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Esterase D

ESD, esterase D
This gene encodes a serine hydrolase that belongs to the esterase D family. The encoded enzyme is active toward numerous substrates including O-acetylated sialic acids, and it may be involved in the recycling of sialic acids. This gene is used as a genetic marker for retinoblastoma and Wilson's disease. [provided by RefSeq, Feb 2009] (from NCBI)
Top mentioned proteins: ESI, Phosphoglucomutase, ACID, Lactoylglutathione Lyase, HAD
Papers on ESD
Clinical impact of endoscopic clip closure of perforations during endoscopic submucosal dissection for colorectal tumors.
Matsuda et al., Tokyo, Japan. In Gastrointest Endosc, Feb 2016
BACKGROUND AND AIMS: Despite advances in endoscopic submucosal dissection (ESD), perforation can still occur.
Effects of administration of a proton pump inhibitor before endoscopic submucosal dissection for differentiated early gastric cancer with ulcer.
Lee et al., Puch'ŏn, South Korea. In Gastric Cancer, Jan 2016
CONCLUSIONS: Administration of PPI before ESD in differentiated EGC meeting the expanded criteria is effective to heal the ulcer lesion and reduce the mean procedure time.
Epilepsy spectrum disorders: A concept in need of validation or refutation.
Loeb et al., Kansas City, United States. In Med Hypotheses, Nov 2015
In these cases the condition could be conceptualized as an epilepsy spectrum disorder (ESD) with significant treatment implications.
Genome-wide association meta-analyses to identify common genetic variants associated with hallux valgus in Caucasian and African Americans.
Jordan et al., Boston, United States. In J Med Genet, Nov 2015
near the ESD gene (p=0.000000721×10(-7)).
Factors associated with healing of artificial ulcer after endoscopic submucosal dissection with reference to Helicobacter pylori infection, CYP2C19 genotype, and tumor location: Multicenter randomized trial.
Furuta et al., Hamamatsu, Japan. In Dig Endosc, Oct 2015
Herein, we investigated whether healing speed of artificial ulcers formed after endoscopic submucosal dissection (ESD) was influenced by H. pylori infection, CYP2C19 genotype, or other factors.
Efficacy and safety of proton pump inhibitors (PPIs) plus rebamipide for endoscopic submucosal dissection-induced ulcers: a meta-analysis.
Dong et al., Wuhan, China. In Intern Med, 2013
OBJECTIVE: To compare the efficacy of proton pump inhibitors (PPIs) with rebamipide versus PPIs alone for the treatment of ulcers after endoscopic submucosal dissection (ESD).
Preventing and controlling bleeding in gastric endoscopic submucosal dissection.
Lee et al., Seoul, South Korea. In Clin Endosc, 2013
Although techniques and instruments for endoscopic submucosal dissection (ESD) have improved, bleeding is still the most common complication.
Role of antisecretory agents for gastric endoscopic submucosal dissection.
Wang et al., Tokyo, Japan. In Dig Endosc, 2013
Gastric endoscopic submucosal dissection (ESD) causes artificial gastric ulcers and there is no consensus regarding the optimal perioperative management in terms of prevention of intra- or postoperative bleeding and promotion of healing.
Activity-based proteomics: identification of ABHD11 and ESD activities as potential biomarkers for human lung adenocarcinoma.
Weder et al., Zürich, Switzerland. In J Proteomics, 2011
Abhydrolase domain-containing protein 11 and Esterase D predict the development of distant metastases and the presence of aggressive lung adenocarcinomas.
Molecular analysis of sex chromosome-linked mutants in the silkworm Bombyx mori.
Shimada et al., Tokyo, Japan. In J Genet, 2010
Besides the Fem gene, the quantitative egg size-determining gene Esd is thought to be present on the W chromosome, based on the observation that ZWW triploid moths produce larger eggs than ZZW triploids.
[Recent advances on EUS, EUS-FNA].
Koizumi et al., Iceland. In Nihon Rinsho, 2010
3D-EUS have introduced not only preciseness for preoperative evaluation for EMR and ESD but also surface rendering similar to endoscopic image.
Crystal structure of human esterase D: a potential genetic marker of retinoblastoma.
Liu et al., Beijing, China. In Faseb J, 2009
catalytic triad residues--Ser-153, His264, and Asp230--involved in catalysis. Mutagenesis of any of the catalytic triad residues to alanine abolished the enzyme activity. Backbone amides of Leu54 & Met150 are involved in the formation of the oxyanion hole
Proteomic surveillance of retinal autoantigens in endogenous uveitis: implication of esterase D and brain-type creatine kinase as novel autoantigens.
Takeuchi et al., Tokyo, Japan. In Mol Vis, 2007
Autoantibodies to EsteD and BB-CK produced in experimental autoimmune uveoretinitis -induced mice were also detected in some endogenous uveitis patients, suggesting that these proteins might be autoantigens.
[The use of discrete characters in discriminant analysis for diagnosis of pulmonary tuberculosis and for classification of patients differing in treatment efficiency based on polymorphisms at nine codominant loci-HP, GC, TF, PI, PGM1, GLO1, C3, ACP1 and ESD].
Perel'man et al., Moscow, Russia. In Genetika, 2003
The analysis involved the data on nine polymorphic codominant loci: HP, GC, TF, PI, PGM1, GLO1, C3, ACP1, and ESD. The loci were selected by significance of differences in genotype frequencies between tuberculosis patients and healthy controls
Human retinoblastoma susceptibility gene: cloning, identification, and sequence.
Lee et al., In Science, 1987
A gene encoding a messenger RNA (mRNA) of 4.6 kilobases (kb), located in the proximity of esterase D, was identified as the retinoblastoma susceptibility (RB) gene on the basis of chromosomal location, homozygous deletion, and tumor-specific alterations in expression.
Somatic inactivation of genes on chromosome 13 is a common event in retinoblastoma.
Phillips et al., In Nature, 1983
Through family studies and analysis of patients with congenital chromosome abnormalities, the germ-line mutation responsible for the hereditary form of the eye tumour, retinoblastoma, has been assigned to the q14 region on chromosome 13 and closely linked to an enzyme called esterase D (ESD).
Gene for hereditary retinoblastoma assigned to human chromosome 13 by linkage to esterase D.
Benedict et al., In Science, 1983
Evaluation of three families with hereditary retinoblastoma demonstrates close linkage of the gene for this tumor with the genetic locus for esterase D. These results assign the gene for the hereditary form of retinoblastoma to band q14 on chromosome 13, the same region which is affected in the chromosome deletion form of this eye tumor, and therefore suggest a common underlying mechanism in the pathogenesis of these two forms of retinoblastoma.
Patient with 13 chromosome deletion: evidence that the retinoblastoma gene is a recessive cancer gene.
Sparkes et al., In Science, 1983
Although a constitutional chromosomal deletion including 13q14 has been found to date in all retinoblastoma patients whose esterase D activity is 50 percent of normal, one female patient has been found who has 50 percent esterase D activity in all normal cells examined but no deletion of 13q14 at the 550-band level.
Regional assignment of genes for human esterase D and retinoblastoma to chromosome band 13q14.
Yunis et al., In Science, 1980
The expression of human esterase D was evaluated quantitatively and qualitatively in five persons with partial deletions or duplications of chromosome 13.
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