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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Aug 2016.

Ataxin 7-like 3

Ataxin 7-like 3, ATXN7L3
Top mentioned proteins: Histone, USP22, H2A, DSS1, Domino
Papers on Ataxin 7-like 3
The Cellular Protein Complex Associated with a Transforming Region of E1A Contains c-MYC.
Chinnadurai et al., Saint Louis, United States. In J Virol, 2014
The same E1A region additionally interacted with the constituents of a deubiquitinase complex consisting of USP22, ATXN7, and ATXN7L3 via TRRAP.
The tightly controlled deubiquitination activity of the human SAGA complex differentially modifies distinct gene regulatory elements.
GeneRIF
Tora et al., Illkirch-Graffenstaden, France. In Mol Cell Biol, 2011
Downregulation of ATXN7L3 by short hairpin RNA speci fi cally inactivated the SAGA deubiquitination activity, leading to a strong increase of global H2B ubiquitination and a moderate increase of H2A ubiquitination.
The structural plasticity of SCA7 domains defines their differential nucleosome-binding properties.
GeneRIF
Kieffer et al., Illkirch-Graffenstaden, France. In Embo Rep, 2010
The solution structures of the SCA7 domain of both ATXN7 and ATXN7L3 reveal a new, common zinc-finger motif at the heart of two distinct folds, providing a molecular basis for the observed functional differences.
A TFTC/STAGA module mediates histone H2A and H2B deubiquitination, coactivates nuclear receptors, and counteracts heterochromatin silencing.
GeneRIF
Devys et al., Tokyo, Japan. In Mol Cell, 2008
ATXN7L3, USP22, & ENY2 are required cofactors for full transcriptional activity by nuclear receptors. The deubiquitinase activity of TFTC/STAGA HAT counteracts heterochromatin silencing & is a positive cofactor for in vivo nuclear receptor activation.
Genome-wide survey for biologically functional pseudogenes.
Lagergren et al., Stockholm, Sweden. In Plos Comput Biol, 2006
Two processed sequences are notable, their conservation since the human-mouse split being as high as most protein-coding genes; one is derived from the protein Ataxin 7-like 3 (ATX7NL3), and one from the Spinocerebellar ataxia type 1 protein (ATX1).
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