Charlottesville, United States. In Neurochem Res, Dec 2015
Glutamate-loaded vesicles undergo removal of Synapsin I by CaM kinase II-mediated phosphorylation, transforming to the release-ready pool.
Gonadotrophin-releasing hormone signalling downstream of calmodulin.
Haifa, Israel. In J Neuroendocrinol, 2012
Gonadotrophin gene expression is also regulated by GnRH-induced CaM-dependent kinases (CaMKs); CaMKI is able to derepress the histone deacetylase-inhibition of β-subunit gene expression, whereas CaMKII appears to be essential for the GnRH-activation of all three subunit genes.
Neurogenetics of slow axonal transport: from cells to animals.
Mumbai, India. In J Neurogenet, 2012
Recent reports have unraveled the molecular basis of the transport of certain slow component proteins, such as the neurofilament subunits, tubulin, and certain soluble enzymes such as Ca(2+)/calmodulin-dependent protein kinase IIa (CaM kinase IIa), etc., in tissue cultured neurons.