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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 25 Jan 2016.

Apolipoprotein Ea

apolipoprotein E
Top mentioned proteins: apolipoprotein E, AGE, HAD, CAN, V1a
Papers on apolipoprotein E
Neuregulin-1 attenuates development of nephropathy in a type 1 diabetes mouse model with high cardiovascular risk.
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De Keulenaer et al., Antwerp, Belgium. In Am J Physiol Endocrinol Metab, 19 Feb 2016
We tested this hypothesis in a hypercholesterolemic apolipoprotein E-deficient (ApoE-/-) type 1 diabetes mouse model, prone to the development of cardiomyopathy, atherosclerosis and nephropathy, and compared the effects of NRG-1 with insulin.
Notch1 Inhibition Reduces Low Shear Stress-Induced Plaque Formation.
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Zhang et al., Jinan, China. In Int J Biochem Cell Biol, 16 Feb 2016
Apolipoprotein E-deficient (ApoE(-/-)) mice were fed with high fat diet and administered intraperitoneally with DAPT (a γ-secretase inhibitor).
Proteomics Approach Identifies Factors Associated With the Response to Low-Density Lipoprotein Apheresis Therapy in Patients With Steroid-Resistant Nephrotic Syndrome.
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Uchida et al., Yokohama, Japan. In Ther Apher Dial, 14 Feb 2016
Comparative analysis of the column-bound proteins between responders and non-responders by 2-DE demonstrated that apolipoprotein E (APOE) and SAP levels were increased in non-responders as compared with responders.
ATRQβ-001 vaccine prevents atherosclerosis in apolipoprotein E-null mice.
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Chen et al., Wuhan, China. In J Hypertens, 14 Feb 2016
The purpose of this study was to investigate whether ATRQβ-001 vaccine would prevent atherosclerosis in apolipoprotein E-null (ApoE) mice.
The role of endoplasmic reticulum stress in hippocampal insulin resistance.
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Feldman et al., Ann Arbor, United States. In Exp Neurol, 13 Feb 2016
In the current study, we investigated ER stress in the hippocampus of 3 different mouse models of metabolic syndrome: the C57BL6 mouse on a high fat (HF) diet; apolipoprotein E, leptin, and apolipoprotein B-48 deficient (ApoE 3KO) mice; and the low density lipoprotein receptor, leptin, and apolipoprotein B-48 deficient (LDLR 3KO) mice.
High homocysteine and epistasis between MTHFR and APOE: Association with cognitive performance in the elderly.
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Guaita et al., Milano, Italy. In Exp Gerontol, 13 Feb 2016
MTHFR C677T TT was associated with higher tHcy but did not affect cognitive performance per se; however, when combined with the apolipoprotein E (APOE)-ε4 allele it was a risk factor for lower executive performance, independently of tHcy levels.
The APOE epsilon 4 polymorphism does not predict late onset depression: the Three-City Study.
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Tzourio et al., Bordeaux, France. In Neurobiol Aging, 31 Jan 2016
UNASSIGNED: The apolipoprotein E ε4 allele (APOE4) is an established risk factor for dementia; however, conflicting findings have been reported as to whether this phenotype confers a heightened risk for late onset depression (LOD) independent of dementia.
Mitochondrial DNA-LL-37 Complex Promotes Atherosclerosis by Escaping from Autophagic Recognition.
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Impact
Lai et al., Kunming, China. In Immunity, 15 Jan 2016
Mouse model studies indicated that Cramp-mtDNA complex aggravated atherosclerotic lesion formation in apolipoprotein E-deficient mice and antibody treatment against the complex alleviated the lesion.
Cognitive Function in Patients With Colorectal Cancer Who Do and Do Not Receive Chemotherapy: A Prospective, Longitudinal, Controlled Study.
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Impact
Tannock et al., Sydney, Australia. In J Clin Oncol, 01 Jan 2016
Blood tests included cytokines, clotting factors, apolipoprotein E genotype, and sex hormones.
Pdcd4 deficiency enhances macrophage lipoautophagy and attenuates foam cell formation and atherosclerosis in mice.
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Zhang et al., Jinan, China. In Cell Death Dis, 31 Dec 2015
Importantly, Pdcd4 deficiency in mice significantly upregulated macrophage autophagy in local plaques along with attenuated lipid accumulation and atherosclerotic lesions in high-fat-fed Apolipoprotein E knockout mice.
Interleukin 18 function in atherosclerosis is mediated by the interleukin 18 receptor and the Na-Cl co-transporter.
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Shi et al., Boston, United States. In Nat Med, Jul 2015
Here, we show that absence of the IL18 receptor (IL18r) does not affect atherosclerosis in apolipoprotein E-deficient (Apoe(-/-)) mice, nor does it affect IL18 cell surface binding to or signaling in endothelial cells.
Prevalence of amyloid PET positivity in dementia syndromes: a meta-analysis.
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Brooks et al., Maastricht, Netherlands. In Jama, Jun 2015
MAIN OUTCOMES AND MEASURES: Estimated prevalence of positive amyloid PET scans according to diagnosis, age, and apolipoprotein E (APOE) ε4 status, using the generalized estimating equations method.
Prevalence of cerebral amyloid pathology in persons without dementia: a meta-analysis.
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Impact
Zetterberg et al., Maastricht, Netherlands. In Jama, Jun 2015
MAIN OUTCOMES AND MEASURES: Prevalence of amyloid pathology on positron emission tomography or in cerebrospinal fluid according to AD risk factors (age, apolipoprotein E [APOE] genotype, sex, and education) estimated by generalized estimating equations.
A systematic review and meta-analysis of plasma amyloid 1-42 and tau as biomarkers for Alzheimer's disease.
Rani et al., Coimbatore, India. In Sage Open Med, 2014
CONCLUSION: This analysis pinpoints that plasma Aβ42 and tau could not serve as reliable markers independently for diagnosis of Alzheimer's disease and a cohort study with age, sex and apolipoprotein E correction is warranted for their possible use as Alzheimer's disease markers.
Association between ARNTL (BMAL1) rs2278749 polymorphism T >C and susceptibility to Alzheimer disease in a Chinese population.
Zhang et al., China. In Genet Mol Res, 2014
This case-control study examined the genotypes of apolipoprotein E (APOE e4) and BMAL1 rs2278749 T/C using restriction fragment length polymorphism and the TaqMan assay, respectively.
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