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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 08 Dec 2016.

Interleukin 36, alpha

IL-1F6, interleukin-1 family, member-6, FIL1epsilon
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Papers on IL-1F6
Interleukin-36 (IL-36) ligands require processing for full agonist (IL-36α, IL-36β, and IL-36γ) or antagonist (IL-36Ra) activity.
GeneRIF
Sims et al., Seattle, United States. In J Biol Chem, 2012
Interleukin-36 (IL-36) ligands require processing for full agonist (IL-36alpha, IL-36beta, and IL-36gamma) or antagonist (IL-36Ra) activity
IL-36R ligands are potent regulators of dendritic and T cells.
GeneRIF
Gabay et al., Genève, Switzerland. In Blood, 2011
A critical role of IL-36R ligands in the interface between innate and adaptive immunity, leading to the stimulation of T helper responses.
IL-1F5, -F6, -F8, and -F9: a novel IL-1 family signaling system that is active in psoriasis and promotes keratinocyte antimicrobial peptide expression.
GeneRIF
Gudjonsson et al., Ann Arbor, United States. In J Immunol, 2011
Transcripts for IL-1F5, -1F6, -1F8, and -1F9 are all significantly increased in the involved skin of bitransgenic mice compared with their monotransgenic controls.
Local overexpression of interleukin-1 family, member 6 relates to the development of tubulointerstitial lesions.
GeneRIF
Kon et al., Japan. In Lab Invest, 2010
local overexpression related to the development of tubulointerstitial lesions
Externalization of the leaderless cytokine IL-1F6 occurs in response to lipopolysaccharide/ATP activation of transduced bone marrow macrophages.
GeneRIF
Gabel et al., Seattle, United States. In J Immunol, 2009
IL-1F6 can be externalized via a stimulus-coupled mechanism comparable to that used by IL-1beta
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