Fibroblast Growth Factor 10
Comparative study on the differentiation of mesenchymal stem cells between fetal and postnatal rat spinal cord niche.
Shenyang, China. In Cell Transplant, Dec 2015
In addition, we found that the expression of brain-derived neurotrophic factor (BDNF), NT-3, FGF-8, TGF-α, epidermal growth factor (EGF), and insulin-like growth factor (IGF) decreased with age, and the expression of FGF-2, FGF-10, FGF-20, TGF-α, and PDGF increased with age.
The Roles of Growth Factors in Keratinocyte Migration.
Chicago, United States. In Adv Wound Care (new Rochelle), May 2015
This review will highlight the growth factors, particularly transforming growth factor-α (TGF-α), heparin-binding epidermal growth factor (HB-EGF), insulin-like growth factor 1 (IGF-1), fibroblast growth factor 7 (FGF-7), FGF-10, and hepatocyte growth factor (HGF), which have conclusively been shown to be the most motogenic for KCs.
Harnessing the potential of lung stem cells for regenerative medicine.
Melbourne, Australia. In Int J Biochem Cell Biol, 2014
We also discuss the role of mesenchymal progenitor cells in maintaining the structural integrity of the lung and propose a model in which mesenchymal cells act as the quintessential architects of lung regeneration by providing molecular signals, such as FGF-10, to regulate the fate and specificity of epithelial stem and progenitor cells.
Hereditary disorders affecting the lacrimal system.
Iowa City, United States. In Curr Opin Ophthalmol, 2014
These disorders can be categorized into the following groups: fibroblast growth factor 10 associated disorders, tumor protein p63 associated disorders, disorders associated with the development of the branchial apparatus, and disorders associated with autonomic dysfunction.
FGF9-Pitx2-FGF10 signaling controls cecal formation in mice.
Los Angeles, United States. In Dev Biol, 2012
Using tissue specific Fgf9 versus Pitx2 loss of function approaches in the gut epithelium and/or mesenchyme, we determined that FGF9 signals to the mesenchyme via Pitx2 to induce mesenchymal Fgf10 expression, which leads to epithelial cecal bud formation.
Mutations in different components of FGF signaling in LADD syndrome.
Köln, Germany. In Nat Genet, 2006
We identified heterozygous mutations in the tyrosine kinase domains of the genes encoding fibroblast growth factor receptors 2 and 3 (FGFR2, FGFR3) in LADD families, and in one further LADD family, we detected a mutation in the gene encoding fibroblast growth factor 10 (FGF10), a known FGFR ligand.