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Family with sequence similarity 129, member A

Niban, C1orf24
expressed in renal carcinomas in Tsc2 gene mutant Eker rats but is not expressed in normal kidney; may be a marker for early stage renal carcinogenesis [RGD, Feb 2006] (from NCBI)
Top mentioned proteins: POLYMERASE, TMC, CAN, V1a, SET
Papers on Niban
microRNA-106b-mediated down-regulation of C1orf24 expression induces apoptosis and suppresses invasion of thyroid cancer.
Cerutti et al., São Paulo, Brazil. In Oncotarget, Oct 2015
We previously showed that C1orf24 expression is increased in thyroid carcinomas.
Expression of Niban in renal interstitial fibrosis.
Tao et al., Changsha, China. In Nephrology (carlton), 2014
We identified the role of protein Niban in apoptosis of tumour cells.
AKT-dependent phosphorylation of Niban regulates nucleophosmin- and MDM2-mediated p53 stability and cell apoptosis.
Lu et al., Houston, United States. In Embo Rep, 2012
Although Niban is highly expressed in human cancer cells, the cellular functions of Niban remain largely unknown.
The new molecular markers DDIT3, STT3A, ARG2 and FAM129A are not useful in diagnosing thyroid follicular tumors.
Warren et al., Oslo, Norway. In Mod Pathol, 2012
DDIT3, STT3A, ARG2 and FAM129A immunohistochemistry does not appear to be useful in the diagnosis of thyroid follicular neoplasias, as they do not reliably distinguish follicular thyroid carcinoma from follicular thyroid adenoma.
STT3A, C1orf24, TFF3: putative markers for characterization of follicular thyroid neoplasms from fine-needle aspirates.
Zanation et al., Chapel Hill, United States. In Laryngoscope, 2011
Gene-expression data suggest a difference in expression between STT3A, Clorf24, and TFF3 in FAs versus carcinomas that may be detected from an FNA sample. Findings must be validated from preoperative FNAs in larger numbers
PVALB, a new Hürthle adenoma diagnostic marker identified through gene expression.
Riggins et al., São Paulo, Brazil. In J Clin Endocrinol Metab, 2011
The combination of PVALB and C1orf24 increased specificity to >97% and maintained sensitivity for detection of carcinoma.
Frequent expression of Niban in head and neck squamous cell carcinoma and squamous dysplasia.
Hino et al., Tokyo, Japan. In Head Neck, 2010
The expression of Niban frequently begins in the early stages of head and neck squamous carcinoma and remains upregulated throughout the carcinogenic process. Niban may be a good molecular marker of HNSCC.
The endoplasmic reticulum stress-inducible protein Niban regulates eIF2alpha and S6K1/4E-BP1 phosphorylation.
Hino et al., Tokyo, Japan. In Biochem Biophys Res Commun, 2007
Together these results suggest that Niban is involved in the ER stress response, and that Niban can modulate cell death signaling by regulating translation.
A novel tumor marker, Niban, is expressed in subsets of thyroid tumors and Hashimoto's thyroiditis.
Hino et al., Tokyo, Japan. In Hum Pathol, 2006
Niban expression is up-regulated in various types of thyroid tumors.
Diagnosis of suspicious thyroid nodules using four protein biomarkers.
Riggins et al., Baltimore, United States. In Clin Cancer Res, 2006
Previously, using expression profiling, we found that a combination of transcript expression levels from DDIT3, ARG2, C1orf24, and ITM1 distinguished between FTA and FTC.
[Pathogenesis of differentiated thyroid cancer (papillary and follicular)].
Cerutti et al., São Paulo, Brazil. In Arq Bras Endocrinol Metabol, 2005
Several genes, as TRgamma, PTEN, PKAR1A, DDIT3, ARG2, ITM1 and C1orf24--some discovered by techniques of differential gene expression--, have been recently implicated in the pathogenesis of FTC.
Hepatic angiomyolipoma and hepatic stellate cells share a similar gene expression profile.
Torbenson et al., Baltimore, United States. In Hum Pathol, 2005
Three of 5 novel markers that were identified in the AML, RRAD (Ras-related associated with diabetes), CTSK (cathepsin K), and NIBAN were also found to be overexpressed in activated stellate cells compared with quiescent or myofibroblastic stellate cells.
Multistep renal carcinogenesis in the Eker (Tsc 2 gene mutant) rat model.
Hino, Tokyo, Japan. In Curr Mol Med, 2004
To search for such genetic alterations, we identified genes (Niban and Erc) that were expressed more abundantly in renal tumors than in the normal kidney.I will review this unique model for the study of multistep renal carcinogenesis and discuss cancer prevention and delay of carcinogenesis.
Niban gene is commonly expressed in the renal tumors: a new candidate marker for renal carcinogenesis.
Hino et al., Tokyo, Japan. In Oncogene, 2004
We previously isolated the novel gene Niban expressed in renal carcinogenesis of Eker rats.
A preoperative diagnostic test that distinguishes benign from malignant thyroid carcinoma based on gene expression.
Riggins et al., São Paulo, Brazil. In J Clin Invest, 2004
Four genes (DDIT3, ARG2, ITM1, and C1orf24) differed between the two classes FTC and FTA, and a linear combination of expression levels distinguished FTC from FTA with an estimated predictive accuracy of 0.83.
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