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Apolipoprotein A-I binding protein

apolipoprotein A-I binding protein, AI-BP, apoA-I binding protein, APOA1BP, apolipoprotein A-I binding protein of
The product of this gene interacts with apolipoprotein A-I (apoA-I), the major apolipoprotein of high-density lipoproteins (HDLs). It is secreted into some bodily fluids, and its synthesis and secretion are stimulated in vitro by incubating cells with apoA-I. The human genome contains related pseudogenes. [provided by RefSeq, Jul 2008] (from NCBI)
Top mentioned proteins: HDL, apolipoprotein A-I, OUT, Apo, CAN
Papers on apolipoprotein A-I binding protein
AIBP: A Novel Molecule at the Interface of Cholesterol Transport, Angiogenesis, and Atherosclerosis.
New
Fang et al., Houston, United States. In Methodist Debakey Cardiovasc J, Jul 2015
We have identified a protein called apoA-I binding protein (AIBP) that augments HDL functionality by accelerating cholesterol efflux.
Role of G-quadruplex located at 5' end of mRNAs.
Maiti et al., Delhi, India. In Biochim Biophys Acta, 2014
RESULTS: PG4 sequences in 5' UTR of AKT interacting protein (AKTIP), cathepsin B (CTSB) and forkhead box E3 (FOXE3) mRNAs form G-quadruplex whereas it is unable to form G-quadruplex in apolipoprotein A-I binding protein (APOA1BP).
Overexpression of nuclear apoptosis-inducing factor 1 altered the proteomic profile of human gastric cancer cell MKN45 and induced cell cycle arrest at G1/S phase.
Huang et al., Beijing, China. In Plos One, 2013
We found five proteins (proteasome 26S subunit 2, proteasome 26S subunit 13, NADH dehydrogenase Fe-S protein 1, chaperonin containing TCP1 subunit 3 and thioredoxin reductase 1) that were up-regulated and three proteins (ribonuclease inhibitor 1, 14-3-3 protein epsilon isoform and apolipoprotein A-I binding protein) that were down-regulated in the MKN45 cells overexpressing NAIF1.
Genome-wide meta-analysis identifies new susceptibility loci for migraine.
Impact
International Headache Genetics Consortium et al., Cambridge, United Kingdom. In Nat Genet, 2013
Brain tissue expression quantitative trait locus analysis suggests potential functional candidate genes at four loci: APOA1BP, TBC1D7, FUT9, STAT6 and ATP5B.
Control of angiogenesis by AIBP-mediated cholesterol efflux.
Impact
Miller et al., San Diego, United States. In Nature, 2013
Here we show that apoA-I binding protein (AIBP) accelerates cholesterol efflux from endothelial cells to HDL and thereby regulates angiogenesis.
Treatment of aortoiliac occlusive or dilatative disease concomitant with kidney transplantation: how and when?
Dionigi et al., Varese, Italy. In Int J Surg, 2012
MAIN FINDINGS: AI pathology distribution was as follows: 15 iliac stenoses treated with thromboendarterectomy (TEA), 2 Leriche syndrome and 1 aortic aneurism treated with an aortobisiliac bypass (AI-BP), and 3 aneurysms treated with endovascular aortic repair (EVAR).
Identification of unknown protein function using metabolite cocktail screening.
Minor et al., Charlottesville, United States. In Structure, 2012
The YjeF_N proteins, represented by mouse apolipoprotein A-I binding protein and the N-terminal domain of Tm0922, were found to interact with an adenosine diphosphoribose-related substrate and likely serve as ADP-ribosyltransferases. Crystallographic screening of metabolites serves as an efficient tool in functional analyses of uncharacterized proteins.
Proteomic analysis identifies dysfunction in cellular transport, energy, and protein metabolism in different brain regions of atypical frontotemporal lobar degeneration.
GeneRIF
Bahn et al., Cambridge, United Kingdom. In J Proteome Res, 2012
A protein encoded by this locus was found to be differentially expressed in postmortem brains from patients with atypical frontotemporal lobar degeneration.
Extremely conserved ATP- or ADP-dependent enzymatic system for nicotinamide nucleotide repair.
Linster et al., Brussels, Belgium. In J Biol Chem, 2012
We also show that eukaryotic proteins homologous to the N-terminal domain of YjeF (apolipoprotein A-1-binding protein (AIBP) in mammals, YNL200C in yeast) catalyze the epimerization of the S and R forms of NAD(P)HX, thereby allowing, in conjunction with the energy-dependent dehydratase, the repair of both epimers of NAD(P)HX.
Proteomic profiling of naturally protected extraocular muscles from the dystrophin-deficient mdx mouse.
Ohlendieck et al., Ireland. In Biochem Biophys Res Commun, 2010
Desmin, apolipoprotein A-I binding protein and perilipin-3 were found to be increased and gelsolin, gephyrin, transaldolase, and acyl-CoA dehydrogenase were shown to be decreased in mdx extraocular muscles.
Protein profiling analysis of skeletal muscle of a pufferfish, Takifugu rubripes.
Chen et al., Zhenjiang, China. In Mol Biol Rep, 2010
There were six structural proteins, such as alpha-actin, tropomyosin, and myosin heavy chain, and six with known functions such as T-cell receptor alpha chain, 4SNc-Tudor domain protein, SMC3 protein, and Translin associated factor X, as well as nine hypothetical novel proteins, including titin, andretinol dehydrogenase, and apolipoprotein A-I binding protein.
Molecular characterization of EmABP, an apolipoprotein A-I binding protein secreted by the Echinococcus multilocularis metacestode.
Brehm et al., W├╝rzburg, Germany. In Infect Immun, 2009
The encoded protein, EmABP, displays significant homologies to apolipoprotein A-I binding protein (AI-BP) of mammalian origin and to metazoan YjeF_N domain proteins.
Biochemical and structural characterization of apolipoprotein A-I binding protein, a novel phosphoprotein with a potential role in sperm capacitation.
GeneRIF
Herr et al., Charlottesville, United States. In Endocrinology, 2008
AI-BP plays an important role in capacitation possibly providing a link between protein phosphorylation and cholesterol efflux.
Gene expression profiling in hepatocellular carcinoma: upregulation of genes in amplified chromosome regions.
Wilkens et al., Hannover, Germany. In Mod Pathol, 2008
Comparison of significance analysis of microarray and gene set enrichment analysis narrowed down the number of dysregulated genes to 18, with 7 genes localised on 1q22 (SCAMP3, IQGAP3, PYGO2, GPATC4, ASH1L, APOA1BP, and CCT3).
Adiponectin accelerates reverse cholesterol transport by increasing high density lipoprotein assembly in the liver.
GeneRIF
Yamashita et al., Suita, Japan. In Biochem Biophys Res Commun, 2007
Taken together, the current study demonstrates that APN might protect against atherosclerosis by increasing HDL assembly through enhancing ABCA1 pathway and apoA-1 synthesis in the liver.
ApoA-I-binding protein (AI-BP) and its homologues hYjeF_N2 and hYjeF_N3 comprise the YjeF_N domain protein family in humans with a role in spermiogenesis and oogenesis.
Schmitz et al., Regensburg, Germany. In Horm Metab Res, 2007
The screening for additional human YjeF_N domain containing proteins beside the apolipoprotein A-I interacting protein (AI-BP), identified two other genes designated hYjeF_N2-15q23 (formerly human homologue of yeast edc3) and hYjeF_N3-19p13.11
Cell surface-expressed moesin-like HDL/apoA-I binding protein promotes cholesterol efflux from human macrophages.
Yamashita et al., Suita, Japan. In J Lipid Res, 2006
We recently reported the involvement of a glycosylphosphatidylinositol anchor (GPI anchor) in the binding of HDL and apoA-I on human macrophages, and purified an 80 kDa HDL/apoA-I binding protein.
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