gopubmed logo
find other proteinsAll proteins
GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Aug 2016.

Mitochondrial intermediate peptidase

mitochondrial intermediate peptidase, MIP, HMIP, MIPEP
The product of this gene performs the final step in processing a specific class of nuclear-encoded proteins targeted to the mitochondrial matrix or inner membrane. This protein is primarily involved in the maturation of oxidative phosphorylation (OXPHOS)-related proteins. This gene may contribute to the functional effects of frataxin deficiency and the clinical manifestations of Friedreich ataxia. [provided by RefSeq, Jul 2008] (from NCBI)
Top mentioned proteins: MIP, Interleukin-6, MIP-2, CAN, HAD
Papers on mitochondrial intermediate peptidase
Selective solid-phase extraction using molecularly imprinted polymer as a sorbent for the analysis of fenarimol in food samples.
Roy et al., Lucknow, India. In Food Chem, Jun 2016
Binding study of molecularly imprinted and non-imprinted polymer (MIP and NIP) showed that MIP possesses a higher affinity towards this analyte compared to NIP.
Understanding the relationships between molecule structure and imprinting effect of two acetyl-nitrogen heterocyclic compounds.
Luo et al., Beijing, China. In J Mol Recognit, Feb 2016
The TAT-MIP showed excellent imprinting effect, whereas the TRAT-MIP did not.
IL-17 induces MIP-1α expression in primary mouse astrocytes via TRPC channel.
Yao et al., Nanjing, China. In Inflammopharmacology, Feb 2016
UNASSIGNED: Our previous study demonstrated IL-17-mediated induction of MIP-1α through its binding to the cognate IL-17RA and MIP-1α was involved in astrocyte activation.
TCF1 links GIPR signaling to the control of beta cell function and survival.
Drucker et al., Toronto, Canada. In Nat Med, Jan 2016
We demonstrate that mice with selective ablation of GIPR in beta cells (MIP-Cre:Gipr(Flox/Flox); Gipr(-/-βCell)) exhibit lower levels of meal-stimulated insulin secretion, decreased expansion of adipose tissue mass and preservation of insulin sensitivity when compared to MIP-Cre controls.
Molecularly imprinted polymers for separating and sensing of macromolecular compounds and microorganisms.
Kutner et al., Warsaw, Poland. In Biotechnol Adv, Jan 2016
The present review article focuses on gathering, summarizing, and critically evaluating the results of the last decade on separating and sensing macromolecular compounds and microorganisms with the use of molecularly imprinted polymer (MIP) synthetic receptors.
Molecularly imprinted polymer for caffeic acid by precipitation polymerization and its application to extraction of caffeic acid and chlorogenic acid from Eucommia ulmodies leaves.
Haginaka et al., Nishinomiya, Japan. In J Pharm Biomed Anal, Jan 2016
Binding experiments and Scatchard analyses revealed that the binding capacity and affinity of the MIP to CA are higher than those of the NIP.
Molecularly imprinted polymers as recognition materials for electronic tongues.
Kutner et al., Warsaw, Poland. In Biosens Bioelectron, Jan 2016
Merging these two sensing devices led to a synergetic hybrid sensor, an MIP based e-tongue.
Endurance and Resistance Training Affect High Fat Diet-Induced Increase of Ceramides, Inflammasome Expression, and Systemic Inflammation in Mice.
Mooren et al., Gießen, Germany. In J Diabetes Res, Dec 2015
Furthermore, ET reduced levels of MIP-1γ, while RT decreased levels of IL-18, MIP-1γ, Timp-1, and CD40 in serum (p < 0.001), respectively.
The calcium-sensing receptor as a mediator of inflammation.
Garofalo et al., Galveston, United States. In Semin Cell Dev Biol, Sep 2015
We describe in vitro studies in which we demonstrated that peripheral blood mononuclear cells in culture produce the chemokines MIP-1α and RANTES in proportion to the medium calcium concentration and they produce the chemokine MCP-1 in quantities inversely related to medium calcium concentration.
Impaired islet function in commonly used transgenic mouse lines due to human growth hormone minigene expression.
Schraenen et al., Leuven, Belgium. In Cell Metab, 2015
Although this minigene is present in the transgenes as a secondcistron, and thus not thought to be expressed, we found that three commonly used lines, Pdx1-Cre(Late), RIP-Cre, and MIP-GFP, each expressed significant amounts of hGH in pancreatic islets.
[Contemporary concepts in studies on cyclic AMP and its role in the inflammatory reaction].
Wyska et al., Laizhou, China. In Postepy Hig Med Dosw (online), 2014
In the course of the inflammatory response, the increase in cAMP level may lead to an increase in IL-10 expression, inhibition of TNF-α, IL-12, and MIP-1β release, as well as to a reduction inthe permeability of blood vessels.
Inflammatory response mechanisms exacerbating hypoxemia in coexistent pulmonary fibrosis and sleep apnea.
Balachandran et al., Chicago, United States. In Mediators Inflamm, 2014
Tissue inhibitor of metalloproteinase (TIMP-1), interleukin- (IL-) 1α, cytokine-induced neutrophil chemoattractant (CINC-1, CINC-2α/β), lipopolysaccharide induced CXC chemokine (LIX), monokine induced by gamma interferon (MIG-1), macrophage inflammatory protein- (MIP-) 1α, MIP-3α, and nuclear factor- (NF-) κB appear to mediate disease progression.
A genome-wide association study identifies two new lung cancer susceptibility loci at 13q12.12 and 22q12.2 in Han Chinese.
Shen et al., Nanjing, China. In Nat Genet, 2011
The combined analyses identified six well-replicated SNPs with independent effects and significant lung cancer associations (P < 5.0 × 10(-8)) located in TP63 (rs4488809 at 3q28, P = 7.2 × 10(-26)), TERT-CLPTM1L (rs465498 and rs2736100 at 5p15.33, P = 1.2 × 10(-20) and P = 1.0 × 10(-27), respectively), MIPEP-TNFRSF19 (rs753955 at 13q12.12,
Circulating interleukin (IL)-8, IL-2R, IL-12, and IL-15 levels are independently prognostic in primary myelofibrosis: a comprehensive cytokine profiling study.
Pardanani et al., Rochester, United States. In J Clin Oncol, 2011
RESULTS: In total, 127 PMF patients were studied; comparison with normal controls (n = 35) revealed significantly increased interleukin-1β (IL-1β), IL-1RA, IL-2R, IL-6, IL-8, IL-10, IL-12, IL-13, IL-15, tumor necrosis factor α (TNF-α), granulocyte colony-stimulating factor (G-CSF), interferon alfa (IFN-α), macrophage inflammatory protein 1α (MIP-1α), MIP-1β, hepatocyte growth factor (HGF), IFN-γ-inducible protein 10 (IP-10), monokine induced by IFN-γ (MIG), monocyte chemotactic protein 1 (MCP-1), and vascular endothelial growth factor (VEGF) levels and decreased IFN-γ levels.
The rational development of molecularly imprinted polymer-based sensors for protein detection.
Horgan et al., United Kingdom. In Chem Soc Rev, 2011
In this critical review we describe many methods used for imprinting recognition for protein targets in polymers and their incorporation with a number of transducer platforms with the aim of identifying the most promising approaches for the preparation of MIP-based protein sensors (277 references).
share on facebooktweetadd +1mail to friends