Interleukin (IL)-25: Pleiotropic roles in asthma.
Beijing, China. In Respirology, Jan 2016
UNASSIGNED: IL-25, also named IL-17E, is a distinct member of the IL-17 cytokine family, which can promote and augment T helper type 2 (Th2) responses locally or systemically.
Drivers of chronic rhinosinusitis: Inflammation versus infection.
Boston, United States. In J Allergy Clin Immunol, Dec 2015
Conversely, certain innate factors, namely IL-25, IL-33, and thymic stromal lymphopoietin (TSLP), are elaborated by sinus epithelial cells in response to microbial stimulation or airway injury and promote local TH2 inflammation.
Role of Innate Lymphoid Cells in Lung Disease.
Tehrān, Iran. In Iran J Allergy Asthma Immunol, Aug 2015
ILC1 cells encompass natural killer (NK) cells and interferon (IFN)-g releasing cells; ILC2 cells release the Th2 cytokines, IL-5, IL-9 and IL-13 in response to IL-25 and IL-33; and ILC3 cells which release IL-17 and IL-22.
Barrier Epithelial Cells and the Control of Type 2 Immunity.
Gent, Belgium. In Immunity, Aug 2015
These ECs mount a prototypical response made up of chemokines, innate cytokines such as interleukin-1 (IL-1), IL-25, IL-33, and thymic stromal lymphopoietin (TSLP), as well as the alarmins uric acid, ATP, HMGB1, and S100 proteins.