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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Aug 2016.

Adrenergic, beta-1-, receptor

beta 1-adrenergic receptor, X-beta1AR
Top mentioned proteins: HAD, CAN, ACID, V1a, AGE
Papers on beta 1-adrenergic receptor
let-7e replacement yields potent anti-arrhythmic efficacy via targeting beta 1-adrenergic receptor in rat heart.
Yang et al., Harbin, China. In J Cell Mol Med, 2014
Beta-adrenoceptor (β-AR) exerts critical regulation of cardiac function.
Development and crystallization of a minimal thermostabilised G protein-coupled receptor.
Schertler et al., Cambridge, United Kingdom. In Protein Expr Purif, 2009
We recently solved the structure of a thermostabilised turkey beta 1-adrenergic receptor by crystallization in the presence of the detergent octylthioglucoside.
Association of beta 1-adrenergic receptor gene polymorphisms with left ventricular hypertrophy in human essential hypertension.
Hui et al., Beijing, China. In Clin Biochem, 2008
OBJECTIVES: Experimental evidence support a key role for beta (1)-adrenergic receptor (ADRB1) in the modulation of cardiac mass.
[Effect of chronic myocardial infarction on the distribution of beta-adrenoceptors in heart: experiment with dogs].
Huang et al., Harbin, China. In Zhonghua Yi Xue Za Zhi, 2007
Four weeks later metoprolol, a beta 1-adrenergic receptor antagonist, was injected intravenously.
[389A/G polymorphism of the human beta1-adrenergic receptor in patients with acute myocardial infarction].
Yang et al., Shanghai, China. In Zhonghua Yi Xue Yi Chuan Xue Za Zhi, 2007
OBJECTIVE: To investigate the relation between the 389A/G polymorphism in the human beta 1-adrenergic receptor and acute myocardial infarction (AMI).
Inhibition of p38 alpha MAPK rescues cardiomyopathy induced by overexpressed beta 2-adrenergic receptor, but not beta 1-adrenergic receptor.
Vatner et al., Newark, United States. In J Clin Invest, 2007
We examined the role of p38alpha MAPK in mediating cardiomyopathy in mice overexpressing beta(1)-adrenergic receptor (beta(1)-AR) or beta(2)-AR by mating them with dominant-negative p38alpha (DNp38alpha) MAPK mice.
Impaired gene expression of beta 1-adrenergic receptor, but not stimulatory G-protein Gs alpha, in rat ventricular myocardium treated with isoproterenol.
Hara et al., Japan. In Biol Pharm Bull, 2004
We investigated the gene expression of beta(1)-adrenergic receptor (beta(1)AR) and stimulatory G-protein Gsalpha, important signal transduction elements for regulating heart rate and contractility, in ventricle after chronic treatment with isoproterenol (ISO) in rat.
Structural and functional complexity of the humoral response against the Trypanosoma cruzi ribosomal P2 beta protein in patients with chronic Chagas' heart disease.
Levin et al., Buenos Aires, Argentina. In Clin Exp Immunol, 2004
These antibodies also recognize an epitope on the second extracellular loop of the beta 1-adrenergic receptor, inducing a functional response on cardiomyocytes.
Direct evidence for a beta 1-adrenergic receptor-directed autoimmune attack as a cause of idiopathic dilated cardiomyopathy.
Lohse et al., Würzburg, Germany. In J Clin Invest, 2004
Today, dilated cardiomyopathy (DCM) represents the main cause of severe heart failure and disability in younger adults and thus is a challenge for public health.
Modeling beta-adrenergic control of cardiac myocyte contractility in silico.
McCulloch et al., San Diego, United States. In J Biol Chem, 2003
3-Fold overexpression of adenylyl cyclase in the model allowed an 85% higher rate of cyclic AMP synthesis than an equivalent overexpression of beta 1-adrenergic receptor, and manipulating the affinity of Gs alpha for adenylyl cyclase was a more potent regulator of cyclic AMP production.
cAMP-mediated signal transduction and sarcoplasmic reticulum function in heart failure.
Review
Movsesian, Salt Lake City, United States. In Ann N Y Acad Sci, 1998
Instead, there is reason to believe that the selective reduction in beta 1-adrenergic receptor density in failing myocardium is causally related to this compartment-specific decrease in cAMP content through an as-yet-undetermined mechanism.
Beta-adrenergic receptors in failing human myocardium.
Review
Brodde, Halle, Germany. In Basic Res Cardiol, 1995
In the failing human myocardium beta 1-adrenergic receptor number is decreased, and this is accompanied by a reduced beta 1-adrenergic receptor mediated positive inotropic effect.
Is the senescent heart overloaded and already failing?
Review
Swynghedauw et al., Paris, France. In Cardiovasc Drugs Ther, 1994
Changes in specific mRNAs include: a shift in myosin heavy chain (MHC) isogene expression leading to an increased beta MHC content; decreased densities of Ca2+ ATPase of the sarcoplasmic reticulum, beta 1-adrenergic receptor, and muscarinic receptors; and attenuation of the Na+/Ca2+ exchange activity.
Neurologic consequences of hypertension and antihypertensive drug therapy.
Review
van Groen et al., Birmingham, United States. In Curr Opin Nephrol Hypertens, 1994
As a class, the angiotensin-converting enzyme inhibitors most consistently lead to cognitive improvement in the overall hypertensive population, but beta 1-adrenergic receptor blockers and a subset of calcium channel blockers appear to have very similar effects.
Modulation of adrenergic receptors and G-transduction proteins in failing human ventricular myocardium.
Review
Feldman, Baltimore, United States. In Circulation, 1993
This insensitivity is a result of alterations in the function of this signal transduction pathway, including selective downregulation of the beta 1-adrenergic receptor, uncoupling of beta 2-adrenergic receptors from adenylyl cyclase, and an increase in the functional activity of the inhibitory G-protein.
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