Activation of p38- and CRM1-dependent nuclear export promotes E2F1 degradation during keratinocyte differentiation.more suppliers
In PLoS ONE, 2006
... and their sources are as follows: ß-tubulin (E7, Developmental Studies Hybridoma Bank, University of Iowa), ILK (mouse monoclonal, 611802, Transduction Laboratories, Lexington, KY, or rabbit polyclonal, 1979-1, Epitomics, Burlingame, CA), S100A3 (rabbit ...
Prolactin-induced protein in breast cancer.
Iowa City, United States. In Adv Exp Med Biol, 2014
Importantly, PIP is necessary for outside-in activation of integrin-β1 signaling pathway and regulation of key downstream signaling targets of this pathway such as interaction of integrin-β1 with integrin-linked kinase 1 (ILK1) and ErbB2.
Dis-organizing centrosomal clusters: specific cancer therapy for a generic spread?
Mainz, Germany. In Curr Med Chem, 2014
In the present review, we report advances in understanding the biology of centrosomal clustering as well as enlist compounds capable of inducing the formation of multipolar spindles such as indolquinolizines, integrin-linked kinase inhibitors (QLT-0267), noscapinoids (EM011), phthalamide derivatives (TC11), griseofulvin, phenanthridines (PJ-34), CCC1-01, CW069 GF-15, colcemid, nocodazole, paclitaxel, and vinblastine.
Integrin signalling adaptors: not only figurants in the cancer story.
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Torino, Italy. In Nat Rev Cancer, 2010
We specifically underline the contribution of p130 Crk-associated substrate (p130CAS; also known as BCAR1), neural precursor cell expressed, developmentally down-regulated 9 (NEDD9; also known as HEF1), CRK and the integrin-linked kinase (ILK)-pinch-parvin (IPP) complex to cancer, along with the more recently identified p140 Cas-associated protein (p140CAP; also known as SRCIN1).