Papers using
CaMKIV
antibodies
Papers on
CaMKIV
Caffeine and REM sleep deprivation: Effect on basal levels of signaling molecules in area CA1.Alhaider et al., Houston, United States. In Mol Cell Neurosci, Feb 2016
Western blot analysis of basal protein levels of plasticity- and memory-related signaling molecules in hippocampal area CA1 showed significant down regulation of the basal levels of phosphorylated- and total-CaMKII, phosphorylated- and total-CREB as well as those of BDNF and CaMKIV in sleep deprived rats.
Evolutionary and functional perspectives on signaling from neuronal surface to nucleus.Ma et al., New York City, United States. In Biochem Biophys Res Commun, May 2015
This intricate pathway depends on a specific role for multiple Ca(2+)/CaM-dependent kinases and phosphatases: α/βCaMKII phosphorylates γCaMKII to trap CaM; CaN dephosphorylates γCaMKII to dispatch it to the nucleus; and PP2A induces CaM release from γCaMKII so that CaMKK and CaMKIV can trigger CREB phosphorylation.
Mouse models of genetic effects on cognition: relevance to schizophrenia.Weinberger et al., Genova, Italy. In Neuropharmacology, 2012
Based on functional hypotheses, gene modifications within five model systems are described: 1) dopamine (D1, D2, D3, D4, D5, DAT, COMT, MAO); 2) glutamate (GluR-A, NR1, NR2A, NR2B, GRM2, GRM3, GLAST); 3) GABA (α(5), γ(2), α(4), δGABA(A), GABA(B(1)), GAT1); 4) acetylcholine (nAChRβ2, α7, CHRM1); and 5) calcium (CaMKII-α, neurogranin, CaMKKβ, CaMKIV).