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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Aug 2016.

Protein tyrosine phosphatase-like A domain containing 1

B-ind1, butyrate-induced transcript 1, hB-ind1, butyrate-induced transcript B-ind1, Ptplad1
Top mentioned proteins: Rac1, NF-kappaB, Akt, Hsp90, RbAp46
Papers on B-ind1
Genetic Loci implicated in erythroid differentiation and cell cycle regulation are associated with red blood cell traits.
GeneRIF
Kullo et al., Rochester, United States. In Mayo Clin Proc, 2012
Genes in the erythroid differentiation and cell cycle regulation pathways influence interindividual variation in RBC indices. Our results provide insights into the molecular basis underlying variation in RBC traits.
Cochaperone activity of human butyrate-induced transcript 1 facilitates hepatitis C virus replication through an Hsp90-dependent pathway.
GeneRIF
Matsuura et al., Suita, Japan. In J Virol, 2009
These results suggest that an Hsp90-dependent chaperone pathway incorporating hB-ind1 is involved in protein folding in the membranous web for the circumvention of the UPR and that it facilitates HCV replication
Tumor necrosis factor (TNF)-alpha persistently activates nuclear factor-kappaB signaling through the type 2 TNF receptor in chromaffin cells: implications for long-term regulation of neuropeptide gene expression in inflammation.
Anouar et al., Mont-Saint-Aignan, France. In Endocrinology, 2008
TNF-alpha, acting through the TNF-R2, causes an early up-regulation of NF-kappaB, long-lasting induction of the NF-kappaB target gene inhibitor kappaB (IkappaB), and persistent stimulation of other NF-kappaB-associated genes including mitogen-inducible gene-6 (MIG-6), which acts as an IkappaB signaling antagonist, and butyrate-induced transcript 1. Consistent with long-term activation of the NF-kappaB signaling pathway, delayed induction of neuropeptide gene transcription by TNF-alpha in chromaffin cells is blocked by an antagonist of NF-kappaB signaling.
Human butyrate-induced transcript 1 interacts with hepatitis C virus NS5A and regulates viral replication.
GeneRIF
Matsuura et al., Suita, Japan. In J Virol, 2008
results suggest that hB-ind1 plays a crucial role in HCV RNA replication and the propagation of JFH1 virus through interaction with viral and host proteins
Human B-ind1 gene promoter: cloning and regulation by histone deacetylase inhibitors.
GeneRIF
Redeuilh et al., Paris, France. In Gene, 2006
The structure of the B-IND1 gene promoter region was determined.
SSeCKS/Gravin/AKAP12 attenuates expression of proliferative and angiogenic genes during suppression of v-Src-induced oncogenesis.
Gelman et al., Bethesda, United States. In Bmc Cancer, 2005
RESULTS: SSeCKS re-expression resulted in the attenuation of critical Src-induced proliferative and pro-angiogenic gene expression including Afp, Hif-1alpha, Cdc20a and Pdgfr-beta, and conversely, SSeCKS induced several cell cycle regulatory genes such as Ptpn11, Gadd45a, Ptplad1, Cdkn2d (p19), and Rbbp7.
Identification of genes differentially expressed in V79 cells grown as multicell spheroids.
Olive et al., Vancouver, Canada. In Int J Radiat Biol, 2002
RESULTS: Genes upregulated in the outer cell layer of spheroids relative to monolayers included: (1) mts1 (S100A4), a calcium binding protein implicated in proliferation, metastasis, cell adhesion, and angiogenesis; (2) cytochrome c oxidase II; (3) B-ind1, a mediator of Rac-1 signaling; (4) TRAM, an endoplasmic reticulum protein.
B-ind1, a novel mediator of Rac1 signaling cloned from sodium butyrate-treated fibroblasts.
Mester et al., Paris, France. In J Biol Chem, 2000
A novel butyrate-induced transcript B-ind1 has been cloned by this approach.
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