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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Aug 2016.

Kinesin family member 3C

may be a vesicle associated microtubule motor protein involved in muscle cell function [RGD, Feb 2006] (from NCBI)
Top mentioned proteins: KIF3A, KIF3B, CAN, ACID, HAD
Papers on KIF3C
Kinesin-2 KIF3AC: Fast or Slow Either Head Can Start the Processive Run.
Gilbert et al., United States. In J Biol Chem, Jan 2016
The hypothesis tested was whether there is an intrinsic bias within KIF3AC such that either KIF3A or KIF3C initiates the processive run.
Suppression of motor protein KIF3C expression inhibits tumor growth and metastasis in breast cancer by inhibiting TGF-β signaling.
Xiang et al., Qingdao, China. In Cancer Lett, Dec 2015
KIF3C, a member of kinesin superfamily, functions as a motor protein involved in axonal transport in neuronal cells.
Kinesin-2 KIF3AC and KIF3AB Can Drive Long-Range Transport along Microtubules.
Gilbert et al., Madison, United States. In Biophys J, Nov 2015
Loop L11, a component of the microtubule-motor interface and implicated in activating ADP release upon microtubule collision, is significantly extended in KIF3C as compared with other kinesins.
CLIP2 as radiation biomarker in papillary thyroid carcinoma.
Hess et al., München, Germany. In Oncogene, Aug 2015
The genes comprising the first neighbourhood of CLIP2 (BAG2, CHST3, KIF3C, NEURL1, PPIL3 and RGS4) suggest the involvement of CLIP2 in the fundamental carcinogenic processes including apoptosis, mitogen-activated protein kinase signalling and genomic instability.
Whole-exome sequencing improves the diagnosis yield in sporadic infantile spasm syndrome.
Lesca et al., Lyon, France. In Clin Genet, Aug 2015
In addition, we found a possibly pathogenic variant in KIF3C that encodes a kinesin expressed during neural development.
The kinesin-2 family member KIF3C regulates microtubule dynamics and is required for axon growth and regeneration.
Fawcett et al., Cambridge, United Kingdom. In J Neurosci, 2013
Here, we identify KIF3C as a key regulator of axonal growth and regeneration by controlling microtubule dynamics and organization in the growth cone.
Kinesin-2 family in vertebrate ciliogenesis.
Malicki et al., Boston, United States. In Proc Natl Acad Sci U S A, 2012
In contrast to kif3b/kif17 double mutants, simultaneous interference with kif3b and kif3c leads to the complete loss of photoreceptor and hair cell cilia, revealing redundancy of function.
Polymorphisms inside microRNAs and microRNA target sites predict clinical outcomes in prostate cancer patients receiving androgen-deprivation therapy.
Huang et al., Taiwan. In Clin Cancer Res, 2011
KIF3C rs6728684, CDON rs3737336, and IFI30 rs1045747 genotypes remained as significant predictors for disease progression; KIF3C rs6728684, PALLD rs1071738, GABRA1 rs998754, and SYT9 rs4351800 remained as significant predictors for PCSM; and SYT9 rs4351800 remained as a significant predictor for ACM in multivariate models that included clinicopathologic predictors.
The fragile X mental retardation protein is a molecular adaptor between the neurospecific KIF3C kinesin and dendritic RNA granules.
Khandjian et al., Québec, Canada. In Hum Mol Genet, 2008
FMRP acts also as a molecular adaptor between RNA granules and the neurospecific kinesin KIF3C.
Kinesin superfamily protein-derived peptides with the ability to induce glioma-reactive cytotoxic T lymphocytes in human leukocyte antigen-A24+ glioma patients.
Yamanaka et al., Kurume, Japan. In Oncol Rep, 2007
The selected genes included three members of the kinesin superfamily proteins (KIFs): KIF1C, KIF3C, and KIF21B.
Kinesin-2 and photoreceptor cell death: requirement of motor subunits.
Williams et al., In Exp Eye Res, 2006
We have explored the possibility that the genes encoding the kinesin-2 motor proteins (KIF3A, KIF3B, and KIF3C)are linked to retinal disease, by examining retinas of knockout mice.
Development of high-throughput screens for discovery of kinesin adenosine triphosphatase modulators.
Middleton et al., Denver, United States. In Anal Biochem, 2004
The other panel members (kinesin heavy chain, chromokinesin KIF4A, KIF3C, CENP-E, MCAK, and KIFC3) were not significantly inhibited by millimolar levels of monastrol.
Identification and characterization of ASXL2 gene in silico.
Katoh et al., Narashino, Japan. In Int J Oncol, 2003
The DNMT3A-ASXL2-KIF3C locus on human chromosome 2p23.3
Functional analysis of mouse kinesin motor Kif3C.
Goldstein et al., San Diego, United States. In Mol Cell Biol, 2001
Three Kif3 family members (Kif3A, Kif3B, and Kif3C) have been identified and characterized in mice.
Expression of KIF3C kinesin during neural development and in vitro neuronal differentiation.
Cabibbo et al., Milano, Italy. In J Neurochem, 2001
KIF3A, KIF3B and KIF3C are kinesin-related motor subunits of the KIF3 family that associate to form the kinesin-II motor complex in which KIF3C and KIF3B are alternative partners of KIF3A.
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