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Serine/arginine-rich splicing factor 6

B52
The protein encoded by this gene is involved in mRNA splicing and may play a role in the determination of alternative splicing. The encoded nuclear protein belongs to the splicing factor SR family and has been shown to bind with and modulate another member of the family, SFRS12. Alternative splicing results in multiple transcript variants. In addition, two pseudogenes, one on chromosome 17 and the other on the X chromosome, have been found for this gene.[provided by RefSeq, Sep 2010] (from NCBI)
Top mentioned proteins: HAD, HLA-B, HLA-A, CAN, POLYMERASE
Papers on B52
Is there a genetic predisposition to new-onset diabetes after kidney transplantation?
New
Reddy et al., Chennai, India. In Saudi J Kidney Dis Transpl, Nov 2015
Overall, Hepatitis C (P = 0.007), human leukocyte antigen (HLA) B52 (P = 0.03) and lack of HLA A28 (A68/69) (P = 0.03) were associated with TAH.
Bilateral hearing impairment as the initial symptom of sympathetic ophthalmia.
New
Horie et al., Tokyo, Japan. In Am J Otolaryngol, Jul 2015
Patient 1 carried HLA-DR4, HLA-DR15, HLA-A33, HLA-A24, HLA-B44 and HLA-B52, and patient 2 carried HLA-DR4.
A role for the serine/arginine-rich (SR) protein B52/SRSF6 in cell growth and myc expression in Drosophila.
New
Juge et al., Montpellier, France. In Genetics, Apr 2015
Here, we analyzed phenotypes induced by overexpression of the SR protein B52 during Drosophila development and identified several proteins that can rescue these phenotypes.
Effects of rhinovirus species on viral replication and cytokine production.
Gern et al., Madison, United States. In J Allergy Clin Immunol, 2014
METHODS: We cloned full-length cDNA of RV-A16, A36, B52, B72, C2, C15, and C41 from clinical samples and grew clinical isolates of RV-A7 and RV-B6 in cultured cells.
Frequency analysis of HLA class I alleles in Iranian patients with progressive and non-progressive chronic lymphocytic leukemia.
Shokri et al., Stockholm, Sweden. In Hum Immunol, 2014
Similarly, a higher frequency of HLA-B52:01 (p=0.037)
New insights into HLA class I association to Behçet's syndrome in Iranian Azari patients.
Baradaran et al., Tabrīz, Iran. In Auto Immun Highlights, 2013
Remarkably, HLA-B35 frequency was higher in the patient than control group, and the frequency of HLA-B51, HLA-B52, and HLA-BW4 was significantly elevated.
Equilibrium p-T phase diagram of boron: experimental study and thermodynamic analysis.
Kurakevych et al., Paris, France. In Sci Rep, 2012
The high-temperature part of the diagram contains p-T domains of thermodynamic stability of rhombohedral β-B106, orthorhombic γ-B28, pseudo-cubic (tetragonal) t'-B52, and liquid boron (L).
Behçet's syndrome in Iranian Azari people.
Bandehagh et al., Tabrīz, Iran. In Pak J Biol Sci, 2012
HLA-B5, HLA-B35, HLA-51, HLA-B52 and HLA-CW4 presented significantly high frequencies in all patients.
Dual-specificity tyrosine phosphorylation-regulated kinase 1A (Dyrk1A) modulates serine/arginine-rich protein 55 (SRp55)-promoted Tau exon 10 inclusion.
GeneRIF
Liu et al., Nantong, China. In J Biol Chem, 2012
Phosphorylation of SRp55 by Dyrk1A suppressed its ability to promote Tau exon 10 inclusion.
Evidence for upregulation of Bim and the splicing factor SRp55 in melanoma cells from patients treated with selective BRAF inhibitors.
GeneRIF
Hersey et al., Sydney, Australia. In Melanoma Res, 2012
Report upregulation of Bim and the splicing factor SRp55 in melanoma cells from patients treated with selective BRAF inhibitors.
Inhibition of splicing by serine-arginine rich protein 55 (SRp55) causes the appearance of partially spliced HIV-1 mRNAs in the cytoplasm.
GeneRIF
Schwartz et al., Uppsala, Sweden. In Virus Res, 2011
SRp55 aids in the generation of partially spliced and unspliced HIV-1 mRNAs.
Serine- and arginine-rich proteins 55 and 75 (SRp55 and SRp75) induce production of HIV-1 vpr mRNA by inhibiting the 5'-splice site of exon 3.
GeneRIF
Schwartz et al., Uppsala, Sweden. In J Biol Chem, 2010
Serine- and arginine-rich proteins 55 and 75 (SRp55 and SRp75) induce production of HIV-1 vpr mRNA by inhibiting the 5'-splice site of exon 3.
The SR protein B52/SRp55 is required for DNA topoisomerase I recruitment to chromatin, mRNA release and transcription shutdown.
GeneRIF
Tazi et al., Montpellier, France. In Plos Genet, 2010
B52 delivers Topo I to RNA polymerase II-active chromatin loci and DNA topology and mRNA release can be coordinated to control gene expression.
[Alleles of the major histocompatibility system associated with susceptibility to the development of Takayasu's arteritis].
Review
Vargas-Alarcón et al., Mexico. In Gac Med Mex, 2002
On Asian populations TA was associated with HLA-A31, -B52, -B39, -B5 and -DR2, on Arabs with HLA-A2, -A9, -B35 and -DR7, on North-Americans with HLA-DR4 and on Mexican Mestizo with HLA-B5, -B52 and -DR6.
Takayasu's arteritis.
Review
Medina et al., Mexico. In Curr Rheumatol Rep, 2002
A high frequency of haplotype A24-B52-DR2 has been found in Japanese patients, without this association in other populations.
Ulcerative colitis associated with Takayasu's disease in two patients who received proctocolectomy.
Review
Kondo et al., Nerima, Japan. In J Gastroenterol, 2001
HLA typing showed A2, A31(19), B52(5), and DR2(DRB1*1502).
Inflammation and atherosclerosis. Atherosclerotic lesions in Takayasu arteritis.
Review
Kobayashi et al., Tokyo, Japan. In Ann N Y Acad Sci, 2000
Autoimmune processes stimulated by viral infection and other unknown causative factors may play an important role under these pathophysiological conditions because HLA analysis revealed a statistically significant high frequency of haplotype A24-B52-DR2 in these patients in Japan.
[Cardio vascular disease associated with ulcerative colitis].
Review
Hayashi et al., Tokyo, Japan. In Nihon Rinsho, 1999
HLA-B52 and DR2, were highly expressed in the patient with both disease.
Molecular-marker-facilitated investigation of host-plant response to Exserohilum turcicum in maize (Zea mays L.): components of resistance.
Woodman et al., Ames, United States. In Theor Appl Genet, 1994
RFLPs were used to investigate components of host-plant response to Exserohilum turcicum in 150 unselected F2∶3 lines of a B52/Mo17 maize population.
Quantitative and qualitative trait loci affecting host-plant response to Exserohilum turcicum in maize (Zea mays L.).
Martinson et al., Ames, United States. In Theor Appl Genet, 1993
Molecular markers at 103 loci were used to identify the location of quantitative sources of resistance to Exserohilum turcicum in 150 F2∶3 lines of a B52/Mo17 maize population.
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