The survival-promoting peptide Y-P30 enhances binding of pleiotrophin to syndecan-2 and -3 and supports its neuritogenic activity.
In PLoS ONE, 2007
... Cruz Biotechnology, Heidelberg, Germany); α-GFP (green fluorescent protein, sc-8334; Santa Cruz Biotechnology, Heidelberg, Germany); α-GST (glutathione S-transferase, sc-57753; Santa Cruz Biotechnology, Heidelberg, Germany); α-NPM1 (nucleophosmin, ...
Changes in one-carbon metabolism after duodenal-jejunal bypass surgery.
Dublin, Ireland. In Am J Physiol Endocrinol Metab, Feb 2016
In the liver of DJB rats, betaine-homocysteine S-methyltransferase levels were decreased, whereas serine, cystathionine, cysteine, glutathione, cystathionine beta-synthase, glutathione S-transferase, and aldehyde dehydrogenase levels were increased.
Flavonoid transport mechanisms: how to go, and with whom.
Wuhan, China. In Trends Plant Sci, Sep 2015
Recent studies support and further extend previous hypotheses indicating that vacuolar sequestration of flavonoids involves vesicle trafficking, membrane transporters, and glutathione S-transferase (GST).
Polymorphisms of GSTM1, GSTT1, and p53 in Goiânia, Goiás.
Goiânia, Brazil. In Genet Mol Res, 2014
Epidemiological studies have suggested that individuals with homozygous deletion of glutathione S-transferase mu 1 (GSTM1) and glutathione S-transferase theta 1 (GSTT1) are at higher risk of developing several types of neoplasias.
Variations in Antioxidant Genes and Male Infertility.
Guangzhou, China. In Biomed Res Int, 2014
Antioxidant genes, which include catalase (CAT), glutathione peroxidase (GPX), glutathione S-transferase (GST), nitric oxide synthase (NOS), nuclear factor erythroid 2-related factor 2 (NRF2), and superoxide dismutase (SOD), play important roles in spermatogenesis and normal sperm function.
Genetic modifiers of liver disease in cystic fibrosis.
Chapel Hill, United States. In Jama, 2009
OBJECTIVE: To assess whether any of 9 polymorphisms in 5 candidate genes (alpha(1)-antitrypsin or alpha(1)-antiprotease [SERPINA1], angiotensin-converting enzyme [ACE], glutathione S-transferase [GSTP1], mannose-binding lectin 2 [MBL2], and transforming growth factor beta1 [TGFB1]) are associated with severe liver disease in patients with CF.