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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 13 Nov 2015.

Interleukin 17F

mL-1, IL-17F
The protein encoded by this gene is a cytokine that shares sequence similarity with IL17. This cytokine is expressed by activated T cells, and has been shown to stimulate the production of several other cytokines, including IL6, IL8, and CSF2/GM_CSF. This cytokine is also found to inhibit the angiogenesis of endothelial cells and induce endothelial cells to produce IL2, TGFB1/TGFB, and monocyte chemoattractant protein-1. [provided by RefSeq, Jul 2008] (from NCBI)
Top mentioned proteins: IL-17, IL-21, CAN, Interleukin-6, HAD
Papers on mL-1
Growth of Haematococcus pluvialis Flotow in alternative media.
Scardoeli-Truzzi et al., Jaboticabal, Brazil. In Braz J Biol, 10 Dec 2015
The highest number of cells of H. pluvialis has been reported in NPK medium (5.4 × 105cells.mL-1)
A sensitive electrochemiluminescence immunosensor for celiac disease diagnosis based on nanoelectrode ensembles.
Ugo et al., In Anal Chem, 10 Dec 2015
The immunosensor provides ECL signals which scale with anti-tTG concentration with a linearity range between 1.5 ng.mL-1 and 10 µg.mL-1 and a detection limit of 0.5 ng.mL-1.
Nisin is an effective inhibitor of Clostridium difficile vegetative cells and spore germination.
Fliss et al., In J Med Microbiol, 09 Dec 2015
Nisin A and Z both inhibited the growth of all C. difficile isolates and the MIC was estimated at 6.2 µg.mL-1 for nisin Z and 0.8 µg.mL-1 for nisin A. Besides, C. difficile spores were also susceptible to nisin A (25.6 µg.mL-1) which dropped by 40-50 % spore viability.
IL-23/IL-17A Dysfunction Phenotypes Inform Possible Clinical Effects from Anti-IL-17A Therapies.
Bissonnette et al., Portland, United States. In J Invest Dermatol, Aug 2015
Generally, downstream deficiencies (e.g., IL-17A, IL-17F) are associated with fewer disorders compared with upstream deficiencies, suggesting that selectively blocking downstream targets may result in a narrower range of side effects.
Contribution of the IL-17/IL-23 axis to the pathogenesis of inflammatory bowel disease.
Dumitraşcu et al., Cluj-Napoca / Kolozsvár, Romania. In World J Gastroenterol, Jun 2015
An efficient strategy for IBD therapy is represented by the combination of IL-17A and IL-17F in acute IL-17A knockout TNBS-induced colitis, and also definite decrease of the inflammatory process in IL-17F knockout, DSS-induced colitis have been observed.
Role of IL-25 in Immunity.
Parizad et al., Īlām, Iran. In J Clin Diagn Res, Apr 2015
Cytokine IL-17 family consists of 6 members; IL-17A to IL-17F, among which IL-25 has a unique structure and function.
The role of Th1 and Th17 cells in glomerulonephritis.
Rafieian-Kopaei et al., Sārī, Iran. In J Nephropathol, Apr 2015
CONCLUSIONS: Secretion of IL-17A, as well as IL-17F, IL-21, IL-22, suggests that Th17 subset may play a crucial role as a pleiotropic pro-inflammatory Th subset.
Deubiquitinase DUBA is a post-translational brake on interleukin-17 production in T cells.
Dixit et al., San Francisco, United States. In Nature, Mar 2015
T-helper type 17 (TH17) cells that produce the cytokines interleukin-17A (IL-17A) and IL-17F are implicated in the pathogenesis of several autoimmune diseases.
Association between IL-17F rs763780 polymorphism and susceptibility of asthma: a meta-analysis.
Li et al., Xi'an, China. In Int J Clin Exp Med, Dec 2014
Published data on the association between interleukin-17F (IL-17F) rs763780 polymorphism and asthma susceptibility are inconclusive.
Genetic Polymorphisms of IL-17F and TRAF3IP2 Could Be Predictive Factors of the Long-Term Effect of Infliximab against Crohn's Disease.
Tsukamoto et al., Nagasaki, Japan. In Biomed Res Int, Dec 2014
Multivariate logistic regression analysis indicated that the four variable factors, concomitant use of immunomodulators, penetrating disease, a G/G genotype of rs766748 in IL-17F, and a C/C or C/A genotype of rs1883136 in TRAF3IP2, independently contributed to response to IFX after 1 year of treatment.
The intracellular Ca²⁺ channel MCOLN1 is required for sarcolemma repair to prevent muscular dystrophy.
Xu et al., Ann Arbor, United States. In Nat Med, Oct 2014
MCOLN1 (also known as TRPML1, ML1) is an endosomal and lysosomal Ca(2+) channel whose human mutations cause mucolipidosis IV (ML4), a neurodegenerative disease with motor disabilities.
Anticytokine autoantibody-associated immunodeficiency.
Browne, Bethesda, United States. In Annu Rev Immunol, 2013
Examples include anti-interferon (IFN)-γ autoantibodies and disseminated nontuberculous mycobacteria; anti-granulocyte macrophage colony-stimulating factor autoantibodies and cryptococcal meningitis; anti-interleukin (IL)-6 autoantibodies and staphylococcal skin infection; and anti-IL-17A, anti-IL-17F, or anti-IL-22 autoantibodies and mucocutaneous candidiasis in the setting of either APECED (autoimmune polyendocrinopathy, candidiasis, ectodermal dystrophy syndrome) or thymoma.
An ACT1 mutation selectively abolishes interleukin-17 responses in humans with chronic mucocutaneous candidiasis.
Casanova et al., New York City, United States. In Immunity, 2013
Patients with inborn errors of interleukin-17F (IL-17F) or IL-17RA display chronic mucocutaneous candidiasis (CMC).
IL-17 cytokines in immunity and inflammation.
Dong et al., Beijing, China. In Emerg Microbes Infect, 2013
Interleukin 17 (IL-17) and its closest relative, IL-17F, have recently drawn much attention in the field of immunology.
Trypanosoma cruzi trans-sialidase initiates a program independent of the transcription factors RORγt and Ahr that leads to IL-17 production by activated B cells.
Rawlings et al., Córdoba, Argentina. In Nat Immunol, 2013
With both mouse and human primary B cells, we found that exposure to parasite-derived trans-sialidase in vitro was sufficient to trigger modification of the cell-surface mucin CD45, which led to signaling dependent on the kinase Btk and production of IL-17A or IL-17F via a transcriptional program independent of the transcription factors RORγt and Ahr.
The IL6 gene polymorphism -634C>G and IL17F gene polymorphism 7488T>C influence bone mineral density in young and elderly Japanese women.
Fuke et al., Hachiōji, Japan. In Gene, 2012
IL17F gene polymorphism 7488T>C influences bone mineral density.
Interleukin-33 induces interleukin-17F in bronchial epithelial cells.
Hizawa et al., Tsukuba, Japan. In Allergy, 2012
data suggest that the IL-33/IL-17F axis is involved in allergic airway inflammation
Association analysis of IL-17A and IL-17F polymorphisms in Chinese Han women with breast cancer.
Pang et al., Harbin, China. In Plos One, 2011
Results suggested that single-nucleotide polymorphisms (SNPs) in IL-17A but not IL-17F were associated with the risk of breast cancer.
A protective role by interleukin-17F in colon tumorigenesis.
Dong et al., Wuhan, China. In Plos One, 2011
Together, our findings demonstrate a protective role for IL-17F in colon cancer development, possibly via inhibiting tumor angiogenesis
[Dynamics of serum IL-17A, IL-17F and IL-23 in patients with inflammatory bowel disease].
Efremov et al., In Eksp Klin Gastroenterol, 2010
Dynamics of serum IL-17A, IL-17F and IL-23 in patients with inflammatory bowel disease
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