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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Aug 2016.

Kruppel-like factor 16

dopamine receptor regulating factor, DRRF, KLF16, BTEB4
Top mentioned proteins: SP1, V1a, KLF13, KLF11, mSin3A
Papers on dopamine receptor regulating factor
Rasgrf2 controls dopaminergic adaptations to alcohol in mice.
Müller et al., Erlangen, Germany. In Brain Res Bull, 2014
There was no difference in the expression of dopamine transporter (DAT), dopamine receptor regulating factor (DRRF), or dopamine D2 receptor (DRD2) mRNA in the brain between Rasgrf2 KO and WT mice.
Detailed structural-functional analysis of the Krüppel-like factor 16 (KLF16) transcription factor reveals novel mechanisms for silencing Sp/KLF sites involved in metabolism and endocrinology.
GeneRIF
Urrutia et al., Rochester, United States. In J Biol Chem, 2012
Detailed structural-functional analysis of the Kruppel-like factor 16 (KLF16) transcription factor reveals novel mechanisms for silencing Sp/KLF sites involved in metabolism and endocrinology.
Transcriptional modulation of monoaminergic neurotransmission genes by the histone deacetylase inhibitor trichostatin A in neuroblastoma cells.
Keszler et al., Budapest, Hungary. In J Neural Transm, 2012
To this end, short-term parallel cultures of SK-NF-I neuroblastoma cells were treated with TSA either alone or in combination with hypoxia, and mRNA levels of dopamine receptor D3 (DRD3) and D4 (DRD4), dopamine transporter (DAT), dopamine hydroxylase (DBH), dopamine receptor regulating factor (DRRF), catechol-O-methyltransferase (COMT), serotonin receptor 1A (HTR1A), monoamino oxidase A (MAO-A), serotonin transporter (SLC6A4) and tryptophan hydroxylase 2 (TPH2) were determined by quantitative PCR.
A functional family-wide screening of SP/KLF proteins identifies a subset of suppressors of KRAS-mediated cell growth.
Urrutia et al., Rochester, United States. In Biochem J, 2011
Using a comprehensive family-wide screening of the 24 SP/KLF members, we discovered that SP5, SP8, KLF2, KLF3, KLF4, KLF11, KLF13, KLF14, KLF15 and KLF16 inhibit cellular growth and suppress transformation mediated by oncogenic KRAS.
Hypoxia-induced transcription of dopamine D3 and D4 receptors in human neuroblastoma and astrocytoma cells.
Keszler et al., Budapest, Hungary. In Bmc Neurosci, 2008
On the other hand, mRNA levels of type 2 dopamine receptor, dopamine transporter, monoamino oxidase and catechol-O-methyltransferase were unaltered, while those of the dopamine receptor regulating factor (DRRF) were decreased by hypoxia.
Transcriptional auto-regulation of the dopamine receptor regulating factor (DRRF) gene.
GeneRIF
Mouradian et al., Pusan, South Korea. In Mol Cell Endocrinol, 2008
DRRF auto-regulates its own promoter
Identification of the imprinted KLF14 transcription factor undergoing human-specific accelerated evolution.
Scherer et al., Toronto, Canada. In Plos Genet, 2007
Due to the intronless nature of Klf14 and its homology to Klf16, we suggest that the gene is an ancient retrotransposed copy of Klf16.
Bifurcated converging pathways for high Ca2+- and TGFbeta-induced inhibition of growth of normal human keratinocytes.
Huh et al., Okayama, Japan. In Proc Natl Acad Sci U S A, 2005
Sp1 complexed with NFAT1 in high Ca2+-treated cells or with Smad3 in TGFbeta1-treated cells, but not Sp1 alone, replaced KLF16 from the p21WAF1/CIP1 promoter and transcriptionally activated the p21WAF1/CIP1 gene.
Cell and tissue specific expression of human Krüppel-like transcription factors in human ocular surface.
Sapin et al., Clermont-Ferrand, France. In Mol Vis, 2004
RESULTS: We detected the presence of twelve transcripts of KLFs in the cornea (KLF2, KLF3, KLF4, KLF5, KLF6, KLF7, KLF8, KLF10, KLF11, KLF12, KLF13, and KLF16) and eight in the conjunctiva (KLF2, KLF3, KLF4, KLF6, KLF7, KLF10, KLF11, and KLF12).
Genomic organization and promoter characterization of the murine dopamine receptor regulating factor (DRRF) gene.
Mouradian et al., Pusan, South Korea. In Gene, 2003
To study the transcriptional mechanisms by which expression of the dopamine receptor regulating factor (DRRF) gene is regulated, a murine genomic clone was isolated using a DRRF cDNA as probe.
Signaling disrupts mSin3A binding to the Mad1-like Sin3-interacting domain of TIEG2, an Sp1-like repressor.
Urrutia et al., Rochester, United States. In Embo J, 2002
We recently reported that a structurally and functionally related Mad1-like SID is also present in five Sp1-like repressor proteins (TIEG1, TIEG2, BTEB1, BTEB3 and BTEB4), demonstrating that SID-mSin3A interactions have a wider functional impact on transcriptional repression.
Developmental expression of the zinc finger transcription factor DRRF (dopamine receptor regulating factor).
GeneRIF
Mouradian et al., Bethesda, United States. In Mech Dev, 2002
DRRF mRNA is expressed uniquely during development at all time points tested with high levels observed at E12, E14 and E16 in various tissues
A conserved alpha-helical motif mediates the interaction of Sp1-like transcriptional repressors with the corepressor mSin3A.
Urrutia et al., Rochester, United States. In Mol Cell Biol, 2001
Interestingly, the alpha-HRM is conserved in both the TIEG (TIEG1 and TIEG2) and BTEB (BTEB1, BTEB3, and BTEB4) subfamilies of Sp1-like proteins.
Dopamine receptor regulating factor, DRRF: a zinc finger transcription factor.
Mouradian et al., Bethesda, United States. In Proc Natl Acad Sci U S A, 2001
We describe here a zinc finger type transcription factor, designated dopamine receptor regulating factor (DRRF), which binds to GC and GT boxes in the D1A and D2 dopamine receptor promoters and effectively displaces Sp1 and Sp3 from these sequences.
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