gopubmed logo
 
find other proteinsAll proteins
GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Aug 2016.

CCKAR cholecystokinin A receptor

cholecystokinin-A receptor
Top mentioned proteins: Cholecystokinin, HAD, V1a, ACID, Gastrin
Papers on cholecystokinin-A receptor
Cholecystokinin A receptor (CCKAR) gene variation is associated with language lateralization.
Beste et al., Bochum, Germany. In Plos One, 2012
We found a significant association of the cholecystokinin-A receptor (CCKAR) gene variation rs1800857 and language lateralization assessed using the dichotic listening task.
[Frequency combination of a gallstone disease and diabetes].
Review
Iamlikhanova et al., In Eksp Klin Gastroenterol, 2010
Possible mechanisms for the relationship between GSD and DM--gallbladder hypomotility, decrease of cholecystokinin-A receptor (CCK-A) gene expression and decreased sensitivity to CCK, insulin resistance, decreased activity of PPAR-receptors (peroxisome proliferator-activated receptor) in patients with DM.
Anti-inflammatory action of cholecystokinin and melatonin in the rat parotid gland.
Ekström et al., Göteborg, Sweden. In Oral Dis, 2010
The cholecystokinin-A receptor antagonist lorglumide reduced the inhibitory effect of cholecystokinin-8, while the melatonin 2-preferring receptor antagonist luzindole had no effect on the melatonin-induced inhibition.
An intron 1 polymorphism in the cholecystokinin-A receptor gene associated with schizophrenia in males.
Rehfeld et al., Copenhagen, Denmark. In Acta Psychiatr Scand, 2009
OBJECTIVE: To identify whether a genetic variation (rs1800857; IVS1-5T>C) in the neuropeptide cholecystokinin-A receptor (CCKAR) gene is a risk factor in the pathogenesis of schizophrenia.
Parasympathetic non-adrenergic, non-cholinergic transmission in rat parotid glands: effects of cholecystokinin-A and -B receptor antagonists on the secretory response.
Cevik Aras et al., Göteborg, Sweden. In Regul Pept, 2008
In this study, the parasympathetic innervation was stimulated in non-atropinized (in periods of 2 min) or atropinized (in periods of 3 min) pentobarbitone-anaesthetized rats before and after administration of the cholecystokinin-A receptor antagonist lorglumide (48 mg/kg, i.v.) and the cholecystokinin-B receptor antagonist itriglumide (5.5 mg/kg, i.v.).
Disturbance of response to acute thermal pain in naturally occurring cholecystokinin-a receptor gene knockout Otsuka Long-Evans Tokushima Fatty (OLETF) rats.
Funakoshi et al., Tokyo, Japan. In J Pharmacol Sci, 2006
Otsuka Long-Evans Tokushima Fatty (OLETF) rats lack cholecystokinin-A receptor (CCK-AR) because of a genetic abnormality.
Cholecystokinin octapeptide improves cardiac function by activating cholecystokinin octapeptide receptor in endotoxic shock rats.
Xing et al., Shantou, China. In World J Gastroenterol, 2005
The expression of cholecystokinin-A receptor (CCK-AR) and cholecystokinin-B receptor (CCK-BR) mRNA of myocardium in ES rats was examined by reverse transcription polymerase chain reaction (RT-PCR).
Differences in ethanol ingestion between cholecystokinin-A receptor deficient and -B receptor deficient mice.
Funakoshi et al., Tokyo, Japan. In Alcohol Alcohol, 2005
AIMS: Cholecystokinin (CCK) modulates dopamine release in the nucleus accumbens through the CCK-A receptor (CCK-AR).
Mechanisms of exocrine pancreatic toxicity induced by oral treatment with 2,3,7,8-tetrachlorodibenzo-p-dioxin in female Harlan Sprague-Dawley Rats.
Nyska et al., United States. In Toxicol Sci, 2005
The cholecystokinin-A receptor (CCK-A receptor; CCKAR) and duodenal cholecystokinin 8 (CCK) revealed the associations of dioxin treatment with hormonal changes.
Absence of the cholecystokinin-A receptor deteriorates homeostasis of body temperature in response to changes in ambient temperature.
Miyasaka et al., Tokyo, Japan. In Am J Physiol Regul Integr Comp Physiol, 2004
The circadian rhythm of the body core temperature (T(c)) and the effects of changes in ambient temperatures on the homeostasis of T(c) in Otsuka Long Evans Tokushima Fatty (OLETF) rats, which are naturally occurring cholecystokinin (CCK)-A receptor (CCK-AR) gene knockout (-/-) rats, were examined.
Association of cholecystokinin-A receptor gene polymorphisms and panic disorder in Japanese.
Funakoshi et al., Tokyo, Japan. In Am J Med Genet B Neuropsychiatr Genet, 2004
Several lines of evidence have suggested that naturally occurring alterations in cholecystokinin (CCK) systems could contribute to the development of panic disorder (PD).
Association of cholecystokinin-A receptor gene polymorphism with alcohol dependence in a Japanese population.
Funakoshi et al., Tokyo, Japan. In Alcohol Alcohol, 2004
AIMS: Cholecystokinin (CCK), one of the most abundant neurotransmitter peptides, interacts with dopamine.
Enhancement of electroacupuncture-induced analgesic effect in cholecystokinin-A receptor deficient rats.
Bae et al., Seoul, South Korea. In Brain Res Bull, 2004
Previously, we have showed that the cholecystokinin (CCK)-A receptor expression in hypothalamus is closely related with the responsiveness of electroacupuncture (EA)-mediated analgesic effects in rats.
Moleular models for cholecystokinin-A receptor.
Review
Lybrand et al., Nashville, United States. In Pharmacol Toxicol, 2002
Numerous techniques have been used to elucidate the structural basis for interaction of cholecystokinin (CCK)-related peptides with their hormone-binding receptor, the CCK-A receptor (CCK-AR), including structure-activity relationship studies, site-directed mutagenesis, photoaffinity-labeling, and solution NMR analysis of both CCK peptide ligands and peptide fragments derived from the CCK-A receptor.
Phosphorylation and desensitization of the pancreatic cholecystokinin-A receptor.
Review
De Pont et al., Nijmegen, Netherlands. In Digestion, 1996
The eukaryotic cell uses a variety of mechanisms to protect itself from overstimulation.
share on facebooktweetadd +1mail to friends