Gonadotrophin-releasing hormone signalling downstream of calmodulin.
Haifa, Israel. In J Neuroendocrinol, 2012
Gonadotrophin gene expression is also regulated by GnRH-induced CaM-dependent kinases (CaMKs); CaMKI is able to derepress the histone deacetylase-inhibition of β-subunit gene expression, whereas CaMKII appears to be essential for the GnRH-activation of all three subunit genes.
Neurogenetics of slow axonal transport: from cells to animals.
Mumbai, India. In J Neurogenet, 2012
Recent reports have unraveled the molecular basis of the transport of certain slow component proteins, such as the neurofilament subunits, tubulin, and certain soluble enzymes such as Ca(2+)/calmodulin-dependent protein kinase IIa (CaM kinase IIa), etc., in tissue cultured neurons.
CaMKII in cerebral ischemia.
Aurora, United States. In Acta Pharmacol Sin, 2011
However, CaMKII and other members of the CaM kinase family have been implicated in regulation of both neuronal death and survival.