In Hastings Cent Rep, Jan 2016
That trend in funding and interest is reflected, to some degree, in the second article, in which Oscar Zarate and colleagues explore how the Personal Genome Project, an endeavor launched at Harvard University to collect and share personal genetic and health information in order to facilitate research, is perceived by the people who consent to be part of it.
The Marin Lab at the Dawn of Cognitive Neuropsychology.
Moss, Norway. In Cogn Behav Neurol, Sep 2015
This essay discusses the intellectual developments in psychology, linguistics, and behavioral neurology that shaped Oscar Marin's approach to disorders of high cortical function.
Neuropsychological Thoughts, Then and Now: A Tribute to Oscar Marin.
Cambridge, United Kingdom. In Cogn Behav Neurol, Sep 2015
This brief paper, inspired by an invitation to acknowledge and celebrate Oscar Marin's great contributions to cognitive neurology and neuropsychology, reviews the case of a patient, T.P., who had significant deficits of naming, reading, and spelling.
Oscar Marin and the Creation of a Cognitive Neuropsychology Laboratory.
Eugene, United States. In Cogn Behav Neurol, Sep 2015
Created and headed by Oscar Marin and the author, in affiliation with the University of Oregon, the lab brought together students, fellows, and visiting experts in neurology, psychology, psychiatry, neuropsychology, neurobiology, neurophysiology, and computation.
The controversy of sports technology: a systematic review.
Poole, United Kingdom. In Springerplus, 2014
Whilst the sport of golf had the highest number of case studies, it was found that a significant level of attention had been recorded in the cases of Oscar Pistorius use of prosthetic legs in athletics and the use of full length swimsuits in swimming.
Chorea: A Journey through History.
Juiz de Fora, Brazil. In Tremor Other Hyperkinet Mov (n Y), 2014
In 1841, Charles Oscar Waters gave a concise account of a syndrome, likely to be Huntington's disease (HD), later described further by George Huntington and named after him.
Serum amyloid A inhibits RANKL-induced osteoclast formation.
Suwŏn, South Korea. In Exp Mol Med, 2014
SAA not only markedly blocked the expression of several osteoclast-associated genes (TNF receptor-associated factor 6 and osteoclast-associated receptor) but also strongly induced the expression of negative regulators (MafB and interferon regulatory factor 8).