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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Aug 2016.


Msc, MyoR, ABF-1, Musculin
The protein encoded by this gene is a transcriptional repressor capable of binding an E-box element either as a homodimer or as a heterodimer with E2A in vitro. The encoded protein also forms heterodimers with E2A proteins in vivo. This protein is capable of inhibiting the transactivation capability of E47, an E2A protein, in mammalian cells. This gene is a downstream target of the B-cell receptor signal transduction pathway. [provided by RefSeq, Jul 2008] (from NCBI)
Top mentioned proteins: CAN, MyoD, POD1, ACID, E12
Papers using Msc antibodies
Redox-shift of the pheromone-binding protein in the silkmoth Antheraea polyphemus
Leal Walter S. et al., In Journal of Chemical Ecology, 1994
... Msc I and Bam HI or Xho I (New England Biolabs, Ipswich, MA, USA), and ...
Papers on Msc
MyoR modulates cardiac conduction by repressing Gata4.
Munshi et al., Dallas, United States. In Mol Cell Biol, Feb 2015
We identify here MyoR as a novel transcription factor expressed in Cx30.2(+) cells of the AVN.
Transcription factor ABF-1 suppresses plasma cell differentiation but facilitates memory B cell formation.
Lin et al., Taipei, Taiwan. In J Immunol, 2014
In this study, we show that activated B cell factor-1 (ABF-1), which encodes a basic helix-loop-helix transcriptional repressor, participates in this regulation.
Deep Sequencing the microRNA profile in rhabdomyosarcoma reveals down-regulation of miR-378 family members.
Dominici et al., Roma, Italy. In Bmc Cancer, 2013
Ectopic expression of miR-378a-3p caused significant changes in apoptosis, cell migration, cytoskeleton organization as well as a modulation of the muscular markers MyoD1, MyoR, desmin and MyHC.
Basic helix-loop-helix transcriptional factor MyoR regulates BMP-7 in acute kidney injury.
Hishikawa et al., Tokyo, Japan. In Am J Physiol Renal Physiol, 2013
MyoR was originally identified as a transcriptional repressor in embryonic skeletal muscle precursors, but its function in adult kidney has not been clarified.
Myogenesis in the sea urchin embryo: the molecular fingerprint of the myoblast precursors.
Arnone et al., Napoli, Italy. In Evodevo, 2012
Interestingly, evolutionarily conserved myogenic factors such as Mef2, MyoR and Six1/2 are not expressed in sea urchin myoblasts but are found in other mesodermal domains of the tip of the archenteron.
Genome-wide binding of the basic helix-loop-helix myogenic inhibitor musculin has substantial overlap with MyoD: implications for buffering activity.
Tapscott et al., Seattle, United States. In Skelet Muscle, 2012
BACKGROUND: Musculin (MSC) is a basic helix-loop-helix transcription factor that inhibits myogenesis during normal development and contributes to the differentiation defect in rhabdomyosarcoma.
Myor/ABF-1 mRNA [corrected] Expression Marks Follicular Helper T Cells but Is Dispensable for Tfh Cell Differentiation and Function In Vivo.
Andris et al., Brussels, Belgium. In Plos One, 2012
MyoR/ABF-1 is a basic helix-loop-helix transcription factor that plays a role in the differentiation of the skeletal muscle and Hodgkin lymphoma.
Musculin and TCF21 coordinate the maintenance of myogenic regulatory factor expression levels during mouse craniofacial development.
Carvajal et al., London, United Kingdom. In Development, 2012
Data show that correct levels of expression of Myf5 and MyoD during mouse craniofacial development result from activation by musculin and TCF21 through direct binding to specific enhancers.
Transforming growth factor β receptor type 1 is essential for female reproductive tract integrity and function.
Matzuk et al., Houston, United States. In Plos Genet, 2011
Molecular analysis further demonstrated the dysregulation of several cell differentiation and migration genes (e.g., Krt12, Ace2, and MyoR) that are potentially associated with female reproductive tract development.
Abnormal mitoferrin-1 expression in patients with erythropoietic protoporphyria.
Bloomer et al., Birmingham, United States. In Exp Hematol, 2011
Data show that abnormal MFRN1 expression can contribute to erythropoietic protoporphyria phenotype in some patients, probably by causing a reduction in FECH activity.
Dynamic control of head mesoderm patterning.
Dietrich et al., London, United Kingdom. In Development, 2011
In phase 3, spreading Fgf signalling drives the a-p expansion of MyoR and Tbx1 expression along the pharynx, with RA limiting the expansion of MyoR.
MicroRNA-378 targets the myogenic repressor MyoR during myoblast differentiation.
Dutta et al., Charlottesville, United States. In J Biol Chem, 2011
a feed-forward loop where MyoD indirectly down-regulates MyoR via miR-378
Competitive binding of musclin to natriuretic peptide receptor 3 with atrial natriuretic peptide.
Shimomura et al., Ōsaka, Japan. In J Endocrinol, 2009
Musclin binds to NPR3 competitively with ANP and may affect ANP concentrations in a local or systemic manner.
Hypoxia induces erythroid-specific 5-aminolevulinate synthase expression in human erythroid cells through transforming growth factor-beta signaling.
Shibahara et al., Sendai, Japan. In Febs J, 2009
hypoxia induces the expression of mitoferrin mRNAs through separate mechanisms in erythroid cells
Muscle development: forming the head and trunk muscles.
Kioussi et al., Corvallis, United States. In Acta Histochem, 2007
The homeobox gene Pitx2, the T-box gene Tbx1, and the bHLH genes Tcf21 and Msc encode transcription factors that play roles in specifying progenitor cells that will give rise to branchiomeric muscles of the head.
Intrinsic inhibition of transcription factor E2A by HLH proteins ABF-1 and Id2 mediates reprogramming of neoplastic B cells in Hodgkin lymphoma.
Dörken et al., Berlin, Germany. In Nat Immunol, 2006
The B cell-specific transcription factor program was disrupted by overexpression of the helix-loop-helix proteins ABF-1 and Id2.
Allele-specific repression of lymphotoxin-alpha by activated B cell factor-1.
Kwiatkowski et al., Oxford, United Kingdom. In Nat Genet, 2004
role in repressing lymphotoxin alpha
Control of facial muscle development by MyoR and capsulin.
Olson et al., Dallas, United States. In Science, 2003
findings identify MyoR and capsulin as unique transcription factors for the development of specific head muscles
Chromosomal ARS and CEN elements bind specifically to the yeast nuclear scaffold.
Gasser et al., Lausanne, Switzerland. In Cell, 1988
The scaffold association has been closely mapped at the ARS1 locus, and appears to include the 11 bp ARS consensus, but not the ABF-1 binding site.
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