, United States. In Cell, 2002
Ci-155 proteolysis depends on phosphorylation at three sites by Protein Kinase A (PKA).
Protein kinase D regulates the fission of cell surface destined transport carriers from the trans-Golgi network.
Malhotra et al., San Diego, United States. In Cell, 2001
When a kinase inactive form of Protein Kinase D (PKD-K618N) was expressed in HeLa cells, it localized to the trans-Golgi network (TGN) and caused extensive tubulation.
Analogs of cyclic adenosine monophosphate: correlation of inhibition of Purkinje Neurons with Protein Kinase Activation.
Henriksen et al., In Science, 1975
with their reported potency in activating cyclic AMP-dependent protein kinase.
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