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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Aug 2016.

Telomere fusion

Drosophila ATM, atm/tefu
Top mentioned proteins: Atm, ACID, Mre11, PCNA, Haptoglobin
Papers on Drosophila ATM
TCTP directly regulates ATM activity to control genome stability and organ development in Drosophila melanogaster.
Choi et al., Taej┼Ćn, South Korea. In Nat Commun, 2012
We identify Drosophila ATM (dATM) as a direct binding partner of dTCTP and describe a mechanistic basis for dATM activation by dTCTP.
ATM kinase inhibition in glial cells activates the innate immune response and causes neurodegeneration in Drosophila.
Wassarman et al., Madison, United States. In Proc Natl Acad Sci U S A, 2012
Mutation of the C-terminal amino acid in Drosophila ATM inhibited the kinase activity and caused neuron and glial cell death in the adult brain and a reduction in mobility and longevity.
Drosophila ATM and ATR have distinct activities in the regulation of meiotic DNA damage and repair.
GeneRIF
McKim et al., United States. In J Cell Biol, 2011
ATM is primarily required for the meiotic DSB repair response, which includes functions in DNA damage repair and negative feedback control over the level of programmed DSBs during meiosis.
Molecular genetic characterization of Drosophila ATM conserved functional domains.
GeneRIF
Campbell et al., Edmonton, Canada. In Genome, 2010
Molecular genetic characterization of Drosophila ATM conserved functional domains.
Epigenetic telomere protection by Drosophila DNA damage response pathways.
Brodsky et al., Worcester, United States. In Plos Genet, 2006
Mutations in Drosophila DNA damage response genes such as atm/tefu, mre11, or rad50 disrupt telomere protection and localization of the telomere-associated proteins HP1 and HOAP, suggesting that recognition of chromosome ends contributes to telomere protection.
Drosophila ATM and ATR checkpoint kinases control partially redundant pathways for telomere maintenance.
GeneRIF
Rong et al., Bethesda, United States. In Proc Natl Acad Sci U S A, 2005
Results suggest that ATM and ATR protect telomere integrity by safeguarding chromatin architecture that favors the loading of telomere-elongating, capping, and silencing proteins.
Drosophila ATM and Mre11 are essential for the G2/M checkpoint induced by low-dose irradiation.
GeneRIF
Rong et al., Bethesda, United States. In Genetics, 2005
Drosophila ATM and Mre11 are essential for the G2/M checkpoint induced by low-dose irradiation.
ATM is required for telomere maintenance and chromosome stability during Drosophila development.
Campbell et al., Edmonton, Canada. In Curr Biol, 2004
Here, we show that Drosophila ATM function is essential for normal adult development.
Telomere protection without a telomerase; the role of ATM and Mre11 in Drosophila telomere maintenance.
GeneRIF
Rong et al., Bethesda, United States. In Curr Biol, 2004
ATM checkpoint kinase plays a role in telomere maintenance that is independent of telomerase regulation.
The Drosophila ATM ortholog, dATM, mediates the response to ionizing radiation and to spontaneous DNA damage during development.
Settleman et al., United States. In Curr Biol, 2004
Two protein kinases, ataxia-telangiectasia mutated (ATM) and ATM and Rad-3 related (ATR), are sensors for DNA damage.
Phenotypic analysis of separation-of-function alleles of MEI-41, Drosophila ATM/ATR.
Su et al., Davis, United States. In Genetics, 2003
Here we report the generation of a null allele of mei-41, Drosophila ATM/ATR homolog, and the use of it to document a semidominant effect on a larval mitotic checkpoint and methyl methanesulfonate (MMS) sensitivity.
The Drosophila ATM homologue Mei-41 has an essential checkpoint function at the midblastula transition.
Theurkauf et al., Stony Brook, United States. In Curr Biol, 1999
RESULTS: We show that embryos lacking Mei-41, a Drosophila homologue of the ATM tumor suppressor, proceed through unusually short syncytial mitoses, fail to terminate syncytial division following mitosis 13, and degenerate without forming cells.
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