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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 08 Dec 2016.

Ubiquitin-conjugating enzyme E2B, RAD6 homology

HR6B, E2(14k), ubiquitin-conjugating enzyme E2(14k)
may be involved in protein catabolism in response to fasting [RGD, Feb 2006] (from NCBI)
Papers using HR6B antibodies
Loss of HR6B ubiquitin-conjugating activity results in damaged synaptonemal complex structure and increased crossing-over frequency during the male meiotic prophase.
Supplier
Kirkpatrick David T., In PLoS ONE, 2002
... Hr6b genes were amplified using primer sets (Table S2) from cDNA and sub-cloned into pGADT7 and pGBKT7 plasmids (Clontech).
Papers on HR6B
The ubiquitin-conjugating enzyme HR6B is required for maintenance of X chromosome silencing in mouse spermatocytes and spermatids.
GeneRIF
Baarends et al., Rotterdam, Netherlands. In Bmc Genomics, 2009
HR6B represses major satellite repeat transcription in spermatocytes, and functions in the maintenance of X chromosome silencing in spermatocytes and spermatids.
Increased phosphorylation and dimethylation of XY body histones in the Hr6b-knockout mouse is associated with derepression of the X chromosome.
GeneRIF
Grootegoed et al., Rotterdam, Netherlands. In J Cell Sci, 2007
HR6B exerts control over different histone modifications in spermatocytes and spermatids, and that this function contributes to the postmeiotic maintenance of X chromosome silencing.
The N-end rule ubiquitin-conjugating enzyme, HR6B, is up-regulated by nerve growth factor and required for neurite outgrowth.
GeneRIF
Klimaschewski et al., Innsbruck, Austria. In Mol Cell Neurosci, 2005
Therefore, N-end rule ubiquitination is required for neuronal differentiation of PC12 cells and may be involved in axonal regeneration of peripheral neurons.(HR6B enzyme, rat)
Spermatid nuclear and sperm periaxonemal anomalies in the mouse Ube2b null mutant.
GeneRIF
Xu et al., Paris, France. In Mol Reprod Dev, 2003
Ube2b deficient mice are shown to present male infertility and sperm head shape anomalies.
Loss of HR6B ubiquitin-conjugating activity results in damaged synaptonemal complex structure and increased crossing-over frequency during the male meiotic prophase.
GeneRIF
Grootegoed et al., Rotterdam, Netherlands. In Mol Cell Biol, 2003
The present findings reveal a specific requirement for the ubiquitin-conjugating activity of HR6B in relation to dynamic aspects of the synaptonemal complex and meiotic recombination in spermatocytes.
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