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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Aug 2016.

ATPIF1 ATPase inhibitory factor 1

ATPase inhibitory factor
Top mentioned proteins: ATPase, IF1, V1a, Ros, SNAIL
Papers on ATPase inhibitory factor
Down-regulation of oxidative phosphorylation in the liver by expression of the ATPase inhibitory factor 1 induces a tumor-promoter metabolic state.
New
Cuezva et al., Madrid, Spain. In Oncotarget, Dec 2015
UNASSIGNED: The ATPase Inhibitory Factor 1 (IF1) is an inhibitor of the mitochondrial H+-ATP synthase that regulates the activity of both oxidative phosphorylation (OXPHOS) and cell death.
ATPase inhibitory factor 1 is a potential prognostic marker for the migration and invasion of glioma.
New
Wang et al., Zhengzhou, China. In Oncol Lett, Oct 2015
UNASSIGNED: Adenosine triphosphatase inhibitory factor 1 (IF1) has previously been considered to be a driving oncogene in human cancers.
The shrimp mitochondrial FoF1-ATPase inhibitory factor 1 (IF1).
New
Muhlia-Almazan et al., Hermosillo, Mexico. In J Bioenerg Biomembr, Oct 2015
In this study, we report two variants of the mitochondrial FOF1-ATPase Inhibitory Factor 1 (IF1) ubiquitously expressed across tissues of the Litopenaeus vannamei transcriptome: the IF1_Lv1 and the IF1_Lv2.
ATPase inhibitory factor 1 is a prognostic marker and contributes to proliferation and invasion of human gastric cancer cells.
New
Cui et al., Wuhan, China. In Biomed Pharmacother, Mar 2015
ATPase inhibitory factor 1 (IF1) has recently been considered as a potential oncogene in human cancers.
Silencing of ATPase Inhibitory Factor 1 Inhibits Cell Growth via Cell Cycle Arrest in Bladder Cancer.
Shuin et al., Kōchi, Japan. In Pathobiology, 2014
OBJECTIVE: The role of the ATPase inhibitory factor 1 (IF1) is inhibit the hydrolase activity of F1Fo-ATPase when oxidative phosphorylation is impaired.
Reciprocal activation between ATPase inhibitory factor 1 and NF-κB drives hepatocellular carcinoma angiogenesis and metastasis.
Liu et al., Harbin, China. In Hepatology, 2014
Here we evaluate the effects of ATPase inhibitory factor 1 (IF1), an inhibitor of the mitochondrial H(+)-adenosine triphosphate (ATP) synthase, on HCC angiogenesis and metastasis.
Upregulation of both heme oxygenase-1 and ATPase inhibitory factor 1 renders tumoricidal activity by synthetic flavonoids via depleting cellular ATP.
Kim et al., Suwŏn, South Korea. In Bioorg Med Chem Lett, 2014
Heme oxygenase-1 (HO-1) and ATPase inhibitory factor (ATPIF) 1 is often overexpressed in different types of cancer cells.
In vivo inhibition of the mitochondrial H+-ATP synthase in neurons promotes metabolic preconditioning.
Cuezva et al., Madrid, Spain. In Embo J, 2014
The expression of the ATPase inhibitory factor 1 (IF1) is a strategy used by cancer cells to inhibit the activity of the H(+)-ATP synthase to generate a ROS signal that switches on cellular programs of survival.
Genome-wide RNAi screen identifies ATPase inhibitory factor 1 (ATPIF1) as essential for PARK2 recruitment and mitophagy.
Screaton et al., Ottawa, Canada. In Autophagy, 2013
In a genome-wide RNAi screen searching for genes that are required for PARK2 translocation to the mitochondria, we identified ATPase inhibitory factor 1 (ATPIF1/IF1) as essential for PARK2 recruitment and mitophagy in cultured cells.
Degradation of IF1 controls energy metabolism during osteogenic differentiation of stem cells.
Cuezva et al., Madrid, Spain. In Embo Rep, 2013
Herein, we demonstrate that the ATPase inhibitory factor 1 (IF1) is expressed in hMSCs and in prostate and colon stem cells but is not expressed in the differentiated cells.
Expression, regulation and clinical relevance of the ATPase inhibitory factor 1 in human cancers.
Cuezva et al., Madrid, Spain. In Oncogenesis, 2012
Recent findings in colon cancer cells indicate that inhibition of the mitochondrial H(+)-adenosine triphosphate (ATP) synthase by the ATPase inhibitory factor 1 (IF1) promotes aerobic glycolysis and a reactive oxygen species (ROS)-mediated signal that enhances proliferation and cell survival.
Mitochondrial Atpif1 regulates haem synthesis in developing erythroblasts.
Impact
Paw et al., Boston, United States. In Nature, 2012
To investigate this we used zebrafish genetic screens and cloned mitochondrial ATPase inhibitory factor 1 (atpif1) from a zebrafish mutant with profound anaemia, pinotage (pnt (tq209)).
The mitochondrial ATPase inhibitory factor 1 triggers a ROS-mediated retrograde prosurvival and proliferative response.
Cuezva et al., Madrid, Spain. In Mol Cell, 2012
Recent findings indicate that prevalent human carcinomas overexpress the mitochondrial ATPase Inhibitory Factor 1 (IF1).
Binding of the inhibitor protein IF(1) to bovine F(1)-ATPase.
GeneRIF
Walker et al., Cambridge, United Kingdom. In J Mol Biol, 2011
The N-terminal region of IF(1) destabilizes the interaction of the inhibitor with F(1)-ATPase and may assist in removing the inhibitor from its binding site when F(1)F(o)-ATPase is making ATP
Up-regulation of the ATPase inhibitory factor 1 (IF1) of the mitochondrial H+-ATP synthase in human tumors mediates the metabolic shift of cancer cells to a Warburg phenotype.
Cuezva et al., Madrid, Spain. In J Biol Chem, 2010
During ischemia, the ATP hydrolytic activity of the enzyme is inhibited by the ATPase inhibitory factor 1 (IF1).
Glutamic acid in the inhibitory site of mitochondrial ATPase inhibitor, IF(1), participates in pH sensing in both mammals and yeast.
GeneRIF
Ichikawa et al., Ōsaka, Japan. In J Biochem, 2008
in addition to His49, Glu26 participates in pH sensing in bovine IF(1), and the mechanism of pH sensing mediated by Glu26 is different from the dimer-tetramer model proposed previously
Marinobufagenin, an endogenous alpha-1 sodium pump ligand, in hypertensive Dahl salt-sensitive rats.
Bagrov et al., Baltimore, United States. In Hypertension, 2001
Recently, we have shown that mammalian tissues contain a bufadienolide Na(+)/K(+)-ATPase inhibitory factor, marinobufagenin (MBG), that exhibits greater affinity for the alpha-1 than alpha-3 sodium pump isoform.
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