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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 08 Dec 2016.

TNFAIP3 interacting protein 3

Lind, Listeria-induced, ABIN-3
Papers on Lind
A pro-inflammatory role for A20 and ABIN family proteins in human fibroblast-like synoviocytes in rheumatoid arthritis.
Ishihara et al., Kurashiki, Japan. In Immunol Lett, 2012
recombinant transgene facilitates the transcription of pro-inflammatory cytokines in fibroblast-like synoviocytes in rheumatoid arthritis
Adiponectin inhibits pro-inflammatory signaling in human macrophages independent of interleukin-10.
Libby et al., Boston, United States. In J Biol Chem, 2009
adiponectin augmented the expression of A20, suppressor of cytokine signaling (SOCS) 3, B-cell CLL/lymphoma (BCL) 3, TNF receptor-associated factor (TRAF) 1, and TNFAIP3-interacting protein (TNIP) 3.
ABINs: A20 binding inhibitors of NF-kappa B and apoptosis signaling.
Beyaert et al., Gent, Belgium. In Biochem Pharmacol, 2009
This review defines ABIN-3 based on three different parameters: ability to bind A20; ability to inhibit NF-kappaB activation upon overexpression; the presence of specific short amino acid regions of strong homology, designated ABIN homology domains.
Expression of the NF-kappaB inhibitor ABIN-3 in response to TNF and toll-like receptor 4 stimulation is itself regulated by NF-kappaB.
Beyaert et al., Gent, Belgium. In J Cell Mol Med, 2008
Enhanced expression of ABIN-3 in monocytes is associated with sepsis
ABIN-3: a molecular basis for species divergence in interleukin-10-induced anti-inflammatory actions.
Schreiber et al., Saint Louis, United States. In Mol Cell Biol, 2007
ABIN-3 is, thus, an IL-10-induced gene product capable of attenuating NF-kappaB in human macrophages yet is inoperative in mice and represents a basis for species-specific differences in IL-10 actions.
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